Parvalbumin overexpression alters immune‐mediated increases in intracellular calcium, and delays disease onset in a transgenic model of familial amyotrophic lateral sclerosis
暂无分享,去创建一个
S. Appel | D. Beers | L. Siklós | R. G. Smith | M. Alexianu | A. H. Mohamed | Yasushi Otsuka | B. Ho | D. Mosier | R. E. McAlhany | Milena E. Kozovska | R. McAlhany
[1] A. Pullen,et al. Ultrastructural analysis of spinal motoneurones from mice treated with IgG from ALS patients, healthy individuals, or disease controls , 2000, Journal of the Neurological Sciences.
[2] B. Keller,et al. Calcium dynamics and buffering in oculomotor neurones from mouse that are particularly resistant during amyotrophic lateral sclerosis (ALS)‐related motoneurone disease , 2000, The Journal of physiology.
[3] P. Stieg,et al. Functional role of caspase-1 and caspase-3 in an ALS transgenic mouse model. , 2000, Science.
[4] O. Uchitel,et al. Amyotrophic lateral sclerosis IgG‐treated neuromuscular junctions develop sensitivity to L‐type calcium channel blocker , 2000, Muscle & nerve.
[5] M. Ward,et al. Mitochondrial membrane potential and neuronal glutamate excitotoxicity: mortality and millivolts , 2000, Trends in Neurosciences.
[6] S. Appel,et al. Ultrastructural evidence of calcium involvement in experimental autoimmune gray matter disease , 2000, Journal of neuroscience research.
[7] S. Appel,et al. Resistance of extraocular motoneuron terminals to effects of amyotrophic lateral sclerosis sera , 2000, Neurology.
[8] F E Bloom,et al. Differential vulnerability of oculomotor, facial, and hypoglossal nuclei in G86R superoxide dismutase transgenic mice , 2000, The Journal of comparative neurology.
[9] Margaret A. Johnson,et al. Mitochondrial enzyme activity in amyotrophic lateral sclerosis: Implications for the role of mitochondria in neuronal cell death , 1999, Annals of neurology.
[10] R. Adalbert,et al. Calcium-containing endosomes at oculomotor terminals in animal models of ALS. , 1999, Neuroreport.
[11] Ole A. Andreassen,et al. Neuroprotective effects of creatine in a transgenic animal model of amyotrophic lateral sclerosis , 1999, Nature Medicine.
[12] D. Figlewicz,et al. Glutamate Potentiates the Toxicity of Mutant Cu/Zn-Superoxide Dismutase in Motor Neurons by Postsynaptic Calcium-Dependent Mechanisms , 1998, The Journal of Neuroscience.
[13] M. Gurney,et al. Intracellular Calcium Parallels Motoneuron Degeneration in SOD-1 Mutant Mice , 1998, Journal of neuropathology and experimental neurology.
[14] J. Kong,et al. Massive Mitochondrial Degeneration in Motor Neurons Triggers the Onset of Amyotrophic Lateral Sclerosis in Mice Expressing a Mutant SOD1 , 1998, The Journal of Neuroscience.
[15] Lin Jin,et al. Aberrant RNA Processing in a Neurodegenerative Disease: the Cause for Absent EAAT2, a Glutamate Transporter, in Amyotrophic Lateral Sclerosis , 1998, Neuron.
[16] Kwang-Woo Lee,et al. Amyotrophic lateral sclerosis: Serum factors enhance spontaneous and evoked transmitter release at the neuromuscular junction , 1998, Muscle & nerve.
[17] Robert H. Brown,et al. Evidence of Increased Oxidative Damage in Both Sporadic and Familial Amyotrophic Lateral Sclerosis , 1997, Journal of neurochemistry.
[18] F. Poccia,et al. Expression of a Cu,Zn superoxide dismutase typical of familial amyotrophic lateral sclerosis induces mitochondrial alteration and increase of cytosolic Ca2+ concentration in transfected neuroblastoma SH‐SY5Y cells , 1997, FEBS letters.
[19] L. Colom,et al. Amyotrophic Lateral Sclerosis Immunoglobulins Increase Intracellular Calcium in a Motoneuron Cell Line , 1997, Experimental Neurology.
[20] M. Dubois‐Dauphin,et al. Bcl-2: prolonging life in a transgenic mouse model of familial amyotrophic lateral sclerosis. , 1997, Science.
[21] Junying Yuan,et al. Inhibition of ICE slows ALS in mice , 1997, Nature.
[22] S. Appel,et al. Altered Calcium Homeostasis and Ultrastructure in Motoneurons of Mice Caused by Passively Transferred Anti‐motoneuronal IgG , 1997, Journal of neuropathology and experimental neurology.
