Botulinum toxin type A reduces hyperalgesia and TRPV1 expression in rats with neuropathic pain.
暂无分享,去创建一个
Jianguo Cheng | Lizu Xiao | W. Qu | Jeffery J. Muir | Deren Zhang | Yuanyan Zhuang | Haowen Liang | Zhuang Yu
[1] K. Aoki,et al. Updates on the antinociceptive mechanism hypothesis of botulinum toxin A. , 2011, Parkinsonism & related disorders.
[2] Jianguo Cheng,et al. Botulinum toxin decreases hyperalgesia and inhibits P2X3 receptor over-expression in sensory neurons induced by ventral root transection in rats. , 2011, Pain medicine.
[3] E. Park,et al. Effect of botulinum toxin on expression of nerve growth factor and transient receptor potential vanilloid 1 in urothelium and detrusor muscle of rats with bladder outlet obstruction-induced detrusor overactivity. , 2011, Urology.
[4] S. Mackey,et al. Subcutaneous injection of botulinum toxin a is beneficial in postherpetic neuralgia. , 2010, Pain medicine.
[5] M. Salković-Petrisic,et al. Botulinum toxin type A reduces pain supersensitivity in experimental diabetic neuropathy: bilateral effect after unilateral injection. , 2010, European journal of pharmacology.
[6] P. Chabrier,et al. Different antinociceptive effects of botulinum toxin type A in inflammatory and peripheral polyneuropathic rat models. , 2009, European journal of pharmacology.
[7] I. Tseng,et al. Botulinum toxin for diabetic neuropathic pain , 2009, Neurology.
[8] S. Mackey,et al. Sympathetic block with botulinum toxin to treat complex regional pain syndrome , 2009, Annals of neurology.
[9] L. Arendt-Nielsen,et al. Subcutaneous Botulinum toxin type A reduces capsaicin-induced trigeminal pain and vasomotor reactions in human skin , 2009, PAIN®.
[10] F. Morain,et al. Botulinum toxin type a induces direct analgesic effects in chronic neuropathic pain , 2008, Annals of neurology.
[11] C. M. Flores,et al. Critical evaluation of the colocalization between calcitonin gene-related peptide, substance P, transient receptor potential vanilloid subfamily type 1 immunoreactivities, and isolectin B4 binding in primary afferent neurons of the rat and mouse. , 2007, The journal of pain : official journal of the American Pain Society.
[12] Ji-Tian Xu,et al. Role of phosphorylation of ERK in induction and maintenance of LTP of the C‐fiber evoked field potentials in spinal dorsal horn , 2006, Journal of neuroscience research.
[13] R. Paus,et al. Hair cycle control by vanilloid receptor-1 (TRPV1): evidence from TRPV1 knockout mice. , 2006, The Journal of investigative dermatology.
[14] Ji-Tian Xu,et al. The role of tumor necrosis factor-alpha in the neuropathic pain induced by Lumbar 5 ventral root transection in rat , 2006, PAIN.
[15] L. Arendt-Nielsen,et al. The effects of Botulinum Toxin type A on capsaicin-evoked pain, flare, and secondary hyperalgesia in an experimental human model of trigeminal sensitization , 2006, Pain.
[16] D. Clapham,et al. An introduction to TRP channels. , 2006, Annual review of physiology.
[17] K. Aoki. Review of a proposed mechanism for the antinociceptive action of botulinum toxin type A. , 2005, Neurotoxicology.
[18] D. Cockayne,et al. Decreased sensory receptors P2X3 and TRPV1 in suburothelial nerve fibers following intradetrusor injections of botulinum toxin for human detrusor overactivity. , 2005, The Journal of urology.
[19] Bernd Nilius,et al. TRP channels: a TR(I)P through a world of multifunctional cation channels , 2005, Pflügers Archiv.
[20] R. Alp,et al. Botulinum Toxin and Intractable Trigeminal Neuralgia , 2005, Clinical neuropharmacology.
[21] R. Schwabe,et al. Anandamide induces necrosis in primary hepatic stellate cells , 2005, Hepatology.
[22] R. Paus,et al. A hot new twist to hair biology: involvement of vanilloid receptor-1 (VR1/TRPV1) signaling in human hair growth control. , 2005, The American journal of pathology.
