Kim-1/Tim-1 and Immune cells: Shifting Sands
暂无分享,去创建一个
[1] S. Holdsworth,et al. Endogenous Tim-1 (Kim-1) promotes T-cell responses and cell-mediated injury in experimental crescentic glomerulonephritis. , 2012, Kidney international.
[2] P. Rennert. Novel roles for TIM-1 in immunity and infection. , 2011, Immunology letters.
[3] M. Sayegh,et al. Regulatory B cells are identified by expression of TIM-1 and can be induced through TIM-1 ligation to promote tolerance in mice. , 2011, The Journal of clinical investigation.
[4] S. Holdsworth,et al. Tim-1 promotes cisplatin nephrotoxicity. , 2011, American journal of physiology. Renal physiology.
[5] V. Kuchroo,et al. Tim‐1 stimulation of dendritic cells regulates the balance between effector and regulatory T cells , 2011, European journal of immunology.
[6] S. Rong,et al. The TIM-1:TIM-4 pathway enhances renal ischemia-reperfusion injury. , 2011, Journal of the American Society of Nephrology : JASN.
[7] A. McKenzie,et al. Tim‐1 is induced on germinal centre B cells through B‐cell receptor signalling but is not essential for the germinal centre response , 2010, Immunology.
[8] M. Okusa,et al. Macrophages, dendritic cells, and kidney ischemia-reperfusion injury. , 2010, Seminars in nephrology.
[9] L. Kane. T Cell Ig and Mucin Domain Proteins and Immunity , 2010, The Journal of Immunology.
[10] Joseph V Bonventre,et al. Kidney injury molecule-1 is a phosphatidylserine receptor that confers a phagocytic phenotype on epithelial cells. , 2008, The Journal of clinical investigation.