The role of receptor internalization in CD95 signaling
暂无分享,去创建一个
C. Hallas | M. Peter | A. Chan | Kyeong-Hee Lee | R. Schickel | C. Feig | S. Schütze | V. Tchikov | Robert Schickel
[1] T. Mimori,et al. Role of membrane sphingomyelin and ceramide in platform formation for Fas-mediated apoptosis , 2005, The Journal of experimental medicine.
[2] M. Peter,et al. Does CD95 have tumor promoting activities? , 2005, Biochimica et biophysica acta.
[3] M. Sarker,et al. SPOTS: signaling protein oligomeric transduction structures are early mediators of death receptor-induced apoptosis at the plasma membrane. , 2004, The Journal of cell biology.
[4] D. Kabelitz,et al. Compartmentalization of TNF receptor 1 signaling: internalized TNF receptosomes as death signaling vesicles. , 2004, Immunity.
[5] M. Peter,et al. CD95 ligand induces motility and invasiveness of apoptosis‐resistant tumor cells , 2004, The EMBO journal.
[6] L. Pelkmans,et al. Not just a sink: endosomes in control of signal transduction. , 2004, Current opinion in cell biology.
[7] A. Strasser,et al. Modifications and intracellular trafficking of FADD/MORT1 and caspase-8 after stimulation of T lymphocytes , 2004, Cell Death and Differentiation.
[8] L. Ricci-Vitiani,et al. CD95 death‐inducing signaling complex formation and internalization occur in lipid rafts of type I and type II cells , 2004, European journal of immunology.
[9] Bianca Habermann,et al. APPL Proteins Link Rab5 to Nuclear Signal Transduction via an Endosomal Compartment , 2004, Cell.
[10] R. Siegel,et al. Ligand-independent redistribution of Fas (CD95) into lipid rafts mediates clonotypic T cell death , 2004, Nature Immunology.
[11] Alicia Algeciras-Schimnich,et al. Two CD95 tumor classes with different sensitivities to antitumor drugs , 2003, Proceedings of the National Academy of Sciences of the United States of America.
[12] A. Motley,et al. Clathrin-mediated endocytosis in AP-2–depleted cells , 2003, The Journal of cell biology.
[13] J. Tschopp,et al. Induction of TNF Receptor I-Mediated Apoptosis via Two Sequential Signaling Complexes , 2003, Cell.
[14] A. Algeciras-Schimnich,et al. Actin dependent CD95 internalization is specific for Type I cells , 2003, FEBS letters.
[15] J. Cidlowski,et al. Molecular evidence for the nuclear localization of FADD , 2003, Cell Death and Differentiation.
[16] P. Krammer,et al. Function and regulation of the CD95 (APO-1/Fas) ligand in the immune system. , 2003, Seminars in immunology.
[17] M. Peter,et al. The CD95 type I/type II model. , 2003, Seminars in immunology.
[18] J. Tschopp,et al. Recruitment of TNF Receptor 1 to Lipid Rafts Is Essential for TNFα-Mediated NF-κB Activation , 2003 .
[19] A. Bird,et al. Fas-associated death domain protein interacts with methyl-CpG binding domain protein 4: A potential link between genome surveillance and apoptosis , 2003, Proceedings of the National Academy of Sciences of the United States of America.
[20] D. Teis,et al. Localization of the MP1-MAPK scaffold complex to endosomes is mediated by p14 and required for signal transduction. , 2002, Developmental cell.
[21] B. Camoretti-Mercado,et al. Fas Resistance of Leukemic Eosinophils Is Due to Activation of NF-κB by Fas Ligation1 , 2002, The Journal of Immunology.
[22] M. Peter,et al. Inactivation of Caspase-8 on Mitochondria of Bcl-xL-expressing MCF7-Fas Cells , 2002, The Journal of Biological Chemistry.
[23] Alicia Algeciras-Schimnich,et al. Molecular Ordering of the Initial Signaling Events of CD95 , 2002, Molecular and Cellular Biology.
[24] Sun-Mi Park,et al. Non-apoptotic Signaling Pathways Activated by Soluble Fas Ligand in Serum-starved Human Fibroblasts , 2001, The Journal of Biological Chemistry.
[25] T. Martin. PI(4,5)P(2) regulation of surface membrane traffic. , 2001, Current opinion in cell biology.
[26] J. Tschopp,et al. Ceramide Enables Fas to Cap and Kill* , 2001, The Journal of Biological Chemistry.
[27] R. Kolesnick,et al. CD95 Signaling via Ceramide-rich Membrane Rafts* , 2001, The Journal of Biological Chemistry.
