Nitric oxide synthase in muscular dystrophies: a re-evaluation

[1]  M. Wellner,et al.  Nitric oxide synthase in human skeletal muscles related to defined fibre types , 2006, Histochemistry and Cell Biology.

[2]  M. Wellner,et al.  L-Arginine-NO-cGMP signaling following acute liver injury in the rat. , 2005, Experimental and toxicologic pathology : official journal of the Gesellschaft fur Toxikologische Pathologie.

[3]  J. Frisbee.  Reduced nitric oxide bioavailability contributes to skeletal muscle microvessel rarefaction in the metabolic syndrome. , 2005, American journal of physiology. Regulatory, integrative and comparative physiology.

[4]  M. Wellner,et al.  Compartmentalization of NO signaling cascade in skeletal muscles. , 2005, Biochemical and biophysical research communications.

[5]  K. Kyriacou,et al.  Reversible inflammatory and vacuolar myopathy with vitamin E deficiency in celiac disease , 2005, Muscle & nerve.

[6]  I. Hausmanowa-Petrusewicz,et al.  The involvement of oxidative stress in determining the severity and progress of pathological processes in dystrophin-deficient muscles. , 2005, Acta biochimica Polonica.

[7]  F. Callea,et al.  Co-expression and modulation of neuronal and endothelial nitric oxide synthase in human endothelial cells. , 2004, Journal of molecular and cellular cardiology.

[8]  F. Muntoni,et al.  Absence of neuronal nitric oxide synthase (nNOS) as a pathological marker for the diagnosis of Becker muscular dystrophy with rod domain deletions , 2004, Neuropathology and applied neurobiology.

[9]  G. Wallukat,et al.  Cellular control of nitric oxide synthase expression and activity in rat cardiomyocytes. , 2004, Antioxidants & redox signaling.

[10]  M. Wellner,et al.  An in situ evidence for autocrine function of NO in the vasculature. , 2004, Nitric oxide : biology and chemistry.

[11]  I. Maruyama,et al.  Decreased Total Nitric Oxide Production in Patients with Duchenne Muscular Dystrophy , 2004, Journal of Biomedical Science.

[12]  J. Schröder,et al.  Expression and distribution of the nitric oxide synthases in idiopathic inflammatory myopathies , 2004, Acta Neuropathologica.

[13]  M. Wellner,et al.  Nitric Oxide Synthase Isoforms I, III and Protein Kinase-Cθ in Skeletal Muscle Fibres of Normal and Streptozotocin-induced Diabetic Rats With and Without Ginkgo Biloba Extract Treatment , 2001, The Histochemical Journal.

[14]  O. Baum,et al.  Skeletal Muscle Fibres Show NADPH Diaphorase Activity Associated with Mitochondria, the Sarcoplasmic Reticulum and the NOS-1-containing Sarcolemma , 2000, The Histochemical Journal.

[15]  D. Blottner,et al.  Absence of nitric oxide synthase I despite the presence of the dystrophin complex in human striated muscle , 1997, The Histochemical Journal.

[16]  G. Wallukat,et al.  Inducible nitric oxide synthase in the myocard , 2004, Molecular and Cellular Biochemistry.

[17]  M. Tarnopolsky,et al.  Patients with dystrophinopathy show evidence of increased oxidative stress. , 2003, Free radical biology & medicine.

[18]  G. Vita,et al.  Activation of nuclear factor-κB in inflammatory myopathies and Duchenne muscular dystrophy , 2003, Neurology.

[19]  A. Contestabile,et al.  Nitric Oxide Protects Neuroblastoma Cells from Apoptosis Induced by Serum Deprivation through cAMP-response Element-binding Protein (CREB) Activation* , 2002, The Journal of Biological Chemistry.

[20]  M. Wellner,et al.  Nitric oxide synthase II in rat skeletal muscles , 2002, Histochemistry and Cell Biology.

[21]  D. Tews.  Apoptosis and muscle fibre loss in neuromuscular disorders , 2002, Neuromuscular Disorders.

[22]  Y. Vodovotz,et al.  Nitric oxide and wound repair: role of cytokines? , 2002, Nitric oxide : biology and chemistry.

[23]  M. Lerch,et al.  Vascular smooth muscle and nitric oxide synthase , 2002, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[24]  A. Wöckel,et al.  The specificity of the histochemical NADPH diaphorase reaction for nitric oxide synthase-1 in skeletal muscles is increased in the presence of urea. , 2002, Acta histochemica.

