A Campylobacter jejuni gene associated with immune-mediated neuropathy
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H. Endtz | M. Gilbert | W. Wakarchuk | H. Verbrugh | Nicole van den Braak | A. Belkum | B. Jacobs | W. Ang | P. Godschalk | N. Braak
[1] A. Moran,et al. Sialylation of Lipooligosaccharide Cores Affects Immunogenicity and Serum Resistance of Campylobacter jejuni , 2000, Infection and Immunity.
[2] B. Duim,et al. Molecular Characterization of Campylobacter jejunifrom Patients with Guillain-Barré and Miller Fisher Syndromes , 2000, Journal of Clinical Microbiology.
[3] H. Endtz,et al. Campylobacter jejuni lipopolysaccharides from Guillain–Barré syndrome patients induce IgG anti-GM1 antibodies in rabbits , 2000, Journal of Neuroimmunology.
[4] J. Brisson,et al. Biosynthesis of Ganglioside Mimics in Campylobacter jejuni OH4384 , 2000, The Journal of Biological Chemistry.
[5] P. Thibault,et al. Sialic acid in the lipopolysaccharide of Haemophilus influenzae: strain distribution, influence on serum resistance and structural characterization , 1999, Molecular microbiology.
[6] R. Moor,et al. Mammalian leukocytes contain all the genetic information necessary for the development of a new individual. , 1999, Cloning.
[7] H. Hartung,et al. Guillain-Barré syndrome, CIDP and other chronic immune-mediated neuropathies. , 1998, Current opinion in neurology.
[8] G. Mckhann,et al. Campylobacter jejuni lipopolysaccharides in Guillain-Barré syndrome , 1998, Neurology.
[9] I. Nachamkin,et al. Campylobacter Species and Guillain-Barré Syndrome , 1998, Clinical Microbiology Reviews.
[10] B. Appelmelk,et al. Review: Molecular mimicry of host structures by lipopolysaccharides of Campylobacter and Helicobacter spp.: implications in pathogenesis , 1996 .
[11] 黒木 茂一. Campylobacter jejuni strains from patiensts with Guillain-Barré syndrome belong mostly to Penner serogroup 19 and contain β-N-acetylglucosamine residues , 1994 .
[12] T. Saida,et al. Campylobacter jejuni strains from patients with guillain‐barré syndrome belong mostly to penner serogrpup 19 and contain β‐N‐acetylglucosamine residues , 1993 .