Relatively low levels of calpain expression in juvenile rat correlate with less neuronal apoptosis after spinal cord injury

[1]  Y. Jeong,et al.  Immunohistochemical localization of nerve growth factor, glial fibrillary acidic protein and ciliary neurotrophic factor in mesencephalon, rhombencephalon, and spinal cord of developing mongolian gerbil. , 2002, Journal of veterinary science.

[2]  D. Hovda,et al.  Alterations in BDNF and synapsin I within the occipital cortex and hippocampus after mild traumatic brain injury in the developing rat: reflections of injury-induced neuroplasticity. , 2002, Journal of neurotrauma.

[3]  R. J. Dumont,et al.  Acute Spinal Cord Injury, Part I: Pathophysiologic Mechanisms , 2001, Clinical neuropharmacology.

[4]  B. Bregman,et al.  Differences in Neurotrophic Factor Gene Expression Profiles between Neonate and Adult Rat Spinal Cord after Injury , 2001, Experimental Neurology.

[5]  L. Acarín,et al.  Neuronal, astroglial and microglial cytokine expression after an excitotoxic lesion in the immature rat brain , 2000, The European journal of neuroscience.

[6]  E. Hogan,et al.  Combined TUNEL and double immunofluorescent labeling for detection of apoptotic mononuclear phagocytes in autoimmune demyelinating disease. , 2000, Brain research. Brain research protocols.

[7]  E. Hogan,et al.  Calpain activity and translational expression increased in spinal cord injury , 1999, Brain Research.

[8]  H. Winn,et al.  Methylprednisolone or tirilazad mesylate administration after acute spinal cord injury: 1-year follow up: Results of the third National Acute Spinal Cord Injury Randomized Controlled Trial , 1998 .

[9]  A. Srinivasan,et al.  Apoptosis after traumatic human spinal cord injury. , 1998, Journal of neurosurgery.

[10]  J. Geddes,et al.  Rapid Calpain I Activation and Cytoskeletal Protein Degradation Following Traumatic Spinal Cord Injury: Attenuation with Riluzole Pretreatment , 1997, Journal of neurochemistry.

[11]  N. Banik,et al.  The localization of mcalpain in myelin: immunocytochemical evidence in different areas of rat brain and nerves , 1995, Brain Research.

[12]  E. Hogan,et al.  Role of calpain in spinal cord injury: increased mcalpain immunoreactivity in spinal cord after compression injury in the rat , 1995, Neurochemistry International.

[13]  D. Soppet,et al.  Regulation of neurotrophin receptor expression during embryonic and postnatal development , 1994, The Journal of neuroscience : the official journal of the Society for Neuroscience.

[14]  E. Hogan,et al.  Calcium-activated neutral proteinase (calpain) in rat brain during development: compartmentation and role in myelination. , 1993, Brain research. Developmental brain research.

[15]  L. Belluscio,et al.  NT-3, BDNF, and NGF in the developing rat nervous system: Parallel as well as reciprocal patterns of expression , 1990, Neuron.

[16]  B. Ames,et al.  Decreases in tissue levels of ubiquinol-9 and -10, ascorbate and α-tocopherol following spinal cord impact trauma in rats , 1990, Neuroscience Letters.

[17]  E. Hogan,et al.  Calcium‐activated neutral proteinase in rat brain myelin and subcellular fractions , 1988, Journal of neuroscience research.

[18]  Alfred Reginald Allen,et al.  SURGERY OF EXPERIMENTAL LESION OF SPINAL CORD EQUIVALENT TO CRUSH INJURY OF FRACTURE DISLOCATION OF SPINAL COLUMN: A PRELIMINARY REPORT , 1911 .

[19]  Y. L. Lee,et al.  Chemokine antagonist infusion attenuates cellular infiltration following spinal cord contusion injury in rat , 2000, Journal of neuroscience research.

[20]  J. A. Gruner,et al.  A monitored contusion model of spinal cord injury in the rat. , 1992, Journal of neurotrauma.

[21]  E. Hogan,et al.  Changes in myelin and axonal proteins in CaCl2-induced myelopathy in rat spinal cord. , 1984, Central nervous system trauma : journal of the American Paralysis Association.