[23] L. Colom,et al. Expression of calbindin-D28K in motoneuron hybrid cells after retroviral infection with calbindin-D28K cDNA prevents amyotrophic lateral sclerosis IgG-mediated cytotoxicity. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[24] O. Andreassen,et al. Estimation of the number of somatostatin neurons in the striatum: An in situ hybridization study using the optical fractionator method , 1996, The Journal of comparative neurology.
[25] C. Heizmann,et al. α-Parvalbumin reduces depolarizationminduced elevations of cytosolic free calcium in human neuroblastoma cells , 1996 .
[26] F. Joó,et al. Ultrastructural evidence for altered calcium in motor nerve terminals in amyotrophc lateral sclerosis , 1996, Annals of neurology.
[27] M. Gurney,et al. Benefit of vitamin E, riluzole, and gababapentin in a transgenic model of familial amyotrophic lateral sclerosis , 1996, Annals of neurology.
[28] L. Komuves,et al. Antibodies to calcium channels from ALS patients passively transferred to mice selectively increase intracellular calcium and induce ultrastructural changes in motoneurons , 1995, Synapse.
[29] D. Borchelt,et al. An adverse property of a familial ALS-linked SOD1 mutation causes motor neuron disease characterized by vacuolar degeneration of mitochondria , 1995, Neuron.
[30] W. Snider,et al. Parvalbumin is a marker of ALS-resistant motor neurons. , 1995, Neuroreport.
[31] E. Stefani,et al. Amyotrophic lateral sclerosis immunoglobulins increase Ca2+ currents in a motoneuron cell line , 1995, Annals of neurology.
[32] V. La Bella,et al. The role of calcium‐binding proteins in selective motoneuron vulnerability in amyotrophic lateral sclerosis , 1994, Annals of neurology.
[33] M. Gurney,et al. Development of central nervous system pathology in a murine transgenic model of human amyotrophic lateral sclerosis. , 1994, The American journal of pathology.
[34] M. Gurney,et al. Motor neuron degeneration in mice that express a human Cu,Zn superoxide dismutase mutation. , 1994, Science.
[35] R. Miller,et al. Calcium buffering properties of calbindin D28k and parvalbumin in rat sensory neurones. , 1993, The Journal of physiology.
[36] H. Nojima,et al. Expression of the rat calmodulin gene II in the central nervous system: a 294-base promoter and 68-base leader segment mediates neuron-specific gene expression in transgenic mice. , 1993, Brain research. Molecular brain research.
[37] M. Pericak-Vance,et al. Amyotrophic lateral sclerosis and structural defects in Cu,Zn superoxide dismutase. , 1993, Science.
[38] C. Heizmann,et al. Parvalbumin and calbindin D‐28k in the human motor system and in motor neuron disease , 1993, Neuropathology and applied neurobiology.
[39] E. B. VEDEL JENSEN,et al. The rotator , 1993 .
[40] J. Haines,et al. Mutations in Cu/Zn superoxide dismutase gene are associated with familial amyotrophic lateral sclerosis , 1993, Nature.
[41] D. Choi. Excitotoxic cell death. , 1992, Journal of neurobiology.
[42] P. Emson,et al. Stable transfection of calbindin-D28k into the GH3 cell line alters calcium currents and intracellular calcium homeostasis , 1992, Neuron.
[43] J. Rothstein,et al. Decreased glutamate transport by the brain and spinal cord in amyotrophic lateral sclerosis. , 1992, The New England journal of medicine.
[44] H. Gundersen,et al. Unbiased stereological estimation of the total number of neurons in the subdivisions of the rat hippocampus using the optical fractionator , 1991, The Anatomical record.
[45] E. Stefani,et al. Immunoglobulins from animal models of motor neuron disease and from human amyotrophic lateral sclerosis patients passively transfer physiological abnormalities to the neuromuscular junction. , 1991, Proceedings of the National Academy of Sciences of the United States of America.
[46] M. Celio,et al. Calbindin D-28k and parvalbumin in the rat nervous system , 1990, Neuroscience.
[47] H J Gundersen,et al. The efficiency of systematic sampling in stereology and its prediction * , 1987, Journal of microscopy.
[48] R. Adalbert,et al. Altered calcium homeostasis in spinal motoneurons but not in oculomotor neurons of SOD-1 knockout mice , 2000, Acta Neuropathologica.
[49] C. Heizmann,et al. Alpha-parvalbumin reduces depolarization-induced elevations of cytosolic free calcium in human neuroblastoma cells. , 1996, Cell Calcium.
[50] A. Hirano. Cytopathology of amyotrophic lateral sclerosis. , 1991, Advances in neurology.
[51] E. Stefani,et al. Immune-mediated models of motor neuron destruction in the guinea pig. , 1991, Advances in neurology.
[52] P N Leigh,et al. Cytoskeletal pathology in motor neuron diseases. , 1991, Advances in neurology.
[53] R. Bauer. 6 Electron Spectroscopic Imaging: An Advanced Technique for Imaging and Analysis in Transmission Electron Microscopy , 1988 .