Human perifollicular macrophages undergo apoptosis, express Wnt ligands and switch their polarisation during catagen.

[1]  Y. Tokura,et al.  M2 macrophages promote wound-induced hair neogenesis. , 2018, Journal of dermatological science.

[2]  Young‐Hee Kang,et al.  Chrysin Inhibits Advanced Glycation End Products-Induced Kidney Fibrosis in Renal Mesangial Cells and Diabetic Kidneys , 2018, Nutrients.

[3]  J. Shapiro,et al.  Alopecia areata: Disease characteristics, clinical evaluation, and new perspectives on pathogenesis. , 2018, Journal of the American Academy of Dermatology.

[4]  K. Dev,et al.  Macrophages: Their role, activation and polarization in pulmonary diseases , 2017, Immunobiology.

[5]  S. Biswas,et al.  Metabolic regulation of macrophage phenotype and function , 2017, Immunological reviews.

[6]  R. Paus,et al.  Epithelial-to-Mesenchymal Stem Cell Transition in a Human Organ: Lessons from Lichen Planopilaris. , 2017, The Journal of investigative dermatology.

[7]  R. Paus,et al.  Characterisation of cell cycle arrest and terminal differentiation in a maximally proliferative human epithelial tissue: Lessons from the human hair follicle matrix. , 2017, European journal of cell biology.

[8]  F. Deng,et al.  Wnt signaling pathway protein LEF1 in cancer, as a biomarker for prognosis and a target for treatment. , 2017, American journal of cancer research.

[9]  M. Rose,et al.  Hair and stress: A pilot study of hair and cytokine balance alteration in healthy young women under major exam stress , 2017, PloS one.

[10]  R. Paus,et al.  Vasoactive intestinal peptide, whose receptor‐mediated signalling may be defective in alopecia areata, provides protection from hair follicle immune privilege collapse , 2016, The British journal of dermatology.

[11]  R. Paus,et al.  Human hair follicle organ culture: theory, application and perspectives , 2015, Experimental dermatology.

[12]  L. Beljaars,et al.  The Elusive Antifibrotic Macrophage , 2015, Front. Med..

[13]  B. Andersen,et al.  Resting no more: re‐defining telogen, the maintenance stage of the hair growth cycle , 2015, Biological reviews of the Cambridge Philosophical Society.

[14]  Jung Chul Kim,et al.  A guide to studying human hair follicle cycling in vivo , 2015, The Journal of investigative dermatology.

[15]  R. Paus,et al.  Re-Evaluating Cyclosporine A as a Hair Growth-Promoting Agent in Human Scalp Hair Follicles. , 2015, The Journal of investigative dermatology.

[16]  Wei Hu,et al.  Advanced Glycation End Products Enhance Macrophages Polarization into M1 Phenotype through Activating RAGE/NF-κB Pathway , 2015, BioMed research international.

[17]  P. Cohen,et al.  The PPAR-γ antagonist GW9662 elicits differentiation of M2c-like cells and upregulation of the MerTK/Gas6 axis: a key role for PPAR-γ in human macrophage polarization , 2015, Journal of Inflammation.

[18]  J. Yu,et al.  Overexpression of the Receptor for Advanced Glycation Endproducts (RAGE) Is Associated with Poor Prognosis in Gastric Cancer , 2015, PloS one.

[19]  K. Blagoev,et al.  Niche induced cell death and epithelial phagocytosis regulate hair follicle stem cell pool , 2015, Nature.

[20]  R. Paus,et al.  Macrophages Contribute to the Cyclic Activation of Adult Hair Follicle Stem Cells , 2014, PLoS biology.

[21]  J. Uribarri,et al.  Advanced glycation end product accumulation: a new enemy to target in chronic kidney disease? , 2014, Current opinion in nephrology and hypertension.

[22]  P. Reynolds,et al.  Primary alveolar macrophages exposed to diesel particulate matter increase RAGE expression and activate RAGE signaling , 2014, Cell and Tissue Research.

[23]  R. Paus,et al.  Human epithelial hair follicle stem cells and their progeny: Current state of knowledge, the widening gap in translational research and future challenges , 2014, BioEssays : news and reviews in molecular, cellular and developmental biology.

[24]  P. Rovere-Querini,et al.  Macrophage Plasticity in Skeletal Muscle Repair , 2014, BioMed research international.

[25]  S. Gordon,et al.  The M1 and M2 paradigm of macrophage activation: time for reassessment , 2014, F1000prime reports.

