Effect of prolonged treatment with amlodipine on enhanced vascular contractility in cardiomyopathic hamsters.
暂无分享,去创建一个
A. Kitabatake | Y. Hattori | H. Okamoto | M. Fukao | M. Kanno | I. Sakuma | M. Watanabe | A. Sato | H. Tomioka | M Fukao
[1] K. Urasawa,et al. Chronic effects of enalapril and amlodipine on cardiac remodeling in cardiomyopathic hamster hearts. , 1998, Journal of cardiovascular pharmacology.
[2] A. Kitabatake,et al. A role of myofilament Ca2+ sensitivity in enhanced vascular reactivity in cardiomyopathic hamsters. , 1998, European journal of pharmacology.
[3] T. Hintze,et al. Amlodipine releases nitric oxide from canine coronary microvessels: an unexpected mechanism of action of a calcium channel-blocking agent. , 1998, Circulation.
[4] A. Kitabatake,et al. Different regulation of myofilament Ca2+ sensitivity in beta-escin-skinned cardiac and vascular smooth muscles. , 1997, European journal of pharmacology.
[5] C M O'Connor,et al. Effect of amlodipine on morbidity and mortality in severe chronic heart failure. Prospective Randomized Amlodipine Survival Evaluation Study Group. , 1996, The New England journal of medicine.
[6] Y. Dohi,et al. Benidipine improves endothelial function in renal resistance arteries of hypertensive rats. , 1996, Hypertension.
[7] D. Lamontagne,et al. Modification of aortic contractility in the cardiomyopathic hamster , 1996, British journal of pharmacology.
[8] H. Thurston,et al. Endothelium-dependent relaxation in resistance arteries from spontaneously hypertensive rats: effect of long-term treatment with perindopril, quinapril, hydralazine or amlodipine , 1996, Journal of hypertension.
[9] K. Urasawa,et al. Enhanced expression of beta-adrenergic receptor kinase 1 in the hearts of cardiomyopathic Syrian hamsters, BIO53.58. , 1996, Biochemical and biophysical research communications.
[10] N. Morel,et al. Selective Interaction of the Calcium Antagonist Amlodipine with Calcium Channels in Arteries of Spontaneously Hypertensive Rats , 1994, Journal of cardiovascular pharmacology.
[11] T. Lüscher,et al. Angiotensin Blockade or Calcium Antagonists Improve Endothelial Dysfunction in Hypertension: Studies in Perfused Mesenteric Resistance Arteries , 1994, Journal of cardiovascular pharmacology.
[12] P. Yock,et al. Differential contribution of nitric oxide to regulation of vascular tone in coronary conductance and resistance arteries: intravascular ultrasound studies. , 1994, American heart journal.
[13] M. Yen,et al. DIFFERENT RESPONSES TO ACETYLCHOLINE IN THE PRESENCE OF NITRIC OXIDE INHIBITOR IN RAT AORTAE AND MESENTERIC ARTERIES , 1993, Clinical and experimental pharmacology & physiology.
[14] W. Mayhan,et al. Acetylcholine induces vasoconstriction in the microcirculation of cardiomyopathic hamsters: reversal by L-arginine. , 1992, Biochemical and biophysical research communications.
[15] Mehra,et al. A prospective, randomized, double-blind, crossover study to compare the efficacy and safety of chronic nifedipine therapy with that of isosorbide dinitrate and their combination in the treatment of chronic congestive heart failure. , 1990, Circulation.
[16] C. Liew,et al. Catecholamines, calcium and cardiomyopathy. , 1988, The American journal of cardiology.
[17] Pankaj Kumar,et al. Calcium Transport Properties of Cardiac Sarcoplasmic Reticulum From Cardiomyopathic Syrian Hamsters (BIO 53.58 and 14.6): Evidence for a Quantitative Defect in Dilated Myopathic Hearts Not Evident in Hypertrophic Hearts , 1988, Circulation research.
[18] E. Sonnenblick,et al. Microvascular reactivity of the myopathic Syrian hamster cremaster muscle. , 1987, Cardiovascular research.
[19] H R Figulla,et al. Inhomogenous capillary flow and its prevention by verapamil and hydralazine in the cardiomyopathic Syrian hamster. , 1987, Circulation.
[20] J. Rouleau,et al. Chronic renal and neurohumoral effects of the calcium entry blocker nisoldipine in patients with congestive heart failure. , 1987, Journal of the American College of Cardiology.
[21] E. Sonnenblick,et al. Microvascular spasm as a cause of cardiomyopathies and the calcium-blocking agent verapamil as potential primary therapy. , 1985, The American journal of cardiology.
[22] J. Elbrink,et al. Increased contractility in vascular smooth muscle of dystrophic hamsters. , 1983, Canadian journal of physiology and pharmacology.
[23] E. Sonnenblick,et al. Microvascular Spasm in the Cardiomyopathic Syrian Hamster: A Preventable Cause of Focal Myocardial Necrosis , 1982, Circulation.
[24] G. Jasmin,et al. Hereditary polymyopathy and cardiomyopathy in the syrian hamster. I. Progression of heart and skeletal muscle lesions in the UM‐X7.1 line , 1982, Muscle & nerve.
[25] D. Wyse. On the "Normalization" of active developed force of isolated helical strips of muscular and elastic arteries for variation in wall thickness. , 1980, Journal of pharmacological methods.
[26] E. Sonnenblick,et al. HEREDITARY AND ACQUIRED CARDIOMYOPATHIES IN EXPERIMENTAL ANIMALS: MECHANICAL, BIOCHEMICAL, AND STRUCTURAL FEATURES * , 1979, Annals of the New York Academy of Sciences.
[27] E. Bajusz,et al. SECTION OF BIOLOGICAL AND MEDICAL SCIENCES: A NEW DISEASE MODEL OF CHRONIC CONGESTIVE HEART FAILURE: STUDIES ON ITS PATHOGENESIS* , 1968 .
[28] D. Murdoch,et al. Amlodipine , 1991, Drugs.