Induction of Placental Heme Oxygenase-1 Is Protective Against TNFα-induced Cytotoxicity and Promotes Vessel Relaxation
暂无分享,去创建一个
Asif Ahmed | B. Bussolati | J. S. St John | I. Rushton | Xian Zhang | Mahbubur Rahman | S. Nijjar | C. Acevedo | J. S. St. John
[1] P. Baker,et al. Relationship of Cell Adhesion Molecule Expression to Endothelium-Dependent Relaxation in Normal Pregnancy and Pregnancies Complicated With Preeclampsia or Fetal Growth Restriction , 1999, The Journal of the Society for Gynecologic Investigation: JSGI.
[2] L. Kobzik,et al. Hypoxia induces severe right ventricular dilatation and infarction in heme oxygenase-1 null mice. , 1999, The Journal of clinical investigation.
[3] A. Choi,et al. Exogenous administration of heme oxygenase-1 by gene transfer provides protection against hyperoxia-induced lung injury. , 1999, The Journal of clinical investigation.
[4] I. Sargent,et al. Preeclampsia: an excessive maternal inflammatory response to pregnancy. , 1999, American journal of obstetrics and gynecology.
[5] D. Faller. ENDOTHELIAL CELL RESPONSES TO HYPOXIC STRESS , 1999, Clinical and experimental pharmacology & physiology.
[6] L. Turka,et al. Antibody-induced transplant arteriosclerosis is prevented by graft expression of anti-oxidant and anti-apoptotic genes , 1998, Nature Medicine.
[7] K. Furuyama,et al. Developmental changes of gene expression in heme metabolic enzymes in rat placenta , 1998, FEBS letters.
[8] C. Schneeberger,et al. Expression of endothelial (type III) nitric oxide synthase in cytotrophoblastic cell lines: regulation by hypoxia and inflammatory cytokines. , 1998, Placenta.
[9] C. Graham,et al. Heme oxygenase and nitric oxide synthase in the placenta of the guinea-pig during gestation. , 1998, Placenta.
[10] Q. Chen,et al. Cytotoxicity and apoptosis produced by arachidonic acid in HepG2 cells overexpressing human cytochrome P-4502E1. , 1998, Alcoholism, clinical and experimental research.
[11] I. Johnson,et al. Plasma from pre‐eclamptic women and functional change in myometrial resistance arteries , 1998, British journal of obstetrics and gynaecology.
[12] A. Ahmed,et al. Hemeoxygenase-1 inhibits human myometrial contractility via carbon monoxide and is upregulated by progesterone during pregnancy. , 1998, The Journal of clinical investigation.
[13] Martin Whittle,et al. Cellular localization of AT1 receptor mRNA and protein in normal placenta and its reduced expression in intrauterine growth restriction. Angiotensin II stimulates the release of vasorelaxants. , 1998, The Journal of clinical investigation.
[14] H. Schröder,et al. The nitric oxide donor SIN-1 protects endothelial cells from tumor necrosis factor-alpha-mediated cytotoxicity: possible role for cyclic GMP and heme oxygenase. , 1997, Journal of molecular and cellular cardiology.
[15] N. Abraham,et al. Heme Induces the Expression of Adhesion Molecules ICAM-1, VCAM-1, and E Selectin in Vascular Endothelial Cells , 1997, Proceedings of the Society for Experimental Biology and Medicine. Society for Experimental Biology and Medicine.
[16] Rui Wang,et al. Carbon monoxide‐induced vasorelaxation and the underlying mechanisms , 1997, British journal of pharmacology.
[17] A. Cederbaum,et al. Cytotoxicity and Apoptosis Produced by Arachidonic Acid in Hep G2 Cells Overexpressing Human Cytochrome P4502E1* , 1997, The Journal of Biological Chemistry.
[18] D. Kniss,et al. Role of VEGF receptor-1 (Flt-1) in mediating calcium-dependent nitric oxide release and limiting DNA synthesis in human trophoblast cells. , 1997, Laboratory investigation; a journal of technical methods and pathology.
[19] I. Greer,et al. Inducible (type II) nitric oxide synthase in human placental villous tissue of normotensive, pre-eclamptic and intrauterine growth-restricted pregnancies. , 1997, Placenta.
[20] S. Moncada. Nitric Oxide in the Vasculature: Physiology and Pathophysiology , 1997, Annals of the New York Academy of Sciences.
[21] W. Durante,et al. Nitric oxide induces heme oxygenase-1 gene expression and carbon monoxide production in vascular smooth muscle cells. , 1997, Circulation research.
[22] S. Ferry,et al. Cardiovascular sequelae of toxaemia of pregnancy. , 1997, Heart.
[23] S. Walsh,et al. TNFα concentrations and mRNA expression are increased in preeclamptic placentas , 1996 .
[24] N. Gorbunov,et al. Nitric oxide/carbon monoxide. A molecular switch for myocardial preservation during ischemia. , 1996, Circulation.
[25] R. A. Johnson,et al. Heme oxygenase substrates acutely lower blood pressure in hypertensive rats. , 1996, The American journal of physiology.
[26] U. Ekblad,et al. Lipid peroxidation and antioxidants in normal and pre-eclamptic pregnancies. , 1996, Placenta.
[27] T. Nakaki,et al. Nitric oxide in vascular remodeling. , 1996, Japanese heart journal.