[26]  B. Kempkes,et al.  Macrophage Polarisation: an Immunohistochemical Approach for Identifying M1 and M2 Macrophages , 2013, PloS one.

[27]  R. Paus,et al.  Lichen planopilaris is characterized by immune privilege collapse of the hair follicle's epithelial stem cell niche , 2013, The Journal of pathology.

[28]  R. Paus,et al.  The immune system of mouse vibrissae follicles: cellular composition and indications of immune privilege , 2013, Experimental dermatology.

[29]  R. Paus,et al.  Loss of γδ T Cells Results in Hair Cycling Defects. , 2013, The Journal of investigative dermatology.

[30]  R. Nau,et al.  Palmitoylethanolamide stimulates phagocytosis of Escherichia coli K1 and Streptococcus pneumoniae R6 by microglial cells , 2012, Journal of Neuroimmunology.

[31]  R. Ramasamy,et al.  Receptor for AGE (RAGE): signaling mechanisms in the pathogenesis of diabetes and its complications , 2011, Annals of the New York Academy of Sciences.

[32]  G. Arismendi-Morillo,et al.  Ultrastructural Characterization of Macrophage-like Mononuclear Leukocytes in Human Astrocytic Tumors , 2010, Ultrastructural pathology.

[33]  R. Paus,et al.  Methods in hair research: how to objectively distinguish between anagen and catagen in human hair follicle organ culture , 2010, Experimental dermatology.

[34]  S. Gordon,et al.  Alternative activation of macrophages: an immunologic functional perspective. , 2009, Annual review of immunology.

[35]  Ralf Paus,et al.  The Hair Follicle as a Dynamic Miniorgan , 2009, Current Biology.

[36]  R. Paus,et al.  Maintenance of hair follicle immune privilege is linked to prevention of NK cell attack. , 2008, The Journal of investigative dermatology.

[37]  K. Basler,et al.  Wntless, a Conserved Membrane Protein Dedicated to the Secretion of Wnt Proteins from Signaling Cells , 2006, Cell.

[38]  A. Schmidt,et al.  RAGE is a multiligand receptor of the immunoglobulin superfamily: implications for homeostasis and chronic disease , 2002, Cellular and Molecular Life Sciences CMLS.

[39]  I. Hassinen,et al.  Further insight into mechanism of action of clodronate: inhibition of mitochondrial ADP/ATP translocase by a nonhydrolyzable, adenine-containing metabolite. , 2002, Molecular pharmacology.

[40]  R Paus,et al.  A comprehensive guide for the accurate classification of murine hair follicles in distinct hair cycle stages. , 2001, The Journal of investigative dermatology.

[41]  D. Tobin,et al.  The human hair follicle immune system: cellular composition and immune privilege , 2000, The British journal of dermatology.

[42]  R Paus,et al.  The biology of hair follicles. , 1999, The New England journal of medicine.

[43]  T. Imamura,et al.  Localization of rat FGF-5 protein in skin macrophage-like cells and FGF-5S protein in hair follicle: possible involvement of two Fgf-5 gene products in hair growth cycle regulation. , 1998, The Journal of investigative dermatology.

[44]  S. Eichmüller,et al.  Generation and cyclic remodeling of the hair follicle immune system in mice. , 1998, The Journal of investigative dermatology.

[45]  B. Hermes,et al.  „Vernarbende” Alopezien Anmerkungen zur Klassifikation, Differentialdiagnose und Pathobiologie , 1998, Der Hautarzt.

[46]  R. Paus,et al.  Mast cells as modulators of hair follicle cycling , 1995, Experimental dermatology.

[47]  T. Kealey,et al.  Human hair growth in vitro: a model for the study of hair follicle biology. , 1994, Journal of dermatological science.

[48]  A. Messenger,et al.  Cyclosporin A prolongs human hair growth in vitro. , 1993, The Journal of investigative dermatology.

[49]  A. Orekhov,et al.  The impact of interferon-regulatory factors to macrophage differentiation and polarization into M1 and M2. , 2018, Immunobiology.

[50]  P. Murray Macrophage Polarization. , 2017, Annual review of physiology.

[51]  R. Paus,et al.  A 'hairy' privilege. , 2005, Trends in immunology.

[52]  P. Parakkal Morphogenesis of the hair follicle during catagen , 2004, Zeitschrift für Zellforschung und Mikroskopische Anatomie.