To the Editor.— When a psychiatric disorder has been shown to aggregate (or "run") in families, the psychiatric geneticist usually wants to determine the degree to which that aggregation results from familial/environmental vs genetic factors. Twin studies may be used to address this question. The interpretation of such studies has usually focused on the comparison of concordance rates (probandwiseconcordance is defined as the risk of illness in the cotwin of a proband-twin) in monozygotic (MZ, or identical) and dizygotic (DZ, or fraternal) twins. While a significantly higher concordance rate in MZ vs DZ twins is strong evidence for the existence ofsomedegree of genetic involvement in the disorder under consideration, without a model for genetic and nongenetic familial transmission, it is not possible to make any more quantitative statements about themagnitudeof such involvement. However, to circumvent the complexity of genetic models, several "model-free" "rules of thumb"
[1]
P. McGuffin,et al.
Twin concordance for DSM-III schizophrenia. Scrutinizing the validity of the definition.
,
1987,
Archives of general psychiatry.
[2]
K. Kendler,et al.
Overview: a current perspective on twin studies of schizophrenia.
,
1983,
The American journal of psychiatry.
[3]
S P Azen,et al.
OBTAINING CONFIDENCE INTERVALS FOR THE RISK RATIO IN COHORT STUDIES
,
1978
.
[4]
M. G. Allen.
Twin studies of affective illness.
,
1976,
Archives of general psychiatry.
[5]
J. Shields,et al.
Cross-national diagnosis of schizophrenia in twins. The heritability and specificity of schizophrenia.
,
1972,
Archives of general psychiatry.