Ineffectiveness of organic calcium channel blockers in antagonizing long-term potentiation

[1]  P. C. Schwindt,et al.  The induction and modification of voltage-sensitive responses in cat neocortical nuerons by N-methyl-d-aspartate , 1986, Brain Research.

[2]  A. Ganong,et al.  Long-term potentiation in the hippocampus involves activation of N-methyl-D-aspartate receptors , 1984, Brain Research.

[3]  D. Durand,et al.  Impairment of long-term potentiation in rat hippocampus following chronic ethanol treatment , 1984, Brain Research.

[4]  R. Delorenzo,et al.  Micromolar-affinity benzodiazepine receptors regulate voltage-sensitive calcium channels in nerve terminal preparations. , 1984, Proceedings of the National Academy of Sciences of the United States of America.

[5]  B. R. Sastry,et al.  Verapamil counteracts the masking of long-lasting potentiation of hippocampal population spike produced by 2-amino-5-phosphonovalerate. , 1984, Life sciences.

[6]  G. Lynch,et al.  Intracellular injections of EGTA block induction of hippocampal long-term potentiation , 1983, Nature.

[7]  R. Dingledine N‐methyl aspartate activates voltage‐dependent calcium conductance in rat hippocampal pyramidal cells. , 1983, The Journal of physiology.

[8]  D. Prince,et al.  Electrophysiology of isolated hippocampal pyramidal dendrites , 1982, The Journal of neuroscience : the official journal of the Society for Neuroscience.

[9]  S. Snyder,et al.  Autoradiographic visualization of [3H]nitrendipine binding sites in rat brain: localization to synaptic zones. , 1982, European journal of pharmacology.

[10]  J. Miller,et al.  Calcium-induced long-term potentiation in the hippocampus , 1982, Neuroscience.

[11]  W. Douglas,et al.  Slowing effects of dopamine and calcium‐channel blockers on frequency of sodium spikes in rat pars intermedia cells , 1982, The Journal of physiology.

[12]  D. Durand,et al.  Ethanol in low doses augments calcium-mediated mechanisms measured intracellularly in hippocampal neurons. , 1982, Science.

[13]  W. Abraham,et al.  Augmentation of short-term plasticity in CA1 of rat hippocampus after chronic ethanol treatment , 1981, Brain Research.

[14]  D. Durand,et al.  Effects of low concentrations of ethanol on CA1 hippocampal neurons in vitro , 1981 .

[15]  T. Godfraind,et al.  The inhibition by flunarizine of the norepinephrine-evoked contraction and calcium influx in rat aorta and mesenteric arteries. , 1981, The Journal of pharmacology and experimental therapeutics.

[16]  H. Jahnsen,et al.  The effects of a benzodiazepine on the hyperpolarizing and the depolarizing responses of hippocampal cells to GABA , 1981, Brain Research.

[17]  P. Andersen,et al.  Possible mechanisms for long‐lasting potentiation of synaptic transmission in hippocampal slices from guinea‐pigs. , 1980, The Journal of physiology.

[18]  D. Prince,et al.  A calcium-activated hyperpolarization follows repetitive firing in hippocampal neurons. , 1980, Journal of neurophysiology.

[19]  M. Blaustein,et al.  The effects of some organic "calcium antagonists" on calcium influx in presynaptic nerve terminals. , 1979, Molecular pharmacology.

[20]  I. Spector,et al.  The calcium action potential and a prolonged calcium dependent after‐hyperpolarization in mouse neuroblastoma cells. , 1979, The Journal of physiology.

[21]  G. Lynch,et al.  The relationship between extracellular calcium concentrations and the induction of hippocampal long-term potentiation , 1979, Brain Research.

[22]  D. Prince,et al.  Intradendritic recordings from hippocampal neurons. , 1979, Proceedings of the National Academy of Sciences of the United States of America.

[23]  T. Ehara,et al.  The voltage- and time-dependent effects of (-)-verapamil on the slow inward current in isolated cat ventricular myocardium. , 1978, The Journal of pharmacology and experimental therapeutics.

[24]  G. Lynch,et al.  Synaptic transmission is required for initiation of long-term potentiation , 1978, Brain Research.

[25]  T. Teyler,et al.  Transient heterosynaptic depression in the hippocampal slice , 1978, Brain Research Bulletin.

[26]  J. V. van Beek,et al.  Effect of flunarizine on calcium-induced responses of peripheral vascular smooth muscle. , 1978, Archives internationales de pharmacodynamie et de therapie.

[27]  P. Schwartzkroin,et al.  Probable calcium spikes in hippocampal neurons , 1977, Brain Research.

[28]  B. McNaughton,et al.  Physiological identification and analysis of dentate granule cell responses to stimulation of the medial and lateral perforant pathways in the rat , 1977, The Journal of comparative neurology.

[29]  P. Andersen,et al.  Specific long-lasting potentiation of synaptic transmission in hippocampal slices , 1977, Nature.

[30]  J. Macdonald Substitution of extracellular sodium ions blocks the voltage-dependent decrease of input conductance evoked by L-aspartate. , 1984, Canadian journal of physiology and pharmacology.

[31]  G. Collingridge,et al.  Excitatory amino acids in synaptic transmission in the Schaffer collateral‐commissural pathway of the rat hippocampus. , 1983, The Journal of physiology.

[32]  P. Andersen,et al.  Calcium dependency of synaptic long-lasting potentiation in the hippocampal slice. , 1979, Acta physiologica Scandinavica.

[33]  A. Fleckenstein Specific pharmacology of calcium in myocardium, cardiac pacemakers, and vascular smooth muscle. , 1977, Annual review of pharmacology and toxicology.

[34]  H. Kita,et al.  The effects of the "calcium-antagonist" verapamil on muscle action potentials in the frog and crayfish and on neuromuscular transmission in the crayfish. , 1975, Comparative biochemistry and physiology. C: Comparative pharmacology.