[25]  T. Rando.  Role of nitric oxide in the pathogenesis of muscular dystrophies: A “two hit” hypothesis of the cause of muscle necrosis , 2001, Microscopy research and technique.

[26]  O. Baum,et al.  Nitric oxide synthase-1 is enriched in fast-twitch oxidative myofibers , 2001, Cell and Tissue Research.

[27]  S R Shi,et al.  Antigen Retrieval Techniques , 2001, The journal of histochemistry and cytochemistry : official journal of the Histochemistry Society.

[28]  Marc Montminy,et al.  Transcriptional regulation by the phosphorylation-dependent factor CREB , 2001, Nature Reviews Molecular Cell Biology.

[29]  K. Wang,et al.  The calpain family and human disease. , 2001, Trends in molecular medicine.

[30]  James M. Wilson,et al.  iNOS Expression In Dystrophinopathies Can Be Reduced By Somatic Gene Transfer of Dystrophin or Utrophin , 2001, Molecular medicine.

[31]  R. Crosbie.  NO vascular control in Duchenne muscular dystrophy , 2001, Nature Medicine.

[32]  G. D. Thomas,et al.  Functional muscle ischemia in neuronal nitric oxide synthase-deficient skeletal muscle of children with Duchenne muscular dystrophy. , 2000, Proceedings of the National Academy of Sciences of the United States of America.

[33]  D. Ginty,et al.  Dynamic regulation of neuronal NO synthase transcription by calcium influx through a CREB family transcription factor-dependent mechanism. , 2000, Proceedings of the National Academy of Sciences of the United States of America.

[34]  A. Serdaroğlu,et al.  The serum nitric oxide levels in patients with Duchenne muscular dystrophy , 2000, Brain and Development.

[35]  T. Rando,et al.  Dystrophin mutations predict cellular susceptibility to oxidative stress , 2000, Muscle & nerve.

[36]  J. E. Anderson,et al.  A role for nitric oxide in muscle repair: nitric oxide-mediated activation of muscle satellite cells. , 2000, Molecular biology of the cell.

[37]  U. Förstermann,et al.  NO synthase II in mouse skeletal muscle is associated with caveolin 3. , 1999, The Biochemical journal.

[38]  I. Nonaka,et al.  α1-Syntrophin Gene Disruption Results in the Absence of Neuronal-type Nitric-oxide Synthase at the Sarcolemma but Does Not Induce Muscle Degeneration* , 1999, The Journal of Biological Chemistry.

[39]  D. Bredt.  NO skeletal muscle derived relaxing factor in Duchenne muscular dystrophy. , 1998, Proceedings of the National Academy of Sciences of the United States of America.

[40]  A. Yao,et al.  Transcriptional regulation of inducible nitric oxide synthase in cultured neonatal rat cardiac myocytes. , 1997, Circulation research.

[41]  J. Leiden.  The genetics of dilated cardiomyopathy--emerging clues to the puzzle. , 1997, The New England journal of medicine.

[42]  Hans J. Tanke,et al.  Fluorochrome-labeled Tyramides: Use in Immunocytochemistry and Fluorescence In Situ Hybridization , 1997, The journal of histochemistry and cytochemistry : official journal of the Histochemistry Society.

[43]  M. Nakane,et al.  Inducible NO synthase II and neuronal NO synthase I are constitutively expressed in different structures of guinea pig skeletal muscle: implications for contractile function , 1996, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[44]  J. Stull,et al.  Neuronal nitric oxide synthase and dystrophin-deficient muscular dystrophy. , 1996, Proceedings of the National Academy of Sciences of the United States of America.

[45]  D. Bredt,et al.  Selective loss of sarcolemmal nitric oxide synthase in Becker muscular dystrophy , 1996, The Journal of experimental medicine.

[46]  K. Aldape,et al.  Nitric oxide synthase complexed with dystrophin and absent from skeletal muscle sarcolemma in Duchenne muscular dystrophy , 1995, Cell.

[47]  James B. Mitchell,et al.  Nitric oxide protects against cellular damage and cytotoxicity from reactive oxygen species. , 1993, Proceedings of the National Academy of Sciences of the United States of America.

[48]  R. Adams,et al.  PRINCIPLES OF MYOPATHOLOGY AS ILLUSTRATED IN THE NUTRITIONAL MYOPATHY OF THE ROTTNEST QUOKKA (SETONIX BRACHYURUS) , 1966, Annals of the New York Academy of Sciences.