Histopathological, immunohistochemical criteria and confocal laser-scanning data of arthrofibrosis.

[1]  G. G. Stokes "J." , 1890, The New Yale Book of Quotations.

[2]  G. Benagiano,et al.  Human amniotic glycodelin actively regulates changes in β-catenin immunoreactivity in cultured human umbilical vein endothelial cells (HUVEC) , 2012, The journal of maternal-fetal & neonatal medicine : the official journal of the European Association of Perinatal Medicine, the Federation of Asia and Oceania Perinatal Societies, the International Society of Perinatal Obstetricians.

[3]  R. Atit,et al.  Wnt/β-catenin signaling is hyperactivated in systemic sclerosis and induces Smad-dependent fibrotic responses in mesenchymal cells. , 2012, Arthritis and rheumatism.

[4]  Michael N. Wosczyna,et al.  Multipotent progenitors resident in the skeletal muscle interstitium exhibit robust BMP‐dependent osteogenic activity and mediate heterotopic ossification , 2012, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.

[5]  B. de Crombrugghe,et al.  β-catenin is a central mediator of pro-fibrotic Wnt signaling in systemic sclerosis , 2012, Annals of the Rheumatic Diseases.

[6]  G. Duda,et al.  Increased BMP expression in arthrofibrosis after TKA , 2012, Knee Surgery, Sports Traumatology, Arthroscopy.

[7]  T. Gehrke,et al.  [Joint endoprosthesis pathology. Histopathological diagnostics and classification]. , 2011, Der Pathologe.

[8]  J. Sohn,et al.  Blockade of the Wnt/β-catenin pathway attenuates bleomycin-induced pulmonary fibrosis. , 2011, The Tohoku journal of experimental medicine.

[9]  J. Parvizi,et al.  Mast cells and hypoxia drive tissue metaplasia and heterotopic ossification in idiopathic arthrofibrosis after total knee arthroplasty , 2010, Fibrogenesis & tissue repair.

[10]  Xi He,et al.  Wnt/ β -catenin signaling: components, mechanisms, and diseases , 2010 .

[11]  Xi He,et al.  Wnt/beta-catenin signaling: components, mechanisms, and diseases. , 2009, Developmental cell.

[12]  T. Gansukh,et al.  Twenty‐three neutrophil granulocytes in 10 high‐power fields is the best histopathological threshold to differentiate between aseptic and septic endoprosthesis loosening , 2009, Histopathology.

[13]  J. W. Wroński,et al.  Analysis of APC, alpha-, beta-catenins, and N-cadherin protein expression in aggressive fibromatosis (desmoid tumor). , 2009, Pathology, research and practice.

[14]  R. Sciot,et al.  β-Catenin Overexpression in Dupuytren’s Disease Is Unrelated to Disease Recurrence , 2009, Clinical orthopaedics and related research.

[15]  M. Otto,et al.  Histopathologische Diagnostik in der Endoprothetik , 2009, Der Orthopäde.

[16]  R. Richardson,et al.  Differential requirement for beta-catenin in epithelial and fiber cells during lens development. , 2008, Developmental biology.

[17]  W. Nelson,et al.  Adherens and tight junctions: structure, function and connections to the actin cytoskeleton. , 2008, Biochimica et biophysica acta.

[18]  J. Brand,et al.  Lokalisation von TGF-β und PDGF und deren Bedeutung für die Pathogenese der Arthrofibrose , 2008, Der Unfallchirurg.

[19]  D. O'Gorman,et al.  β-Catenin Signaling in Fibroproliferative Disease , 2007 .

[20]  D. O'Gorman,et al.  Beta-catenin signaling in fibroproliferative disease. , 2007, The Journal of surgical research.

[21]  G. Burmester,et al.  Synovitis score: discrimination between chronic low‐grade and high‐grade synovitis , 2006, Histopathology.

[22]  A. Adili,et al.  Predictive risk factors for stiff knees in total knee arthroplasty. , 2006, The Journal of arthroplasty.

[23]  R. Burgkart,et al.  [Therapy of arthrofibrosis after total knee arthroplasty]. , 2006, Der Orthopade.

[24]  V. Bühren,et al.  Therapie der Arthrofibrose nach Kniegelenkendoprothetik , 2006, Der Orthopäde.

[25]  A. Weiler,et al.  α-Smooth muscle actin containing contractile fibroblastic cells in human knee arthrofibrosis tissue , 2004, Archives of Orthopaedic and Trauma Surgery.

[26]  L. Frommelt,et al.  [Proposal for the classification of the periprosthetic membrane from loosened hip and knee endoprostheses]. , 2004, Der Pathologe.

[27]  S. Kirschner,et al.  Vorschlag für eine Konsensus-Klassifikation der periprothetischen Membran gelockerter Hüft- und Knieendoprothesen , 2004, Der Pathologe.

[28]  G. Gabbiani,et al.  Alpha-smooth muscle actin is transiently expressed by myofibroblasts during experimental wound healing. , 1990, Laboratory investigation; a journal of technical methods and pathology.