NAP1L1‐MLLT10 is a rare recurrent translocation that is associated with HOXA activation and poor treatment response in T‐cell acute lymphoblastic leukaemia
暂无分享,去创建一个
E. Macintyre | T. Fest | V. Asnafi | A. Trinquand | A. Contet | C. Schmitt | A. Cieslak | A. Touzart | J. Bond | M. Escoffre | M. Muller | T. Marchand
[1] E. Macintyre,et al. Cryptic XPO1-MLLT10 translocation is associated with HOXA locus deregulation in T-ALL. , 2014, Blood.
[2] S. Chiaretti,et al. DDX3X-MLLT10 fusion in adults with NOTCH1 positive T-cell acute lymphoblastic leukemia , 2014, Haematologica.
[3] Vahid Asnafi,et al. The prognosis of CALM-AF10-positive adult T-cell acute lymphoblastic leukemias depends on the stage of maturation arrest , 2013, Haematologica.
[4] C. Mecucci,et al. New MLLT10 gene recombinations in pediatric T-acute lymphoblastic leukemia. , 2013, Blood.
[5] Kiran C. Bobba,et al. The genetic basis of early T-cell precursor acute lymphoblastic leukaemia , 2012, Nature.
[6] K. Nagata,et al. Functional characterization of human nucleosome assembly protein 1‐like proteins as histone chaperones , 2010, Genes to cells : devoted to molecular & cellular mechanisms.
[7] Yi Zhang,et al. hDOT1L Links Histone Methylation to Leukemogenesis , 2005, Cell.
[8] E. Macintyre,et al. Analysis of TCR, pT alpha, and RAG-1 in T-acute lymphoblastic leukemias improves understanding of early human T-lymphoid lineage commitment. , 2003, Blood.
[9] W. Hiddemann,et al. MLL and CALM are fused to AF10 in morphologically distinct subsets of acute leukemia with translocation t(10;11): both rearrangements are associated with a poor prognosis. , 1998, Blood.
[10] A. Hagemeijer,et al. Breakpoint heterogeneity in t(10;11) translocation in AML-M4/M5 resulting in fusion of AF10 and MLL is resolved by fluorescent in situ hybridization analysis. , 1995, Cancer research.