Ciliary Neurotrophic Factor Inhibits Brain and Peripheral Tumor Necrosis Factor Production and, When Coadministered with Its Soluble Receptor, Protects Mice From Lipopolysaccharide Toxicity

[1]  P. Waring,et al.  Leukemia inhibitory factor protects against experimental lethal Escherichia coli septic shock in mice. , 1995, Proceedings of the National Academy of Sciences of the United States of America.

[2]  K. Chien,et al.  Expression cloning of cardiotrophin 1, a cytokine that induces cardiac myocyte hypertrophy. , 1995, Proceedings of the National Academy of Sciences of the United States of America.

[3]  G. Fantuzzi,et al.  Ciliary neurotrophic factor (CNTF) induces serum amyloid A, hypoglycaemia and anorexia, and potentiates IL-1 induced corticosterone and IL-6 production in mice. , 1995, Cytokine.

[4]  L. Shapiro,et al.  Ciliary Neurotrophic Factor Combined with Soluble Receptor Inhibits Synthesis of Proinflammatory Cytokines and Prostaglandin-E2in Vitro , 1994 .

[5]  Moses Rodriguez,et al.  Recombinant human IL-6 suppresses demyelination in a viral model of multiple sclerosis. , 1994, Journal of immunology.

[6]  Peter Brouckaer.  Protection by alpha 1-acid glycoprotein against tumor necrosis factor- induced lethality , 1994, The Journal of experimental medicine.

[7]  J. Williams,et al.  Intratracheal injection of LPS and cytokines. V. LPS induces expression of LIF and LIF inhibits acute inflammation. , 1994, The American journal of physiology.

[8]  G. Fantuzzi,et al.  Defective inflammatory response in interleukin 6-deficient mice , 1994, The Journal of experimental medicine.

[9]  M. Noble,et al.  N-acetyl-L-cysteine is a pluripotent protector against cell death and enhancer of trophic factor-mediated cell survival in vitro. , 1994, Proceedings of the National Academy of Sciences of the United States of America.

[10]  A. Acheson,et al.  Recombinant human CNTF receptor alpha: production, binding stoichiometry, and characterization of its activity as a diffusible factor. , 1994, Biochemistry.

[11]  D. Samols,et al.  Attenuation of complement-mediated acute lung injury in rabbits and transgenic mice by C-reactive protein. , 1994, Chest.

[12]  P. Distefano,et al.  Released form of CNTF receptor alpha component as a soluble mediator of CNTF responses. , 1993, Science.

[13]  R. Faggioni,et al.  Role of tumour necrosis factor and reactive oxygen intermediates in lipopolysaccharide‐induced pulmonary oedema and lethality , 1993, Clinical and experimental immunology.

[14]  A. Billiau,et al.  Protective effect of anti‐interleukin (IL)‐6 antibody against endotoxin, associated with paradoxically increased IL‐6 levels , 1992, European journal of immunology.

[15]  David J. Anderson,et al.  CNTF and LIF act on neuronal cells via shared signaling pathways that involve the IL-6 signal transducing receptor component gp130 , 1992, Cell.

[16]  J. Norton,et al.  Differentiation factor/leukemia inhibitory factor protection against lethal endotoxemia in mice: synergistic effect with interleukin 1 and tumor necrosis factor , 1992, The Journal of experimental medicine.

[17]  J. Gauldie,et al.  Recombinant oncostatin M stimulates the production of acute phase proteins in HepG2 cells and rat primary hepatocytes in vitro. , 1992, Journal of immunology.

[18]  C. Dinarello,et al.  Interleukin-6 as an endogenous pyrogen: induction of prostaglandin E2 in brain but not in peripheral blood mononuclear cells , 1991, Brain Research.

[19]  F. Lottspeich,et al.  Recombinant Human and Rat Ciliary Neurotrophic Factors , 1991, Journal of neurochemistry.

[20]  M. Nyska,et al.  Acute Phase Protein, Serum Amyloid A, Inhibits IL‐1‐ and TNF‐Induced Fever and Hypothalamic PGE2 in Mice , 1991, Scandinavian journal of immunology.

[21]  L. Chedid,et al.  Influence of endogenous glucocorticoid on endotoxin-induced production of circulating TNF-alpha. , 1991, Lymphokine and cytokine research.

[22]  R. Neta,et al.  Interleukin-1 and interleukin-6 act synergistically to stimulate the release of adrenocorticotropic hormone in vivo. , 1991, Lymphokine and cytokine research.

[23]  D. Mason.  Genetic variation in the stress response: susceptibility to experimental allergic encephalomyelitis and implications for human inflammatory disease. , 1991, Immunology today.

[24]  J. Sipe,et al.  Direct binding enzyme-linked immunosorbent assay (ELISA) for serum amyloid A (SAA). , 1989, Journal of immunological methods.

[25]  J. Vilček,et al.  IL-6 inhibits lipopolysaccharide-induced tumor necrosis factor production in cultured human monocytes, U937 cells, and in mice. , 1989, Journal of immunology.

[26]  H. Baumann,et al.  Hepatocyte-stimulating factor III shares structural and functional identity with leukemia-inhibitory factor. , 1989, Journal of immunology.

[27]  D. Mason,et al.  Spontaneous recovery of rats from experimental allergic encephalomyelitis is dependent on regulation of the immune system by endogenous adrenal corticosteroids , 1989, The Journal of experimental medicine.

[28]  P. Ghezzi,et al.  Adrenalectomy sensitizes mice to the lethal effects of interleukin 1 and tumor necrosis factor , 1988, The Journal of experimental medicine.

[29]  C. Dinarello,et al.  Immunoregulatory feedback between interleukin-1 and glucocorticoid hormones. , 1986, Science.

[30]  D. Goeddel,et al.  Human tumor necrosis factor. Production, purification, and characterization. , 1985, The Journal of biological chemistry.

[31]  P. Campbell,et al.  Enzyme changes in neonatal skeletal muscle: effect of thyroid deficiency. , 1978, The American journal of physiology.

[32]  D. Rodbard,et al.  Simultaneous analysis of families of sigmoidal curves: application to bioassay, radioligand assay, and physiological dose-response curves. , 1978, The American journal of physiology.

[33]  T. Haley,et al.  Pharmacological effects produced by intracerebral injection of drugs in the conscious mouse. , 1957, British journal of pharmacology and chemotherapy.

[34]  G. Piazza,et al.  Lysophosphatidic Acid Induction of Transforming Growth Factors α and β: Modulation of Proliferation and Differentiation in Cultured Human Keratinocytes and Mouse Skin , 1995 .

[35]  G. Fantuzzi,et al.  Chlorpromazine specifically inhibits peripheral and brain TNF production, and up-regulates IL-10 production, in mice. , 1994, Immunology.

[36]  K. Tracey,et al.  Tumor necrosis factor, other cytokines and disease. , 1993, Annual review of cell biology.

[37]  J. M. Lipton,et al.  Central administration of the peptide alpha-MSH inhibits inflammation in the skin. , 1991, Peptides.

[38]  J. Gauldie,et al.  Interferon f 32 / B-cell stimulatory factor type 2 shares identity with monocyte-derived hepatocyte-stimulating factor and regulates the major acute phase protein response in liver cells ( inflammation / acute phase reactants / gene regulation ) , 2022 .