Robust Generation of Quiescent Porcine Valvular Interstitial Cell Cultures
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M. Yacoub | N. Latif | K. Masters | C. Bouten | N. C. A. van Engeland | A. Porras | A. McCormack | Evelyn Marchbanks
[1] K. Anseth,et al. Role of cell–matrix interactions on VIC phenotype and tissue deposition in 3D PEG hydrogels , 2016, Journal of tissue engineering and regenerative medicine.
[2] K. Eliceiri,et al. Calcific Aortic Valve Disease Is Associated with Layer-Specific Alterations in Collagen Architecture , 2016, PloS one.
[3] R. Magin,et al. Active tissue stiffness modulation controls valve interstitial cell phenotype and osteogenic potential in 3D culture. , 2016, Acta biomaterialia.
[4] Marcella Trombetta,et al. Cells and extracellular matrix interplay in cardiac valve disease: because age matters , 2016, Basic Research in Cardiology.
[5] L. Wilkins. Development of Aortic Valve Disease in Familial Hypercholesterolemic Swine: Implications for Elucidating Disease Etiology , 2016, Journal of the American Heart Association.
[6] Kristi S Anseth,et al. Microarray analyses to quantify advantages of 2D and 3D hydrogel culture systems in maintaining the native valvular interstitial cell phenotype. , 2016, Biomaterials.
[7] M. Yacoub,et al. Modulation of Human Valve Interstitial Cell Phenotype and Function Using a Fibroblast Growth Factor 2 Formulation , 2015, PloS one.
[8] Magdi H Yacoub,et al. Expression of smooth muscle cell markers and co-activators in calcified aortic valves. , 2015, European heart journal.
[9] Meghan A Bowler,et al. In vitro models of aortic valve calcification: solidifying a system. , 2015, Cardiovascular pathology : the official journal of the Society for Cardiovascular Pathology.
[10] Joshua D. Hutcheson,et al. Directing Valvular Interstitial Cell Myofibroblast‐Like Differentiation in a Hybrid Hydrogel Platform , 2015, Advanced healthcare materials.
[11] L. Leinwand,et al. Cardiac valve cells and their microenvironment—insights from in vitro studies , 2014, Nature Reviews Cardiology.
[12] Guang Yao. Modelling mammalian cellular quiescence , 2014, Interface Focus.
[13] K. Masters,et al. Manipulation of valve composition to elucidate the role of collagen in aortic valve calcification , 2014, BMC Cardiovascular Disorders.
[14] Mark W. Tibbitt,et al. Hydrogels preserve native phenotypes of valvular fibroblasts through an elasticity-regulated PI3K/AKT pathway , 2013, Proceedings of the National Academy of Sciences.
[15] Tom H. Cheung,et al. Molecular regulation of stem cell quiescence , 2013, Nature Reviews Molecular Cell Biology.
[16] A. Gotlieb,et al. The progression of calcific aortic valve disease through injury, cell dysfunction, and disruptive biologic and physical force feedback loops. , 2013, Cardiovascular pathology : the official journal of the Society for Cardiovascular Pathology.
[17] Joshua D. Hutcheson,et al. 5-HT(2B) antagonism arrests non-canonical TGF-β1-induced valvular myofibroblast differentiation. , 2012, Journal of molecular and cellular cardiology.
[18] K. Masters,et al. A time course investigation of the statin paradox among valvular interstitial cell phenotypes. , 2012, American journal of physiology. Heart and circulatory physiology.
[19] K. Billiar,et al. Investigating the role of substrate stiffness in the persistence of valvular interstitial cell activation. , 2012, Journal of biomedical materials research. Part A.
[20] A. Teleman,et al. Insulin/IGF signaling drives cell proliferation in part via Yorkie/YAP. , 2012, Developmental biology.
[21] April M. Kloxin,et al. Redirecting Valvular Myofibroblasts into Dormant Fibroblasts through Light-mediated Reduction in Substrate Modulus , 2012, PloS one.
