Aldose Reductase-Deficient Mice Develop Nephrogenic Diabetes Insipidus
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Mark A. Knepper | Heddwen L. Brooks | Stephen S. M. Chung | S. Tam | M. Knepper | H. Brooks | Sookja K. Chung | B. Ko | Stephen S. M. Chung | Horace T. B. Ho | Janice W. S. Law | Ben C. B. Ko | Sidney C. F. Tam | J. Law | S. S. Chung
[1] D. V. Vander Jagt,et al. Substrate specificity of human aldose reductase: identification of 4-hydroxynonenal as an endogenous substrate. , 1995, Biochimica et biophysica acta.
[2] L. Welt,et al. Defect in the renal tubular reabsorption of water associated with potassium depletion in rats. , 1957, The American journal of physiology.
[3] S. Gapstur,et al. Role of cAMP-phosphodiesterase isozymes in pathogenesis of murine nephrogenic diabetes insipidus. , 1991, The American journal of physiology.
[4] C. Nishimura,et al. The involvement of aldose reductase in diabetic complications. , 1988, Diabetes/metabolism reviews.
[5] F. Epstein,et al. The effect of hypercalcemia induced by calciferol upon renal concentrating ability. , 1958, The Journal of clinical investigation.
[6] J. Gill,et al. On the impairment of renal concentrating ability in prolonged hypercalcemia and hypercalciuria in man. , 1961, The Journal of clinical investigation.
[7] M. M. Bradford. A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding. , 1976, Analytical biochemistry.
[8] H. Hers. Le mécanisme de la transformation de glucose en fructose par les vésicules séminales , 1956 .
[9] M. Knepper. Molecular physiology of urinary concentrating mechanism: regulation of aquaporin water channels by vasopressin. , 1997, The American journal of physiology.
[10] G. Rastelli,et al. Diabetes complications and their potential prevention: Aldose reductase inhibition and other approaches , 1999, Medicinal research reviews.
[11] S. Chung,et al. Identification and Characterization of Multiple Osmotic Response Sequences in the Human Aldose Reductase Gene* , 1997, The Journal of Biological Chemistry.
[12] N. Calcutt,et al. Effects of sorbitol dehydrogenase deficiency on nerve conduction in experimental diabetic mice. , 1998, Diabetes.
[13] C. Yabe-Nishimura,et al. Aldose reductase in glucose toxicity: a potential target for the prevention of diabetic complications. , 1998, Pharmacological reviews.
[14] S. Nielsen,et al. Regulation of collecting duct water channel expression by vasopressin in Brattleboro rat. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[15] D. V. Vander Jagt,et al. Reduction of trioses by NADPH-dependent aldo-keto reductases. Aldose reductase, methylglyoxal, and diabetic complications. , 1992, The Journal of biological chemistry.
[16] P. Burbach,et al. The molecular biology of the Brattleboro rat , 1990 .
[17] K. Sato,et al. Monkey 3-deoxyglucosone reductase: tissue distribution and purification of three multiple forms of the kidney enzyme that are identical with dihydrodiol dehydrogenase, aldehyde reductase, and aldose reductase. , 1993, Archives of biochemistry and biophysics.
[18] K. Jung,et al. ORE, a Eukaryotic Minimal Essential Osmotic Response Element , 1996, The Journal of Biological Chemistry.
[19] A. Manitius,et al. On the mechanism of impairment of renal concentrating ability in potassium deficiency. , 1960, The Journal of clinical investigation.
[20] W. Macdonald. Congenital nephrogenic diabetes insipidus. , 1953, The Medical journal of Australia.
[21] Myron Miller,et al. Renal deficiency associated with diabetes insipidus in the SWR/J mouse , 1975, Physiology & Behavior.
[22] H. Valtin. Hereditary hypothalamic diabetes insipidus in rats (Brattleboro strain). A useful experimental model. , 1967, The American journal of medicine.
[23] N. Copeland,et al. Comparisons of genomic structures and chromosomal locations of the mouse aldose reductase and aldose reductase-like genes. , 2001, European journal of biochemistry.
[24] D. Falconer,et al. Diabetes insipidus associated with oligosyndactyly in the mouse. , 1964 .
[25] L. Feld,et al. Diuretic and natriuretic effects of sorbinil, an aldose reductase inhibitor. , 1991, Pharmacological Research.
[26] R. Balaban,et al. A simple HPLC method for quantitating major organic solutes of renal medulla. , 1989, The American journal of physiology.
[27] S. Nielsen,et al. Vasopressin increases water permeability of kidney collecting duct by inducing translocation of aquaporin-CD water channels to plasma membrane. , 1995, Proceedings of the National Academy of Sciences of the United States of America.
[28] J. Williamson,et al. Glucose-induced metabolic imbalances in the pathogenesis of diabetic vascular disease. , 1991, Diabetes/metabolism reviews.
[29] J. Frøkiaer,et al. Low aquaporin-2 levels in polyuric DI +/+ severe mice with constitutively high cAMP-phosphodiesterase activity. , 1999, American journal of physiology. Renal physiology.
[30] B. V. van Oost,et al. Requirement of human renal water channel aquaporin-2 for vasopressin-dependent concentration of urine. , 1994, Science.
[31] H. Hers. [The mechanism of the transformation of glucose in fructose in the seminal vesicles]. , 1956, Biochimica et biophysica acta.
[32] W. Mcauliffe,et al. Histochemistry and ultrastructure of the interstitium of the renal papilla in rats with hereditary diabetes insipidus (Brattleboro strain). , 1980, The American journal of anatomy.
[33] R. Balaban,et al. Induction of aldose reductase and sorbitol in renal inner medullary cells by elevated extracellular NaCl. , 1987, Proceedings of the National Academy of Sciences of the United States of America.
[34] S. Nielsen,et al. Redistribution of aquaporin-2 water channels induced by vasopressin in rat kidney inner medullary collecting duct. , 1995, The American journal of physiology.
[35] L. Orci,et al. Lack of intramembranous particle clusters in collecting ducts of mice with nephrogenic diabetes insipidus. , 1985, The American journal of physiology.
[36] S. Nielsen,et al. Long-term regulation of four renal aquaporins in rats. , 1996, The American journal of physiology.