A HerpesSimplex VirusType1MutantContaining a Nontransinducing Vmw65Protein Establishes Latent Infection InVivointheAbsenceofViral Replication andReactivates Efficiently fromExplanted Trigeminal Ganglia
暂无分享,去创建一个
[1] W. Flanagan,et al. Identification of the latency-associated transcript promoter by expression of rabbit beta-globin mRNA in mouse sensory nerve ganglia latently infected with a recombinant herpes simplex virus , 1989, Journal of virology.
[2] P. Schaffer,et al. A deletion mutant of the latency-associated transcript of herpes simplex virus type 1 reactivates from the latent state with reduced frequency , 1989, Journal of virology.
[3] D. Altmann,et al. Herpes simplex virus latency. , 1984, Lancet.
[4] S. McKnight,et al. Expression of a truncated viral trans-activator selectively impedes lytic infection by its cognate virus , 1988, Nature.
[5] R. Roeder,et al. A herpesvirus trans-activating protein interacts with transcription factor OTF-1 and other cellular proteins. , 1988, Proceedings of the National Academy of Sciences of the United States of America.
[6] M. Ostrander,et al. MTX5: a cell line expressing biologically active HSV-1 Vmw65 protein. , 1988, Nucleic acids research.
[7] B. Roizman,et al. Host cell proteins bind to the cis-acting site required for virion-mediated induction of herpes simplex virus 1 alpha genes. , 1987, Proceedings of the National Academy of Sciences of the United States of America.
[8] P. Schaffer,et al. Herpes simplex virus type 1 ICP27 is an essential regulatory protein , 1985, Journal of virology.
[9] J. Subak-Sharpe,et al. Latency competence of thirteen HSV-1 temperature-sensitive mutants. , 1980, The Journal of general virology.