The level of butyrylcholinesterase activity increases and the content of the mRNA remains unaffected in brain of senescence-accelerated mouse SAMP8.

[1]  Xiaorui Cheng,et al.  Differential gene expression profiles in the hippocampus of senescence-accelerated mouse , 2007, Neurobiology of Aging.

[2]  O. Combarros,et al.  Interaction between estrogen receptor-α and butyrylcholinesterase genes modulates Alzheimer’s disease risk , 2007, Journal of Neurology.

[3]  S. Brimijoin,et al.  Differential CSF butyrylcholinesterase levels in Alzheimer's disease patients with the ApoE ε4 allele, in relation to cognitive function and cerebral glucose metabolism , 2006, Neurobiology of Disease.

[4]  O. Livnah,et al.  Butyrylcholinesterase attenuates amyloid fibril formation in vitro. , 2006, Proceedings of the National Academy of Sciences of the United States of America.

[5]  D. Butterfield,et al.  The senescence-accelerated prone mouse (SAMP8): A model of age-related cognitive decline with relevance to alterations of the gene expression and protein abnormalities in Alzheimer's disease , 2005, Experimental Gerontology.

[6]  Y. Nomura,et al.  The age-related degeneration of oligodendrocytes in the hippocampus of the senescence-accelerated mouse (SAM) P8: a quantitative immunohistochemical study. , 2005, Biological & pharmaceutical bulletin.

[7]  E. Giacobini,et al.  Cholinesterase inhibitors: new roles and therapeutic alternatives. , 2004, Pharmacological research.

[8]  Z. Shen,et al.  Brain cholinesterases: II. The molecular and cellular basis of Alzheimer's disease. , 2004, Medical hypotheses.

[9]  J. Mallet,et al.  Expression of PRiMA in the mouse brain: membrane anchoring and accumulation of ‘tailed’ acetylcholinesterase , 2003, The European journal of neuroscience.

[10]  J. Morley,et al.  Cholinergic deficits in the septal–hippocampal pathway of the SAM-P/8 senescence accelerated mouse , 2003, Brain Research.

[11]  C. Geula,et al.  Neurobiology of butyrylcholinesterase , 2003, Nature Reviews Neuroscience.

[12]  F. Campoy,et al.  Muscular dystrophy with laminin deficiency decreases the content of butyrylcholinesterase tetramers in sciatic nerves of Lama2dy mice , 2002, Neuroscience Letters.

[13]  W. Banks,et al.  Alzheimer’s disease through the eye of a mouse Acceptance lecture for the 2001 Gayle A. Olson and Richard D. Olson prize , 2002, Peptides.

[14]  S. Brimijoin,et al.  Abundant Tissue Butyrylcholinesterase and Its Possible Function in the Acetylcholinesterase Knockout Mouse , 2000, Journal of neurochemistry.

[15]  Jose L. Gomez,et al.  Amphiphilic properties of acetylcholinesterase monomers in mouse plasma , 1999, Neuroscience Letters.

[16]  C. J. Vidal,et al.  Molecular forms of acetyl‐ and butyrylcholinesterase in normal and dystrophic mouse brain , 1996, Journal of neuroscience research.

[17]  I. Yokoi,et al.  Neurochemical changes related to ageing in the senescence-accelerated mouse brain and the effect of chronic administration of nimodipine , 1995, Mechanisms of Ageing and Development.

[18]  L. Ignarro,et al.  Nitric oxide as a potential pathological mechanism in demyelination: Its differential effects on primary glial cellsin vitro , 1994, Neuroscience.