A presynaptic inositol-5-phosphatase

[1]  Brian J. Stevenson,et al.  end5, end6, and end7: mutations that cause actin delocalization and block the internalization step of endocytosis in Saccharomyces cerevisiae. , 1995, Molecular biology of the cell.

[2]  A. Klippel,et al.  Evidence for phosphatidylinositol 3-kinase as a regulator of endocytosis via activation of Rab5. , 1995, Proceedings of the National Academy of Sciences of the United States of America.

[3]  C. Mitchell,et al.  Phosphatidylinositol 3,4,5‐trisphosphate is a substrate for the 75 kDa inositol polyphosphate 5‐phosphatase and a novel 5‐phosphatase which forms a complex with the p85/p110 form of phosphoinositide 3‐kinase. , 1995, The EMBO journal.

[4]  W P Tate,et al.  Translational termination efficiency in mammals is influenced by the base following the stop codon. , 1995, Proceedings of the National Academy of Sciences of the United States of America.

[5]  P. Majerus,et al.  The protein deficient in Lowe syndrome is a phosphatidylinositol-4,5-bisphosphate 5-phosphatase. , 1995, Proceedings of the National Academy of Sciences of the United States of America.

[6]  Richard G. W. Anderson,et al.  The Appendage Domain of α-Adaptin Is a High Affinity Binding Site for Dynamin (*) , 1995, The Journal of Biological Chemistry.

[7]  T. Takenawa,et al.  ATP-dependent inositide phosphorylation required for Ca2+-activated secretion , 1995, Nature.

[8]  M. Bembenek,et al.  Inhibition of Clathrin Assembly by High Affinity Binding of Specific Inositol Polyphosphates to the Synapse-specific Clathrin Assembly Protein AP-3 (*) , 1995, The Journal of Biological Chemistry.

[9]  P. Majerus,et al.  Inositol Hexakisphosphate Binds to Clathrin Assembly Protein 3 (AP-3/AP180) and Inhibits Clathrin Cage Assembly in Vitro(*) , 1995, The Journal of Biological Chemistry.

[10]  R. Llinás,et al.  The inositol high-polyphosphate series blocks synaptic transmission by preventing vesicular fusion: a squid giant synapse study. , 1994, Proceedings of the National Academy of Sciences of the United States of America.

[11]  K. Mikoshiba,et al.  Synaptotagmin is an inositol polyphosphate binding protein: isolation and characterization as an Ins 1,3,4,5-P4 binding protein. , 1994, Biochemical and biophysical research communications.

[12]  P. De Camilli,et al.  p145, a major Grb2-binding protein in brain, is co-localized with dynamin in nerve terminals where it undergoes activity-dependent dephosphorylation. , 1994, The Journal of biological chemistry.

[13]  R. Greenspan,et al.  Distinct protein forms are produced from alternatively spliced bicistronic glutamic acid decarboxylase mRNAs during development , 1994, Molecular and cellular biology.

[14]  P. De Camilli,et al.  Autoimmunity in Stiff‐Man Syndrome with breast cancer is targeted to the C‐terminal region of human amphiphysin, a protein similar to the yeast proteins, Rvs167 and Rvs161 , 1994, FEBS letters.

[15]  P. Greengard,et al.  Interaction of Grb2 via its Src homology 3 domains with synaptic proteins including synapsin I. , 1994, Proceedings of the National Academy of Sciences of the United States of America.

[16]  T. Tetaz,et al.  Characterization of a cDNA encoding the 43-kDa membrane-associated inositol-polyphosphate 5-phosphatase. , 1994, The Journal of biological chemistry.

[17]  A. Cleves,et al.  SAC1p is an integral membrane protein that influences the cellular requirement for phospholipid transfer protein function and inositol in yeast , 1993, The Journal of cell biology.

[18]  B. Voss,et al.  SAP90, a rat presynaptic protein related to the product of the Drosophila tumor suppressor gene dlg-A. , 1993, The Journal of biological chemistry.

[19]  D. Baltimore,et al.  Identification of a protein that binds to the SH3 region of Abl and is similar to Bcr and GAP-rho. , 1992, Science.

[20]  R. Nussbaum,et al.  The Lowe's oculocerebrorenal syndrome gene encodes a protein highly homologous to inositol polyphosphate-5-phosphatase , 1992, Nature.

[21]  M. Kozak,et al.  An analysis of vertebrate mRNA sequences: intimations of translational control , 1991, The Journal of cell biology.

[22]  P. Majerus,et al.  Cloning and expression of human 75-kDa inositol polyphosphate-5-phosphatase. , 1991, The Journal of biological chemistry.

[23]  P. Seeburg,et al.  RNA editing in brain controls a determinant of ion flow in glutamate-gated channels , 1991, Cell.

[24]  A. Cleves,et al.  An essential role for a phospholipid transfer protein in yeast Golgi function , 1990, Nature.

[25]  A. Cleves,et al.  Mutations in the SAC1 gene suppress defects in yeast Golgi and yeast actin function , 1989, The Journal of cell biology.

[26]  D Botstein,et al.  Suppressors of yeast actin mutations. , 1989, Genetics.

[27]  R. Schekman,et al.  Identification of 23 complementation groups required for post-translational events in the yeast secretory pathway , 1980, Cell.

[28]  S. Emr,et al.  Receptor-mediated protein sorting to the vacuole in yeast: roles for a protein kinase, a lipid kinase and GTP-binding proteins. , 1995, Annual review of cell and developmental biology.