Bradykinin inhibits a potassium M‐like current in rat pheochromocytoma PC12 cells

[1]  D. A. Brown,et al.  On the transduction mechanism for muscarine‐induced inhibition of M‐current in cultured rat sympathetic neurones. , 1989, The Journal of physiology.

[2]  B. Hille,et al.  Protein kinase C is not necessary for peptide-induced suppression of M current or for desensitization of the peptide receptors. , 1989, Proceedings of the National Academy of Sciences of the United States of America.

[3]  J F Storm,et al.  An after‐hyperpolarization of medium duration in rat hippocampal pyramidal cells. , 1989, The Journal of physiology.

[4]  F. Di Virgilio,et al.  Inhibitors of membrane transport system for organic anions block fura-2 excretion from PC12 and N2A cells. , 1988, The Biochemical journal.

[5]  J. Meldolesi,et al.  Generation of inositol phosphates, cytosolic Ca2+, and ionic fluxes in PC12 cells treated with bradykinin. , 1988, The Journal of biological chemistry.

[6]  E. Neher,et al.  Intracellular calcium release mediated by two muscarinic receptor subtypes , 1988, FEBS letters.

[7]  M. Mishina,et al.  Selective coupling with K+ currents of muscarinic acetylcholine receptor subtypes in NG108-15 cells , 1988, Nature.

[8]  P. Pfaffinger.  Muscarine and t-LHRH suppress M-current by activating an IAP- insensitive G-protein , 1988, The Journal of neuroscience : the official journal of the Society for Neuroscience.

[9]  W. Almers,et al.  Agonists that suppress M-current elicit phosphoinositide turnover and Ca2+ transients, but these events do not explain M-current suppression , 1988, Neuron.

[10]  R. Nicoll,et al.  Stimulation of phosphatidylinositol (PI) turnover may mediate the muscarinic suppression of the M-current in hippocampal pyramidal cells , 1988, Neuroscience Letters.

[11]  P. Brehm,et al.  Selective induction of brain type II Na+ channels by nerve growth factor. , 1988, Proceedings of the National Academy of Sciences of the United States of America.

[12]  R. Aldrich,et al.  Voltage-dependent K+ currents and underlying single K+ channels in pheochromocytoma cells , 1988, The Journal of general physiology.

[13]  H. Higashida,et al.  Bradykinin inhibits potassium (M) currents in N1E‐ 115 neuroblastoma cells Responses resemble those in NG108‐15 neuroblastoma x glioma hybrid cells , 1987, FEBS letters.

[14]  A. Constanti,et al.  Calcium‐dependent potassium conductance in guinea‐pig olfactory cortex neurones in vitro. , 1987, The Journal of physiology.

[15]  L. Tertoolen,et al.  Electrophysiological responses to bradykinin and microinjected inositol polyphosphates in neuroblastoma cells , 1987, FEBS letters.

[16]  H. Higashida,et al.  Membrane current responses to intracellular injections of inositol 1,3,4,5‐tetrakisphosphate and inositol 1,3,4‐trisphosphate in NG108‐15 hybrid cells , 1986, FEBS letters.

[17]  S. Halegoua,et al.  Inhibition of growth factor-induced differentiation of PC12 cells by microinjection of antibody to ras p21 , 1986, Nature.

[18]  F. Di Virgilio,et al.  Muscarinic receptor-induced phosphoinositide hydrolysis at resting cytosolic Ca2+ concentration in PC12 cells , 1985, The Journal of cell biology.

[19]  R. Tsien,et al.  A new generation of Ca2+ indicators with greatly improved fluorescence properties. , 1985, The Journal of biological chemistry.

[20]  A. Tischler,et al.  Regulation of muscarinic ligand binding sites by nerve growth factor in PC12 phaeochromocytoma cells , 1982, Nature.

[21]  D. A. Brown,et al.  Muscarinic suppression of a novel voltage-sensitive K+ current in a vertebrate neurone , 1980, Nature.

[22]  L. Greene,et al.  Nerve growth factor-induced increase in electrical excitability and acetylcholine sensitivity of a rat pheochromocytoma cell line , 1977, Nature.

[23]  H. Higashida,et al.  Two polyphosphatidylinositide metabolites control two K+ currents in a neuronal cell , 1986, Nature.