Characterisation of fractalkine/CX3CL1 and fractalkine receptor (CX3CR1) expression in abdominal aortic aneurysm disease.

[1]  Jianbin Wang,et al.  CD16+ monocytes produce IL‐6, CCL2, and matrix metalloproteinase‐9 upon interaction with CX3CL1‐expressing endothelial cells , 2006, Journal of leukocyte biology.

[2]  M. Aoki,et al.  Hypertension Accelerated Experimental Abdominal Aortic Aneurysm Through Upregulation of Nuclear Factor &kgr;B and Ets , 2006, Hypertension.

[3]  A. Blann,et al.  Circulating endothelial cells and endothelial progenitor cells: two sides of the same coin, or two different coins? , 2006, Atherosclerosis.

[4]  S. Carding,et al.  Increased natural killer cell activity in patients with an abdominal aortic aneurysm , 2006, The British journal of surgery.

[5]  S. Kaveri,et al.  Reduced Immunoregulatory CD31+ T Cells in Patients With Atherosclerotic Abdominal Aortic Aneurysm , 2005, Arteriosclerosis, thrombosis, and vascular biology.

[6]  O. Quehenberger,et al.  Expression of Fractalkine (CX3CL1) and its Receptor, CX3CR1, Is Elevated in Coronary Artery Disease and Is Reduced During Statin Therapy , 2005, Arteriosclerosis, thrombosis, and vascular biology.

[7]  K. Porter,et al.  Hypoxic Modulation of Ca2+ Signaling in Human Venous Endothelial Cells , 2005, Journal of Biological Chemistry.

[8]  S. Dewhurst,et al.  Immunohistochemical Assessment of Fractalkine, Inflammatory Cells, and Human Herpesvirus 7 in Human Salivary Glands , 2004, The journal of histochemistry and cytochemistry : official journal of the Histochemistry Society.

[9]  P. Grammas,et al.  Functional modulation of smooth muscle cells by the inflammatory mediator CAP37. , 2004, Microvascular research.

[10]  T. Masuda,et al.  Expression and localization of tumour necrosis factor-α and its converting enzyme in human abdominal aortic aneurysm , 2004 .

[11]  T. Greiner,et al.  Key Roles of CD4+ T Cells and IFN-γ in the Development of Abdominal Aortic Aneurysms in a Murine Model1 , 2004, The Journal of Immunology.

[12]  D. Greaves,et al.  Smooth Muscle Cells in Human Atherosclerotic Plaques Express the Fractalkine Receptor CX3CR1 and Undergo Chemotaxis to the CX3C Chemokine Fractalkine (CX3CL1) , 2003, Circulation.

[13]  B. Chandrasekar,et al.  Fractalkine (CX3CL1) stimulated by nuclear factor kappaB (NF-kappaB)-dependent inflammatory signals induces aortic smooth muscle cell proliferation through an autocrine pathway. , 2003, The Biochemical journal.

[14]  N. Tarantino,et al.  Fractalkine/CX3CL1 production by human aortic smooth muscle cells impairs monocyte procoagulant and inflammatory responses. , 2003, Cytokine.

[15]  P. Debré,et al.  Decreased Atherosclerotic Lesion Formation in CX3CR1/Apolipoprotein E Double Knockout Mice , 2003, Circulation.

[16]  N. Turner,et al.  Simvastatin inhibits human saphenous vein neointima formation via inhibition of smooth muscle cell proliferation and migration. , 2002, Journal of vascular surgery.

[17]  O. Yoshie,et al.  Dual Functions of Fractalkine/CX3C Ligand 1 in Trafficking of Perforin+/Granzyme B+ Cytotoxic Effector Lymphocytes That Are Defined by CX3CR1 Expression , 2002, The Journal of Immunology.

[18]  P. Groot,et al.  Fractalkine Is Expressed by Smooth Muscle Cells in Response to IFN-γ and TNF-α and Is Modulated by Metalloproteinase Activity1 , 2002, The Journal of Immunology.

[19]  J. Norgauer,et al.  Fractalkine induces chemotaxis and actin polymerization in human dendritic cells , 2001, Inflammation Research.

[20]  W. O'Fallon,et al.  Genetic similarity in inflammatory and degenerative abdominal aortic aneurysms: a study of human leukocyte antigen class II disease risk genes. , 2001, Journal of vascular surgery.

[21]  P. Allavena,et al.  Fractalkine (CX3CL1) as an amplification circuit of polarized Th1 responses. , 2001, The Journal of clinical investigation.

[22]  F. Numano Vasa vasoritis, vascu1itis and atherosclerosis , 2000 .

[23]  A. Yamauchi,et al.  Fractalkine-Mediated Endothelial Cell Injury by NK Cells1 , 2000, The Journal of Immunology.

[24]  N. Day,et al.  The association between cigarette smoking and abdominal aortic aneurysms. , 1999, Journal of vascular surgery.

[25]  D. Patel,et al.  Fractalkine and CX3CR1 Mediate a Novel Mechanism of Leukocyte Capture, Firm Adhesion, and Activation under Physiologic Flow , 1998, The Journal of experimental medicine.

[26]  Y. Bobryshev,et al.  Immunophenotypic Analysis of the Aortic Aneurysm Wail Suggests That Vascular Dendritic Cells are Involved in Immune Responses , 1998, Cardiovascular surgery.

[27]  M. Baggiolini Chemokines and leukocyte traffic , 1998, Nature.

[28]  T. Schall,et al.  Identification and Molecular Characterization of Fractalkine Receptor CX3CR1, which Mediates Both Leukocyte Migration and Adhesion , 1997, Cell.

[29]  J. Lindholt,et al.  Serum-elastin-peptides as a predictor of expansion of small abdominal aortic aneurysms. , 1997, European journal of vascular and endovascular surgery : the official journal of the European Society for Vascular Surgery.

[30]  Wei Wang,et al.  A new class of membrane-bound chemokine with a CX3C motif , 1997, Nature.

[31]  F. Blasi,et al.  Detection of Chlamydia pneumoniae but not Helicobacter pylori in atherosclerotic plaques of aortic aneurysms , 1996, Journal of clinical microbiology.

[32]  R. Sayers,et al.  Angiogenesis in abdominal aortic aneurysms. , 1996, European journal of vascular and endovascular surgery : the official journal of the European Society for Vascular Surgery.

[33]  J. Powell,et al.  Pathogenesis of abdominal aortic aneurysm , 1994, The British journal of surgery.