Interferon γ Induces Upregulation and Activation of Caspases 1, 3, and 8 to Produce Apoptosis in Human Erythroid Progenitor Cells
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[1] J. Price,et al. Fas ligand is present in human erythroid colony-forming cells and interacts with Fas induced by interferon gamma to produce erythroid cell apoptosis. , 1998, Blood.
[2] A. Strasser,et al. Fas ligand—cought between scylla and charybdis , 1998, Nature Medicine.
[3] V. Kidd,et al. Proteolytic activities that mediate apoptosis. , 1998, Annual review of physiology.
[4] Aseem Kumar,et al. Defective TNF-α-Induced Apoptosis in STAT1-Null Cells Due to Low Constitutive Levels of Caspases , 1997 .
[5] M. Peter,et al. Advances in apoptosis research. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[6] G. Salvesen,et al. Caspases: Intracellular Signaling by Proteolysis , 1997, Cell.
[7] M. Peter,et al. FLICE Is Predominantly Expressed as Two Functionally Active Isoforms, Caspase-8/a and Caspase-8/b* , 1997, The Journal of Biological Chemistry.
[8] J. Price,et al. Interferon γ Downregulates Stem Cell Factor and Erythropoietin Receptors But Not Insulin-Like Growth Factor-I Receptors in Human Erythroid Colony-Forming Cells , 1997 .
[9] R. Flavell,et al. Activation of the STAT signaling pathway can cause expression of caspase 1 and apoptosis , 1997, Molecular and cellular biology.
[10] G M Cohen,et al. Caspases: the executioners of apoptosis. , 1997, The Biochemical journal.
[11] N. Thornberry,et al. Caspases: killer proteases. , 1997, Trends in biochemical sciences.
[12] M. Bondurant,et al. The roles of Bcl-X(L) and apopain in the control of erythropoiesis by erythropoietin. , 1997, Blood.
[13] P. Fitzpatrick,et al. Interferon-γ Modulates a p53-independent Apoptotic Pathway and Apoptosis-related Gene Expression* , 1997, The Journal of Biological Chemistry.
[14] C. Dai,et al. Polycythemia vera. V. Enhanced proliferation and phosphorylation due to vanadate are diminished in polycythemia vera erythroid progenitor cells: a possible defect of phosphatase activity in polycythemia vera. , 1997, Blood.
[15] Matthias Mann,et al. FLICE is activated by association with the CD95 death‐inducing signaling complex (DISC) , 1997, The EMBO journal.
[16] S. Nagata,et al. Apoptosis by Death Factor , 1997, Cell.
[17] N. Thornberry,et al. Control of apoptosis by proteases. , 1997, Advances in pharmacology.
[18] G. Evan,et al. A License to Kill , 1996, Cell.
[19] David Wallach,et al. Involvement of MACH, a Novel MORT1/FADD-Interacting Protease, in Fas/APO-1- and TNF Receptor–Induced Cell Death , 1996, Cell.
[20] M. Raff,et al. Role of Ced-3/ICE-family proteases in staurosporine-induced programmed cell death , 1996, The Journal of cell biology.
[21] G. Cohen,et al. Benzyloxycarbonyl-Val-Ala-Asp (OMe) fluoromethylketone (Z-VAD.FMK) inhibits apoptosis by blocking the processing of CPP32. , 1996, The Biochemical journal.
[22] S. Orrenius,et al. CPP32/Apopain Is a Key Interleukin 1 Converting Enzyme-like Protease Involved in Fas-mediated Apoptosis (*) , 1996, The Journal of Biological Chemistry.
[23] J. Price,et al. Stem cell factor can overcome inhibition of highly purified human burst‐forming units‐erythroid by interferon γ , 1995, Journal of cellular physiology.
[24] M. Su,et al. Cleavage of Poly(ADP-ribose) Polymerase by Interleukin-1β Converting Enzyme and Its Homologs TX and Nedd-2 (*) , 1995, The Journal of Biological Chemistry.
[25] N. Young,et al. Fas antigen expression on CD34+ human marrow cells is induced by interferon gamma and tumor necrosis factor alpha and potentiates cytokine-mediated hematopoietic suppression in vitro. , 1995, Blood.
[26] J. Camonis,et al. A Novel Protein That Interacts with the Death Domain of Fas/APO1 Contains a Sequence Motif Related to the Death Domain (*) , 1995, The Journal of Biological Chemistry.
[27] J. Inazawa,et al. Human Fas ligand: gene structure, chromosomal location and species specificity. , 1994, International immunology.
[28] S. Marsters,et al. Inhibition of murine erythroid colony formation in vitro by interferon gamma and correction by interferon receptor immunoadhesin. , 1994, Blood.
[29] Takashi Suda,et al. Molecular cloning and expression of the fas ligand, a novel member of the tumor necrosis factor family , 1993, Cell.
[30] N. Davidson,et al. Specific proteolytic cleavage of poly(ADP-ribose) polymerase: an early marker of chemotherapy-induced apoptosis. , 1993, Cancer research.
[31] K. Muta,et al. Apoptosis of human erythroid colony‐forming cells is decreased by stem cell factor and insulin‐like growth factor I as well as erythropoietin , 1993, Journal of cellular physiology.
[32] S. Nagata,et al. A novel protein domain required for apoptosis. Mutational analysis of human Fas antigen. , 1993, The Journal of biological chemistry.
[33] M. Takagi,et al. Interferon‐Gamma inhibits proliferation, but not commitment, of murine granulocyte‐macrophage progenitors , 1992, Journal of cellular physiology.
[34] M. Raff,et al. Social controls on cell survival and cell death , 1992, Nature.
[35] P. Lengyel,et al. The interferon system. A bird's eye view of its biochemistry. , 1992, The Journal of biological chemistry.
[36] S. Krantz,et al. Inhibition of human erythroid colony-forming units by gamma interferon can be corrected by recombinant human erythropoietin. , 1991, Blood.
[37] Atsushi Hase,et al. The polypeptide encoded by the cDNA for human cell surface antigen Fas can mediate apoptosis , 1991, Cell.
[38] H. Horvitz,et al. Mechanisms and functions of cell death. , 1991, Annual review of cell biology.
[39] C. Civin,et al. Purification of human erythroid colony-forming units and demonstration of specific binding of erythropoietin. , 1987, The Journal of clinical investigation.
[40] E. Zanjani,et al. Suppression of normal human erythropoiesis by gamma interferon in vitro. Role of monocytes and T lymphocytes. , 1985, The Journal of clinical investigation.
[41] N. Young,et al. Interferon is the suppressor of hematopoiesis generated by stimulated lymphocytes in vitro. , 1984, Journal of immunology.
[42] G. Klimpel,et al. Gamma interferon (IFN gamma) and IFN alpha/beta suppress murine myeloid colony formation (CFU-C)N: magnitude of suppression is dependent upon level of colony-stimulating factor (CSF). , 1982, Journal of immunology.
[43] William E. Stewart,et al. The Interferon System , 1979, Springer Vienna.