Early afterdepolarizations and cardiac arrhythmias.

[1]  Alan Garfinkel,et al.  So little source, so much sink: requirements for afterdepolarizations to propagate in tissue. , 2010, Biophysical journal.

[2]  J. Weiss,et al.  Irregularly appearing early afterdepolarizations in cardiac myocytes: random fluctuations or dynamical chaos? , 2010, Biophysical journal.

[3]  Lai-Hua Xie,et al.  Arrhythmogenic consequences of intracellular calcium waves. , 2009, American journal of physiology. Heart and circulatory physiology.

[4]  Diana X. Tran,et al.  Bifurcation and chaos in a model of cardiac early afterdepolarizations. , 2009, Physical review letters.

[5]  Diana X. Tran,et al.  Synchronization of chaotic early afterdepolarizations in the genesis of cardiac arrhythmias , 2009, Proceedings of the National Academy of Sciences.

[6]  G. Billman,et al.  Repolarization abnormalities and afterdepolarizations in a canine model of sudden cardiac death. , 2008, American journal of physiology. Regulatory, integrative and comparative physiology.

[7]  Dan M. Roden,et al.  Clinical practice. Long-QT syndrome. , 2008, The New England journal of medicine.

[8]  Zhongju Lu,et al.  Xenografted Adult Human Mesenchymal Stem Cells Provide a Platform for Sustained Biological Pacemaker Function in Canine Heart , 2007, Circulation.

[9]  Andrey Shilnikov,et al.  Origin of bursting through homoclinic spike adding in a neuron model. , 2007, Physical review letters.

[10]  Yoram Rudy,et al.  Subunit Interaction Determines IKs Participation in Cardiac Repolarization and Repolarization Reserve , 2005, Circulation.

[11]  R. Lazzara,et al.  Triggered firing in pulmonary veins initiated by in vitro autonomic nerve stimulation. , 2005, Heart rhythm.

[12]  Andrey Shilnikov,et al.  Transition between tonic spiking and bursting in a neuron model via the blue-sky catastrophe. , 2005, Physical review letters.

[13]  G. Tomaselli,et al.  What Causes Sudden Death in Heart Failure? , 2004, Circulation research.

[14]  Boris Kogan,et al.  Intracellular calcium cycling, early afterdepolarizations, and reentry in simulated long QT syndrome. , 2004, Heart rhythm.

[15]  D. Rosenbaum,et al.  Transmural Electrophysiological Heterogeneities Underlying Arrhythmogenesis in Heart Failure , 2003, Circulation research.

[16]  Colleen E Clancy,et al.  Non-Equilibrium Gating in Cardiac Na+ Channels: An Original Mechanism of Arrhythmia , 2003, Circulation.

[17]  Hua-rong Lu,et al.  Drug-induced long QT in isolated rabbit Purkinje fibers: importance of action potential duration, triangulation and early afterdepolarizations. , 2002, European journal of pharmacology.

[18]  Guy Salama,et al.  Cytosolic Ca2+ triggers early afterdepolarizations and torsade de pointes in rabbit hearts with type 2 long QT syndrome , 2002, The Journal of physiology.

[19]  C. Antzelevitch,et al.  Unique Topographical Distribution of M Cells Underlies Reentrant Mechanism of Torsade de Pointes in the Long-QT Syndrome , 2002, Circulation.

[20]  Junko Kurokawa,et al.  Requirement of a Macromolecular Signaling Complex for β Adrenergic Receptor Modulation of the KCNQ1-KCNE1 Potassium Channel , 2002, Science.

[21]  G. Duker,et al.  Instability and Triangulation of the Action Potential Predict Serious Proarrhythmia, but Action Potential Duration Prolongation Is Antiarrhythmic , 2001, Circulation.

[22]  Robert C. Hilborn,et al.  Chaos and Nonlinear Dynamics , 2000 .

[23]  H. Wellens,et al.  Progress in the understanding of cardiac early afterdepolarizations and torsades de pointes: time to revise current concepts. , 2000, Cardiovascular research.

[24]  N. Peters,et al.  Myocardial architecture and ventricular arrhythmogenesis. , 1998, Circulation.

[25]  A. Castellanos,et al.  Interpretation of outcomes of antiarrhythmic clinical trials: design features and population impact. , 1998, Circulation.

[26]  R Lazzara,et al.  Similarities between early and delayed afterdepolarizations induced by isoproterenol in canine ventricular myocytes. , 1997, Cardiovascular research.

[27]  J Jalife,et al.  Optical mapping of drug-induced polymorphic arrhythmias and torsade de pointes in the isolated rabbit heart. , 1997, Journal of the American College of Cardiology.

[28]  Bertram Pitt,et al.  Effect of d-sotalol on mortality in patients with left ventricular dysfunction after recent and remote myocardial infarction , 1996, The Lancet.

[29]  R. Gilmour,et al.  Triggered activity as a mechanism for inherited ventricular arrhythmias in German shepherd Dogs. , 1996, Journal of the American College of Cardiology.

[30]  A. Panfilov,et al.  Nonstationary vortexlike reentrant activity as a mechanism of polymorphic ventricular tachycardia in the isolated rabbit heart. , 1995, Circulation.

[31]  C. Luo,et al.  A dynamic model of the cardiac ventricular action potential. II. Afterdepolarizations, triggered activity, and potentiation. , 1994, Circulation research.

[32]  P B Corr,et al.  Mechanisms underlying early and delayed afterdepolarizations induced by catecholamines. , 1990, The American journal of physiology.

[33]  J E Saffitz,et al.  Distribution and Three‐Dimensional Structure of Intercellular Junctions in Canine Myocardium , 1989, Circulation research.

[34]  C. N. D'ALNONCOURT,et al.  "Torsade de pointes" tachycardia. Re-entry or focal activity? , 1982, British heart journal.

[35]  H A Fozzard,et al.  Strength—duration curves in cardiac Purkinje fibres: effects of liminal length and charge distribution , 1972, The Journal of physiology.

[36]  A. L. Wit,et al.  Conduction of the Cardiac Impulse , 1972, The Journal of general physiology.

[37]  S. Weidmann,et al.  Effect of current flow on the membrane potential of cardiac muscle , 1951, The Journal of physiology.

[38]  C. Napolitano,et al.  Torsade de Pointes , 2012, Drugs.

[39]  Joseph L Greenstein,et al.  The role of stochastic and modal gating of cardiac L-type Ca2+ channels on early after-depolarizations. , 2005, Biophysical journal.

[40]  M. Weir,et al.  The Cardiac Arrhythmia Suppression Trial Investigators: Preliminary Report: Effect of Encainide and Flecainide on Mortality in a Randomized Trial of Arrhythmia Suppression After Myocardial Infarction. , 1990 .

[41]  W. Rogers,et al.  Preliminary report: effect of encainide and flecainide on mortality in a randomized trial of arrhythmia suppression after myocardial infarction. , 1989, The New England journal of medicine.

[42]  A. Resmini.  The conduction of the cardiac impulse : P. F. Cranefeld, Futura Publ. Co., Mount Kisko, N.Y. (1975), 404 pp., $ 27.50. , 1977 .