Reactive oxygen species in the normal and acutely injured liver.
暂无分享,去创建一个
[1] Hartmut Jaeschke,et al. Reactive oxygen and mechanisms of inflammatory liver injury: Present concepts , 2011, Journal of gastroenterology and hepatology.
[2] C. Zwingmann,et al. Novel mechanisms of protection against acetaminophen hepatotoxicity in mice by glutathione and N‐acetylcysteine , 2010, Hepatology.
[3] W. Junger,et al. Circulating Mitochondrial DAMPs Cause Inflammatory Responses to Injury , 2009, Nature.
[4] H. Jaeschke. Antioxidant Defense Mechanisms , 2010 .
[5] M. Bianchi,et al. HMGB1 loves company , 2009, Journal of leukocyte biology.
[6] H. Jaeschke,et al. Oxidant stress-induced liver injury in vivo: role of apoptosis, oncotic necrosis, and c-Jun NH2-terminal kinase activation. , 2009, American journal of physiology. Gastrointestinal and liver physiology.
[7] H. Jaeschke,et al. Translocation of iron from lysosomes into mitochondria is a key event during oxidative stress‐induced hepatocellular injury , 2008, Hepatology.
[8] Jordi Muntané,et al. Mecanismos de lesión hepatocelular , 2007 .
[9] H. Jaeschke. Mechanisms of Liver Injury. II. Mechanisms of neutrophil-induced liver cell injury during hepatic ischemia-reperfusion and other acute inflammatory conditions. , 2006, American journal of physiology. Gastrointestinal and liver physiology.
[10] Hartmut Jaeschke,et al. Intracellular signaling mechanisms of acetaminophen-induced liver cell death. , 2006, Toxicological sciences : an official journal of the Society of Toxicology.
[11] A. Vollmar,et al. Prevention of Kupffer cell-induced oxidant injury in rat liver by atrial natriuretic peptide. , 1999, American journal of physiology. Gastrointestinal and liver physiology.