[22] K. J. Grande-Allen,et al. Calcific Aortic Valve Disease : Not Simply a Degenerative Process A Review and Agenda for Research from the National Heart and Lung and Blood Institute Aortic Stenosis Working Group , 2012 .
[23] K. Masters,et al. Can valvular interstitial cells become true osteoblasts? A side-by-side comparison. , 2011, The Journal of heart valve disease.
[24] Craig A Simmons,et al. Cell–Matrix Interactions in the Pathobiology of Calcific Aortic Valve Disease: Critical Roles for Matricellular, Matricrine, and Matrix Mechanics Cues , 2011, Circulation research.
[25] Craig A Simmons,et al. The aortic valve microenvironment and its role in calcific aortic valve disease. , 2011, Cardiovascular pathology : the official journal of the Society for Cardiovascular Pathology.
[26] Kristyn S Masters,et al. Regulation of valvular interstitial cell calcification by adhesive peptide sequences. , 2010, Journal of biomedical materials research. Part A.
[27] B. Hinz,et al. The myofibroblast: paradigm for a mechanically active cell. , 2010, Journal of biomechanics.
[28] Kristi S Anseth,et al. Characterization of valvular interstitial cell function in three dimensional matrix metalloproteinase degradable PEG hydrogels. , 2009, Biomaterials.
[29] Kristyn S Masters,et al. Regulation of valvular interstitial cell calcification by components of the extracellular matrix. , 2009, Journal of biomedical materials research. Part A.
[30] Craig A Simmons,et al. Calcification by Valve Interstitial Cells Is Regulated by the Stiffness of the Extracellular Matrix , 2009, Arteriosclerosis, thrombosis, and vascular biology.
[31] Craig A Simmons,et al. Identification and characterization of aortic valve mesenchymal progenitor cells with robust osteogenic calcification potential. , 2009, The American journal of pathology.
[32] J. Cleveland,et al. Pro-osteogenic phenotype of human aortic valve interstitial cells is associated with higher levels of Toll-like receptors 2 and 4 and enhanced expression of bone morphogenetic protein 2. , 2009, Journal of the American College of Cardiology.
[33] Kristi S Anseth,et al. Substrate properties influence calcification in valvular interstitial cell culture. , 2008, The Journal of heart valve disease.
[34] A. Gotlieb,et al. Transforming growth factor-beta regulates in vitro heart valve repair by activated valve interstitial cells. , 2008, The American journal of pathology.
[35] K. Anseth,et al. Fibroblast growth factor represses Smad-mediated myofibroblast activation in aortic valvular interstitial cells , 2008, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[36] V. Joag,et al. The emerging role of valve interstitial cell phenotypes in regulating heart valve pathobiology. , 2007, The American journal of pathology.
[37] H. Jakubowski,et al. Letter by Undas and Jakubowski regarding article, "Relationship between homocysteine and mortality in chronic kidney disease". , 2006, Circulation.
[38] M. Amrani,et al. Role of Human Valve Interstitial Cells in Valve Calcification and Their Response to Atorvastatin , 2006, Circulation.
[39] E. Mohler,et al. Paradoxical Effects of Statins on Aortic Valve Myofibroblasts and Osteoblasts: Implications for End-Stage Valvular Heart Disease , 2005, Arteriosclerosis, thrombosis, and vascular biology.
[40] L. Leinwand,et al. Valvular Myofibroblast Activation by Transforming Growth Factor-&bgr;: Implications for Pathological Extracellular Matrix Remodeling in Heart Valve Disease , 2004, Circulation research.
[41] R. Levy,et al. Progression of aortic valve stenosis: TGF-beta1 is present in calcified aortic valve cusps and promotes aortic valve interstitial cell calcification via apoptosis. , 2003, The Annals of thoracic surgery.
[42] M. Yacoub,et al. Phenotypic and functional characterization of interstitial cells from human heart valves, pericardium and skin. , 2000, The Journal of heart valve disease.
[43] C M Johnson,et al. Porcine cardiac valvular subendothelial cells in culture: cell isolation and growth characteristics. , 1987, Journal of molecular and cellular cardiology.