Increased risk of non-Q wave myocardial infarction after directional atherectomy is platelet dependent: evidence from the EPIC trial. Evaluation of c7E3 for the Prevention of Ischemic Complications.
暂无分享,去创建一个
R. Califf | E. Topol | K. Anderson | H. Weisman | J. Blankenship | J. Lefkovits | S. Worley | J. Talley | G. Stoner | James Blankenship | David Talley | f’GAIL L. Stoner | Iieric J. Topol
[1] E. Topol,et al. Defining the appropriate threshold of creatine kinase elevation after percutaneous coronary interventions. , 1996, American heart journal.
[2] R. Califf,et al. Characteristics and consequences of myocardial infarction after percutaneous coronary intervention: insights from the Coronary Angioplasty Versus Excisional Atherectomy Trial (CAVEAT). , 1995, Journal of the American College of Cardiology.
[3] R. Califf,et al. A multicenter, randomized trial of coronary angioplasty versus directional atherectomy for patients with saphenous vein bypass graft lesions. CAVEAT-II Investigators. , 1995, Circulation.
[4] P. Yock,et al. 730-2 Acute Angiographic, Intravascular Ultrasound and Clinical Results of Directional Atherectomy in the Optimal Atherectomy Restenosis Study , 1995 .
[5] D. Baim,et al. Elevation of the creatine kinase myocardial isoform following otherwise successful directional coronary atherectomy and stenting. , 1994, The American journal of cardiology.
[6] V. Fuster,et al. Effect of an Eccentric Severe Stenosis on Fibrin(ogen) Deposition on Severely Damaged Vessel Wall in Arterial Thrombosis: Relative Contribution of Fibrin(ogen) and Platelets , 1994, Circulation.
[7] D. Altieri,et al. Differential ligand binding specificities of recombinant CD11b/CD18 integrin I-domain. , 1994, The Journal of biological chemistry.
[8] V. Fuster,et al. Characterization of the relative thrombogenicity of atherosclerotic plaque components: implications for consequences of plaque rupture. , 1994, Journal of the American College of Cardiology.
[9] R. Califf,et al. Randomised trial of coronary intervention with antibody against platelet IIb/IIIa iritegrin for reduction of clinical restenosis: results at six months , 1994, The Lancet.
[10] Epic Investigators,et al. Use of a monoclonal antibody directed against the platelet glycoprotein IIb/IIIa receptor in high-risk coronary angioplasty. , 1994, The New England journal of medicine.
[11] D. Baim,et al. Complications of directional coronary atherectomy: incidence, causes, and management. , 1993, The American journal of cardiology.
[12] M. Cowley,et al. Experience with directional coronary atherectomy since pre-market approval. , 1993, The American journal of cardiology.
[13] E. Topol,et al. Results of directional coronary atherectomy during multicenter preapproval testing. The US Directional Coronary Atherectomy Investigator Group. , 1993, The American journal of cardiology.
[14] D. Ricci,et al. A comparison of directional atherectomy with balloon angioplasty for lesions of the left anterior descending coronary artery. , 1993, The New England journal of medicine.
[15] R. Califf,et al. A Comparison of Directional Atherectomy with Coronary Angioplasty in Patients with Coronary Artery Disease , 1993 .
[16] S. Schnitt,et al. Long-term results of directional coronary atherectomy: predictors of restenosis. , 1992, Journal of the American College of Cardiology.
[17] M. Bell,et al. Results of directional atherectomy of primary atheromatous and restenosis lesions in coronary arteries and saphenous vein grafts. , 1992, The American journal of cardiology.
[18] E. Topol,et al. Abrupt vessel closure after directional coronary atherectomy , 1992 .
[19] J. Simpson,et al. Effect of lesion characteristics on outcome of directional coronary atherectomy. , 1991, Journal of the American College of Cardiology.
[20] L. Klein,et al. Incidence and clinical significance of transient creatine kinase elevations and the diagnosis of non-Q wave myocardial infarction associated with coronary angioplasty. , 1991, Journal of the American College of Cardiology.
[21] E J Topol,et al. Coronary morphologic and clinical determinants of procedural outcome with angioplasty for multivessel coronary disease. Implications for patient selection. Multivessel Angioplasty Prognosis Study Group. , 1990, Circulation.
[22] R D Safian,et al. Coronary atherectomy. Clinical, angiographic, and histological findings and observations regarding potential mechanisms. , 1990, Circulation.
[23] V. Fuster,et al. Platelet thrombus formation on collagen type I. A model of deep vessel injury. Influence of blood rheology, von Willebrand factor, and blood coagulation. , 1988, Circulation.
[24] P. Mannucci,et al. Oligospecificity of the cellular adhesion receptor Mac-1 encompasses an inducible recognition specificity for fibrinogen , 1988, The Journal of cell biology.
[25] A. Jaffe,et al. Thrombolysis in myocardial infarction (TIMI) trial—Phase I: Hemorrhagic manifestations and changes in plasma fibrinogen and the fibrinolytic system in patients treated with recombinant tissue plasminogen activator and streptokinase , 1988 .
[26] A. Jaffe,et al. Thrombolysis in Myocardial Infarction (TIMI) Trial--phase I: hemorrhagic manifestations and changes in plasma fibrinogen and the fibrinolytic system in patients treated with recombinant tissue plasminogen activator and streptokinase. , 1988, Journal of the American College of Cardiology.
[27] I. Charo,et al. Platelet glycoprotein IIb-IIIa-like proteins mediate endothelial cell attachment to adhesive proteins and the extracellular matrix. , 1987, The Journal of biological chemistry.
[28] J. Oh,et al. Creatine kinase release after successful percutaneous transluminal coronary angioplasty. , 1985, American heart journal.