[23] P. Reeh,et al. Morphological evidence for functional capsaicin receptor expression and calcitonin gene-related peptide exocytosis in isolated peripheral nerve axons of the mouse , 2004, Neuroscience.
[24] A. Ferrer-Montiel,et al. Regulated Exocytosis Contributes to Protein Kinase C Potentiation of Vanilloid Receptor Activity* , 2004, Journal of Biological Chemistry.
[25] F. Scaravilli,et al. Parallel changes in bladder suburothelial vanilloid receptor TRPV1 and pan‐neuronal marker PGP9.5 immunoreactivity in patients with neurogenic detrusor overactivity after intravesical resiniferatoxin treatment , 2004, BJU international.
[26] M. Schumacher,et al. Expression of vanilloid receptor subtype 1 in cutaneous sensory nerve fibers, mast cells, and epithelial cells of appendage structures , 2004, Experimental dermatology.
[27] K. Aoki,et al. Subcutaneous administration of botulinum toxin A reduces formalin-induced pain , 2004, Pain.
[28] A. Ford,et al. P2X3-immunoreactive nerve fibres in neurogenic detrusor overactivity and the effect of intravesical resiniferatoxin. , 2003, European urology.
[29] P. Reeh,et al. Proton‐induced calcitonin gene‐related peptide release from rat sciatic nerve axons, in vitro, involving TRPV1 , 2003, The European journal of neuroscience.
[30] O. Dolly. Synaptic Transmission: Inhibition of Neurotransmitter Release by Botulinum Toxins , 2003, Headache.
[31] L. Sorkin,et al. Tumor Necrosis Factor-α Induces Mechanical Allodynia after Spinal Nerve Ligation by Activation of p38 MAPK in Primary Sensory Neurons , 2003, The Journal of Neuroscience.
[32] C. Argoff. A Focused Review on the Use of Botulinum Toxins for Neuropathic Pain , 2002, The Clinical journal of pain.
[33] C. Woolf,et al. p38 MAPK Activation by NGF in Primary Sensory Neurons after Inflammation Increases TRPV1 Levels and Maintains Heat Hyperalgesia , 2002, Neuron.
[34] Xin-Fu Zhou,et al. Effect of Lumbar 5 Ventral Root Transection on Pain Behaviors: A Novel Rat Model for Neuropathic Pain without Axotomy of Primary Sensory Neurons , 2002, Experimental Neurology.
[35] R. Meyer,et al. Mechanical hyperalgesia after an L5 ventral rhizotomy or an L5 ganglionectomy in the rat , 2002, Pain.
[36] S. Koizumi,et al. Functional vanilloid receptors in cultured normal human epidermal keratinocytes. , 2002, Biochemical and biophysical research communications.
[37] G. Burnstock. Purine-mediated signalling in pain and visceral perception. , 2001, Trends in pharmacological sciences.
[38] C. Woolf,et al. Spared nerve injury: an animal model of persistent peripheral neuropathic pain , 2000, Pain.
[39] P. Blumberg,et al. Distribution of mRNA for vanilloid receptor subtype 1 (VR1), and VR1-like immunoreactivity, in the central nervous system of the rat and human. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[40] P. Blumberg,et al. Vanilloid (Capsaicin) receptors and mechanisms. , 1999, Pharmacological reviews.
[41] R. Elde,et al. Immunocytochemical localization of the vanilloid receptor 1 (VR1): relationship to neuropeptides, the P2X3 purinoceptor and IB4 binding sites , 1999, The European journal of neuroscience.
[42] D. Julius,et al. The capsaicin receptor: a heat-activated ion channel in the pain pathway , 1997, Nature.
[43] K. Moore,et al. Distribution and colocalization of calcitonin gene-related peptide, tachykinins, and vasoactive intestinal peptide in normal and idiopathic unstable human urinary bladder. , 1997, Laboratory investigation; a journal of technical methods and pathology.
[44] T. Yaksh,et al. Quantitative assessment of tactile allodynia in the rat paw , 1994, Journal of Neuroscience Methods.
[45] P. Holzer. Capsaicin: cellular targets, mechanisms of action, and selectivity for thin sensory neurons. , 1991, Pharmacological reviews.
[46] R. Dubner,et al. A new and sensitive method for measuring thermal nociception in cutaneous hyperalgesia , 1987, Pain.
[47] M. Zimmermann,et al. Ethical guidelines for investigations of experimental pain in conscious animals , 1983, Pain.