Origin and evolution of gene for prolactin-induced protein.

[1]  W. Swanson,et al.  Pervasive Adaptive Evolution in Primate Seminal Proteins , 2005, PLoS genetics.

[2]  Colin N. Dewey,et al.  Sequence and comparative analysis of the chicken genome provide unique perspectives on vertebrate evolution , 2004, Nature.

[3]  N. Mundy,et al.  Rapid evolution by positive Darwinian selection in the extracellular domain of the abundant lymphocyte protein CD45 in primates. , 2004, Molecular biology and evolution.

[4]  Sudhir Kumar,et al.  MEGA3: Integrated software for Molecular Evolutionary Genetics Analysis and sequence alignment , 2004, Briefings Bioinform..

[5]  W. Tian,et al.  Divergent evolution of the prolactin-inducible protein gene and related genes in the mouse genome. , 2004, Gene.

[6]  A. Camarca,et al.  Structural study of GCDFP-15/gp17 in disease versus physiological conditions using a proteomic approach. , 2003, Biochemistry.

[7]  N. Saitou,et al.  Adaptive evolution of the IgA hinge region in primates. , 2002, Molecular biology and evolution.

[8]  S. Sugano,et al.  Search for genes positively selected during primate evolution by 5'-end-sequence screening of cynomolgus monkey cDNAs. , 2002, Genomics.

[9]  R. Ward,et al.  Natural selection on the erythrocyte surface. , 2002, Molecular biology and evolution.

[10]  P. Tucker,et al.  Episodic evolution of pyrin in primates: human mutations recapitulate ancestral amino acid states , 2001, Nature Genetics.

[11]  M. Yoshida,et al.  Identification of two rodent genes encoding homologues to seminal vesicle autoantigen: a gene family including the gene for prolactin-inducible protein. , 2001, Biochemical and biophysical research communications.

[12]  M. Daly,et al.  A map of human genome sequence variation containing 1.42 million single nucleotide polymorphisms , 2001, Nature.

[13]  D. Higgins,et al.  T-Coffee: A novel method for fast and accurate multiple sequence alignment. , 2000, Journal of molecular biology.

[14]  Justin C. Fay,et al.  Hitchhiking under positive Darwinian selection. , 2000, Genetics.

[15]  Z Mishal,et al.  Mapping the CD4 binding domain of gp17, a glycoprotein secreted from seminal vesicles and breast carcinomas. , 2000, Biochemistry.

[16]  G. Manco,et al.  A Novel Aspartyl Proteinase from Apocrine Epithelia and Breast Tumors* , 2000, The Journal of Biological Chemistry.

[17]  N. Saitou,et al.  Evolution of Rh Blood Group Genes Have Experienced Gene Conversions and Positive Selection , 1999, Journal of Molecular Evolution.

[18]  Y. Chen,et al.  Seminal vesicle autoantigen, a novel phospholipid-binding protein secreted from luminal epithelium of mouse seminal vesicle, exhibits the ability to suppress mouse sperm motility. , 1999, The Biochemical journal.

[19]  D. Piatier‐Tonneau,et al.  Potent inhibition of CD4/TCR-mediated T cell apoptosis by a CD4-binding glycoprotein secreted from breast tumor and seminal vesicle cells. , 1999, Journal of immunology.

[20]  N. Saitou,et al.  Conserved evolution of the Rh50 gene compared to its homologous Rh blood group gene. , 1998, Biochemical and biophysical research communications.

[21]  M. Nei,et al.  Positive Darwinian selection after gene duplication in primate ribonuclease genes. , 1998, Proceedings of the National Academy of Sciences of the United States of America.

[22]  Ziheng Yang,et al.  PAML: a program package for phylogenetic analysis by maximum likelihood , 1997, Comput. Appl. Biosci..

[23]  E. Veerman,et al.  In Vivo Binding of the Salivary Glycoprotein EP-GP (Identical to GCDFP-15) to Oral and Non-Oral Bacteria Detection and Identification of EP-GP Binding Species , 1997, Biological chemistry.

[24]  M. Nei,et al.  A new method of inference of ancestral nucleotide and amino acid sequences. , 1995, Genetics.

[25]  J. Thompson,et al.  CLUSTAL W: improving the sensitivity of progressive multiple sequence alignment through sequence weighting, position-specific gap penalties and weight matrix choice. , 1994, Nucleic acids research.

[26]  T Gojobori,et al.  Statistical analysis of nucleotide sequences of the hemagglutinin gene of human influenza A viruses. , 1994, Proceedings of the National Academy of Sciences of the United States of America.

[27]  William R. Taylor,et al.  The rapid generation of mutation data matrices from protein sequences , 1992, Comput. Appl. Biosci..

[28]  R. Shiu,et al.  The prolactin-inducible protein (PIP/GCDFP-15) gene: Cloning, structure and regulation , 1991, Molecular and Cellular Endocrinology.

[29]  M. Affolter,et al.  Primary structure of a new actin-binding protein from human seminal plasma. , 1991, European journal of biochemistry.

[30]  F. Tajima.  Statistical method for testing the neutral mutation hypothesis by DNA polymorphism. , 1989, Genetics.

[31]  J. Hamerton,et al.  The gene for prolactin-inducible protein (PIP), uniquely expressed in exocrine organs, maps to chromosome 7 , 1989, Somatic cell and molecular genetics.

[32]  M. Nei,et al.  Nucleotide substitution at major histocompatibility complex class II loci: evidence for overdominant selection. , 1989, Proceedings of the National Academy of Sciences of the United States of America.

[33]  M. Nei,et al.  Pattern of nucleotide substitution at major histocompatibility complex class I loci reveals overdominant selection , 1988, Nature.

[34]  L. Murphy,et al.  Isolation and sequencing of a cDNA clone for a prolactin-inducible protein (PIP). Regulation of PIP gene expression in the human breast cancer cell line, T-47D. , 1987, The Journal of biological chemistry.

[35]  N. Saitou,et al.  The neighbor-joining method: a new method for reconstructing phylogenetic trees. , 1987, Molecular biology and evolution.

[36]  M. Nei.  Molecular Evolutionary Genetics , 1987 .

[37]  M. Nei,et al.  Simple methods for estimating the numbers of synonymous and nonsynonymous nucleotide substitutions. , 1986, Molecular biology and evolution.

[38]  M. Kimura.  A simple method for estimating evolutionary rates of base substitutions through comparative studies of nucleotide sequences , 1980, Journal of Molecular Evolution.

[39]  G. A. Watterson.  On the number of segregating sites in genetical models without recombination. , 1975, Theoretical population biology.

[40]  J. Vaidya,et al.  Diseases of the breast , 2000, British Journal of Cancer.

[41]  Gapped BLAST and PSI-BLAST: A new , 1997 .

[42]  J. Adachi,et al.  MOLPHY, programs for molecular phylogenetics , 1992 .