[28] A. Pfeifer,et al. A Novel 14-Kilodalton Protein Interacts with the Mitogen-Activated Protein Kinase Scaffold Mp1 on a Late Endosomal/Lysosomal Compartment , 2001, The Journal of cell biology.
[29] A. Strasser,et al. Bcl-2 does not inhibit cell death induced by the physiological Fas ligand: implications for the existence of type I and type II cells , 2000, Cell Death and Differentiation.
[30] J K Frederiksen,et al. Fas preassociation required for apoptosis signaling and dominant inhibition by pathogenic mutations. , 2000, Science.
[31] S. Schmid,et al. Regulation of signal transduction by endocytosis. , 2000, Current opinion in cell biology.
[32] Tobias Meyer,et al. Phosphatidylinositol 4,5-Bisphosphate Functions as a Second Messenger that Regulates Cytoskeleton–Plasma Membrane Adhesion , 2000, Cell.
[33] S. Nagata,et al. Fas ligand-induced apoptosis. , 1999, Annual review of genetics.
[34] M. Peter,et al. Defective CD95/APO-1/Fas signal complex formation in the human autoimmune lymphoproliferative syndrome, type Ia. , 1999, Proceedings of the National Academy of Sciences of the United States of America.
[35] E. White,et al. E1B 19K Inhibits Fas-mediated Apoptosis through FADD-dependent Sequestration of FLICE , 1998, The Journal of cell biology.
[36] S. Nagata,et al. Downregulation of Fas ligand by shedding , 1998, Nature Medicine.
[37] C. Huynh,et al. Identification and characterization of an essential family of inositol polyphosphate 5-phosphatases (INP51, INP52 and INP53 gene products) in the yeast Saccharomyces cerevisiae. , 1998, Genetics.
[38] R. Siegel,et al. Membrane Oligomerization and Cleavage Activates the Caspase-8 (FLICE/MACHα1) Death Signal* , 1998, The Journal of Biological Chemistry.
[39] Y. Tsujimoto,et al. Bcl-2 blocks loss of mitochondrial membrane potential while ICE inhibitors act at a different step during inhibition of death induced by respiratory chain inhibitors. , 1996, Oncogene.
[40] M. Peter,et al. Cytotoxicity‐dependent APO‐1 (Fas/CD95)‐associated proteins form a death‐inducing signaling complex (DISC) with the receptor. , 1995, The EMBO journal.
[41] J. Bonifacino,et al. Interaction of tyrosine-based sorting signals with clathrin-associated proteins. , 1995, Science.
[42] Arul M. Chinnaiyan,et al. FADD, a novel death domain-containing protein, interacts with the death domain of fas and initiates apoptosis , 1995, Cell.
[43] J. Camonis,et al. A Novel Protein That Interacts with the Death Domain of Fas/APO1 Contains a Sequence Motif Related to the Death Domain (*) , 1995, The Journal of Biological Chemistry.
[44] G. M. Di Guglielmo,et al. Compartmentalization of SHC, GRB2 and mSOS, and hyperphosphorylation of Raf‐1 by EGF but not insulin in liver parenchyma. , 1994, The EMBO journal.
[45] A. Shaw,et al. Palmitylation of an amino-terminal cysteine motif of protein tyrosine kinases p56lck and p59fyn mediates interaction with glycosyl-phosphatidylinositol-anchored proteins , 1993, Molecular and cellular biology.
[46] S. Nagata,et al. A novel protein domain required for apoptosis. Mutational analysis of human Fas antigen. , 1993, The Journal of biological chemistry.
[47] P. Möller,et al. Induction of apoptosis by monoclonal antibody anti-APO-1 class switch variants is dependent on cross-linking of APO-1 cell surface antigens. , 1992, Journal of immunology.
[48] M. Pawlita,et al. Purification and molecular cloning of the APO-1 cell surface antigen, a member of the tumor necrosis factor/nerve growth factor receptor superfamily. Sequence identity with the Fas antigen. , 1992, The Journal of biological chemistry.
[49] T. Puck,et al. Cell surface antigens , 1976 .
[50] M. Peter,et al. The CD95(APO-1/Fas) DISC and beyond , 2003, Cell Death and Differentiation.
[51] J. Tschopp,et al. Recruitment of TNF receptor 1 to lipid rafts is essential for TNFalpha-mediated NF-kappaB activation. , 2003, Immunity.
[52] B. Camoretti-Mercado,et al. Fas resistance of leukemic eosinophils is due to activation of NF-kappa B by Fas ligation. , 2002, Journal of immunology.
[53] C. Huynh,et al. Identification and Characterization of an Essential Family of Inositol Polyphosphate 5-Phosphatases ( INP 51 , INP 52 and INP 53 Gene Products ) in the Yeast Saccharomyces cerevisiae , 1998 .