Exposure to p,p'-dichlorodiphenyldichloroethylene (DDE) induces fasting hyperglycemia without insulin resistance in male C57BL/6H mice.

[1]  L. Madsen,et al.  Chronic Consumption of Farmed Salmon Containing Persistent Organic Pollutants Causes Insulin Resistance and Obesity in Mice , 2011, PloS one.

[2]  D. James,et al.  Mapping Insulin/GLUT4 Circuitry , 2011, Traffic.

[3]  L. Mangum,et al.  Exposure to bioaccumulative organochlorine compounds alters adipogenesis, fatty acid uptake, and adipokine production in NIH3T3-L1 cells. , 2011, Toxicology in vitro : an international journal published in association with BIBRA.

[4]  D. Jacobs,et al.  Low Dose Organochlorine Pesticides and Polychlorinated Biphenyls Predict Obesity, Dyslipidemia, and Insulin Resistance among People Free of Diabetes , 2011, PloS one.

[5]  G. Shulman,et al.  Standard operating procedures for describing and performing metabolic tests of glucose homeostasis in mice , 2010, Disease Models & Mechanisms.

[6]  F. Biro,et al.  Childhood obesity and adult morbidities. , 2010, The American journal of clinical nutrition.

[7]  A. Lusis,et al.  Early Hepatic Insulin Resistance Precedes the Onset of Diabetes in Obese C57BLKS-db/db Mice , 2010, Diabetes.

[8]  K. Kristiansen,et al.  Persistent Organic Pollutant Exposure Leads to Insulin Resistance Syndrome , 2009, Environmental health perspectives.

[9]  C. Agardh,et al.  Exposure to p,p′-DDE: A Risk Factor for Type 2 Diabetes , 2009, PloS one.

[10]  A. Klip.  The many ways to regulate glucose transporter 4. , 2009, Applied physiology, nutrition, and metabolism = Physiologie appliquee, nutrition et metabolisme.

[11]  H. Anderson,et al.  Prevalence of diabetes and body burdens of polychlorinated biphenyls, polybrominated diphenyl ethers, and p,p'-diphenyldichloroethene in Great Lakes sport fish consumers. , 2009, Chemosphere.

[12]  H. Anderson,et al.  Organochlorine Exposure and Incidence of Diabetes in a Cohort of Great Lakes Sport Fish Consumers , 2009, Environmental health perspectives.

[13]  S. Caudill,et al.  Levels in the U.S. population of those persistent organic pollutants (2003-2004) included in the Stockholm Convention or in other long range transboundary air pollution agreements. , 2009, Environmental science & technology.

[14]  J. Proietto,et al.  Evaluating the glucose tolerance test in mice. , 2008, American journal of physiology. Endocrinology and metabolism.

[15]  J. Michalek,et al.  Diabetes and Cancer in Veterans of Operation Ranch Hand After Adjustment for Calendar Period, Days of Spraying, and Time Spent in Southeast Asia , 2008, Journal of occupational and environmental medicine.

[16]  J. Wojtaszewski,et al.  Role of Akt substrate of 160 kDa in insulin-stimulated and contraction-stimulated glucose transport. , 2007, Applied physiology, nutrition, and metabolism = Physiologie appliquee, nutrition et metabolisme.

[17]  L. Hagmar,et al.  Exposure to persistent organochlorine pollutants and type 2 diabetes mellitus , 2007, Human & experimental toxicology.

[18]  D. Jacobs,et al.  Association Between Serum Concentrations of Persistent Organic Pollutants and Insulin Resistance Among Nondiabetic Adults , 2007, Diabetes Care.

[19]  D. Jacobs,et al.  A Strong Dose-Response Relation Between Serum Concentrations of Persistent Organic Pollutants and Diabetes , 2006, Diabetes Care.

[20]  C. Kahn,et al.  Divergent regulation of hepatic glucose and lipid metabolism by phosphoinositide 3-kinase via Akt and PKClambda/zeta. , 2006, Cell metabolism.

[21]  J. Michalek,et al.  Insulin sensitivity following agent orange exposure in Vietnam veterans with high blood levels of 2,3,7,8-tetrachlorodibenzo-p-dioxin. , 2004, The Journal of clinical endocrinology and metabolism.

[22]  R. DeFronzo,et al.  Pathogenesis of type 2 diabetes mellitus. , 2004, The Medical clinics of North America.

[23]  M. Rossmeisl,et al.  Variation in type 2 diabetes--related traits in mouse strains susceptible to diet-induced obesity. , 2003, Diabetes.

[24]  D. Ross,et al.  Diet-induced obesity and hepatic gene expression alterations in C57BL/6J and ICAM-1-deficient mice. , 2002, American journal of physiology. Endocrinology and metabolism.

[25]  É. Hajduch,et al.  Protein kinase B (PKB/Akt) – a key regulator of glucose transport? , 2001, FEBS letters.

[26]  J. Friedman,et al.  Decreased Akt kinase activity and insulin resistance in C57BL/KsJ-Leprdb/db mice. , 2000, The Journal of endocrinology.

[27]  A. Klip,et al.  GLUT-4myc ectopic expression in L6 myoblasts generates a GLUT-4-specific pool conferring insulin sensitivity. , 1999, American journal of physiology. Endocrinology and metabolism.

[28]  R B Conolly,et al.  Transplacental and lactational transfer of p,p'-DDE in Sprague-Dawley rats. , 1999, Toxicology and applied pharmacology.

[29]  J. Zierath,et al.  Insulin-Stimulated Akt Kinase Activity Is Reduced in Skeletal Muscle From NIDDM Subjects , 1998, Diabetes.

[30]  M. Pepe,et al.  Predicting obesity in young adulthood from childhood and parental obesity. , 1997, The New England journal of medicine.

[31]  R. DeFronzo.  Insulin resistance: a multifaceted syndrome responsible for NIDDM, obesity, hypertension, dyslipidaemia and atherosclerosis. , 1997, The Netherlands journal of medicine.

[32]  I. Yamaguchi,et al.  Development of hyperglycaemia and insulin resistance in conscious genetically diabetic (C57BL/KsJ-db/db) mice , 1994, Diabetologia.

[33]  A. Porras,et al.  The ras signaling pathway mimics insulin action on glucose transporter translocation. , 1993, Proceedings of the National Academy of Sciences of the United States of America.

[34]  D. Jacobs,et al.  Associations of organochlorine pesticides and polychlorinated biphenyls in visceral vs. subcutaneous adipose tissue with type 2 diabetes and insulin resistance. , 2014, Chemosphere.

[35]  K. Flegal,et al.  Prevalence of obesity in the United States, 2009-2010. , 2012, NCHS data brief.

[36]  L. Hagmar,et al.  Environmental Health: a Global Access Science Source a Cross-sectional Study of the Association between Persistent Organochlorine Pollutants and Diabetes , 2005 .

[37]  F. Matsumura,et al.  Differential effects of 2,3,7,8-tetrachlorodibenzo-p-dioxin on the "adipose- type" and "brain-type" glucose transporters in mice. , 1995, Molecular pharmacology.

[38]  P. Wyss,et al.  Kinetics of distribution and elimination of DDE in rats. , 1991, Xenobiotica; the fate of foreign compounds in biological systems.