[28] A. Jain,et al. Role of bilirubin in pregnancy‐induced hypertension , 1996, International journal of gynaecology and obstetrics: the official organ of the International Federation of Gynaecology and Obstetrics.
[29] R. A. Rutherford,et al. Nitric oxide synthase in human placenta and umbilical cord from normal, intrauterine growth‐retarded and pre‐eclamptic pregnancies , 1995, British journal of pharmacology.
[30] A. Choi,et al. Hemoglobin provides protection against lethal endotoxemia in rats: the role of heme oxygenase-1. , 1995, American journal of respiratory cell and molecular biology.
[31] R. Arngrímsson,et al. Death rates from ischemic heart disease in women with a history of hypertension in pregnancy , 1995 .
[32] K. Vandegriff,et al. Oxidative-stress response in vascular endothelial cells exposed to acellular hemoglobin solutions. , 1995, The American journal of physiology.
[33] M. Gerritsen,et al. Transfection of the human heme oxygenase gene into rabbit coronary microvessel endothelial cells: protective effect against heme and hemoglobin toxicity. , 1995, Proceedings of the National Academy of Sciences of the United States of America.
[34] S. Kourembanas,et al. Smooth muscle cell-derived carbon monoxide is a regulator of vascular cGMP. , 1995, Proceedings of the National Academy of Sciences of the United States of America.
[35] S. Kostyk,et al. Hypoxia inhibits expression of eNOS via transcriptional and posttranscriptional mechanisms. , 1994, The American journal of physiology.
[36] K. Pavelić,et al. Molecular Medicine , 1986, British Journal of Cancer.
[37] J. Polak,et al. Endothelial nitric oxide synthase in the human placenta: regional distribution and proposed regulatory role at the feto-maternal interface. , 1994, Placenta.
[38] L. Myatt,et al. Vasodilator Effects of Parathyroid Hormone, Parathyroid Hormone-Related Protein, and Calcitonin Gene-Related Peptide in the Human Fetal-Placental Circulation , 1994, The Journal of the Society for Gynecologic Investigation: JSGI.
[39] James M. Roberts,et al. Preeclampsia: More Than Pregnancy‐Induced Hypertension , 1994 .
[40] J. Pollock,et al. Immunohistochemical localization of nitric oxide synthase in the human placenta. , 1993, Placenta.
[41] James M. Roberts,et al. Pre-eclampsia: more than pregnancy-induced hypertension , 1993, The Lancet.
[42] G. Vercellotti,et al. Induction of heme oxygenase is a rapid, protective response in rhabdomyolysis in the rat. , 1992, The Journal of clinical investigation.
[43] K. Laderoute,et al. The identification of heme oxygenase as a major hypoxic stress protein in Chinese hamster ovary cells. , 1991, British Journal of Cancer.
[44] L. Myatt,et al. The action of nitric oxide in the perfused human fetal-placental circulation. , 1991, American journal of obstetrics and gynecology.
[45] S. Brennecke,et al. Role of endothelium-derived nitric oxide in maintenance of low fetal vascular resistance in placenta , 1990, The Lancet.
[46] S. Pecorelli,et al. Histological features of uteroplacental vessels in normal and hypertensive patients in relation to birthweight , 1989, British journal of obstetrics and gynaecology.
[47] M. Maines. Heme oxygenase: function, multiplicity, regulatory mechanisms, and clinical applications , 1988, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[48] S. Shibahara,et al. Human heme oxygenase cDNA and induction of its mRNA by hemin. , 1988, European journal of biochemistry.
[49] B. Brüne,et al. Inhibition of platelet aggregation by carbon monoxide is mediated by activation of guanylate cyclase. , 1987, Molecular pharmacology.
[50] B. Ames,et al. Antioxidant activity of albumin-bound bilirubin. , 1987, Proceedings of the National Academy of Sciences of the United States of America.
[51] E. Beutler,et al. Simplified determination of carboxyhemoglobin. , 1984, Clinical chemistry.
[52] W. B. Robertson,et al. FETAL GROWTH RETARDATION AND THE ARTERIES OF THE PLACENTAL BED , 1977, British journal of obstetrics and gynaecology.
[53] F. Reilly,et al. Neurohistochemical evidence supporting an absence of adrenergic and cholinergic innervation in the human placenta and umbilical cord , 1977, The Anatomical record.
[54] Y. Niida,et al. Oxidative stress causes enhanced endothelial cell injury in human heme oxygenase-1 deficiency. , 1999, The Journal of clinical investigation.
[55] N. Abraham,et al. Gene transfer of human heme oxygenase into coronary endothelial cells potentially promotes angiogenesis , 1998, Journal of cellular biochemistry.
[56] D. Mann,et al. Tumor Necrosis Factor- (cid:97) Confers Resistance to Hypoxic Injury in the Adult Mammalian Cardiac Myocyte , 2022 .
[57] D. Willis,et al. Heme oxygenase: A novel target for the modulation of inflammatory response , 1996, Nature Medicine.
[58] S. Walsh,et al. TNF alpha concentrations and mRNA expression are increased in preeclamptic placentas. , 1996, Journal of reproductive immunology.
[59] G. Vercellotti,et al. Endothelial cell heme oxygenase and ferritin induction by heme proteins: a possible mechanism limiting shock damage. , 1992, Transactions of the Association of American Physicians.
[60] L. Myatt,et al. Attenuation of the vasoconstrictor effects of thromboxane and endothelin by nitric oxide in the human fetal-placental circulation. , 1992, American journal of obstetrics and gynecology.