Erythrocyte G Protein-Coupled Receptor Signaling in Malarial Infection
暂无分享,去创建一个
Travis Harrison | Kasturi Haldar | H. Hamm | K. Haldar | N. Mohandas | J. Lomasney | Narla Mohandas | T. Harrison | Jon W Lomasney | Benjamin U Samuel | Thomas Akompong | Heidi Hamm | T. Akompong | B. Samuel | Travis Harrison
[1] E. Yoon,et al. Thrombin Receptors Activate Go Proteins in Endothelial Cells to Regulate Intracellular Calcium and Cell Shape Changes* , 2002, The Journal of Biological Chemistry.
[2] E. Brown,et al. Role of Cholesterol in Formation and Function of a Signaling Complex Involving αvβ3, Integrin-Associated Protein (Cd47), and Heterotrimeric G Proteins , 1999, The Journal of cell biology.
[3] T. Kenakin,et al. Physiological effects of inverse agonists in transgenic mice with myocardial overexpression of the β2-adrenoceptor , 1995, Nature.
[4] K. Haldar,et al. Vacuolar uptake of host components, and a role for cholesterol and sphingomyelin in malarial infection , 2000, The EMBO journal.
[5] W. Peters,et al. The chemotherapy of rodent malaria, XXXVII. The in vivo action of two Mannich bases, WR 194,965 and WR 228,258 and an 8-aminoquinoline WR 225,448. , 1984, Annals of tropical medicine and parasitology.
[6] K. Day,et al. Expression of Plasmodium falciparum trimeric G proteins and their involvement in switching to sexual development. , 2000, Molecular and biochemical parasitology.
[7] Schrier Sl. Red cell membrane biology--introduction. , 1985 .
[8] N. Mohandas,et al. Membrane assembly and remodeling during reticulocyte maturation , 1989 .
[9] P. Schnabel,et al. Characterization of β1‐selectivity, adrenoceptor‐Gs‐protein interaction and inverse agonism of nebivolol in human myocardium , 2001, British journal of pharmacology.
[10] T. Dunwiddie,et al. Antagonists of the Receptor-G Protein Interface Block Gi-coupled Signal Transduction* , 1998, The Journal of Biological Chemistry.
[11] Jonathan E. Allen,et al. Genome sequence of the human malaria parasite Plasmodium falciparum , 2002, Nature.
[12] R. Rosenberg,et al. Pore size of the malaria parasite's nutrient channel. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[13] W. Whitehouse,et al. Invasion of erythrocytes by malaria merozoites. , 1975, Science.
[14] K. Seydel,et al. Detergent-resistant erythrocyte membrane rafts are modified by a Plasmodium falciparum infection. , 2002, Experimental parasitology.
[15] Terry Kenakin,et al. Drug efficacy at G protein-coupled receptors. , 2002, Annual review of pharmacology and toxicology.
[16] Prahlad T. Ram,et al. G Protein Pathways , 2002, Science.
[17] K. Haldar,et al. The Role of Cholesterol and Glycosylphosphatidylinositol-anchored Proteins of Erythrocyte Rafts in Regulating Raft Protein Content and Malarial Infection* , 2001, The Journal of Biological Chemistry.
[18] Heidi E. Hamm,et al. The Many Faces of G Protein Signaling* , 1998, The Journal of Biological Chemistry.
[19] H. Hamm,et al. A Dominant-Negative Strategy for Studying Roles of G Proteins in Vivo * , 1999, The Journal of Biological Chemistry.
[20] L. Parise,et al. Activation of sickle red blood cell adhesion via integrin-associated protein/CD47-induced signal transduction. , 2001, The Journal of clinical investigation.
[21] Ogobara K. Doumbo,et al. The pathogenic basis of malaria , 2002, Nature.