Priceless GEMMs: genetically engineered mouse models for colorectal cancer drug development.
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[1] M. Pai,et al. Consumption of high-fat diet induces tumor progression and epithelial-mesenchymal transition of colorectal cancer in a mouse xenograft model. , 2012, The Journal of nutritional biochemistry.
[2] Steffen Jung,et al. Utilization of Murine Colonoscopy for Orthotopic Implantation of Colorectal Cancer , 2011, PloS one.
[3] Erin M. Coffee,et al. The Dual PI3K/mTOR Inhibitor NVP-BEZ235 Induces Tumor Regression in a Genetically Engineered Mouse Model of PIK3CA Wild-Type Colorectal Cancer , 2011, PloS one.
[4] Stefan Wirtz,et al. Confocal laser endomicroscopy and narrow-band imaging-aided endoscopy for in vivo imaging of colitis and colon cancer in mice , 2011, Nature Protocols.
[5] W. Zhou,et al. Treatment of patient tumor-derived colon cancer xenografts by a TRAIL gene-armed oncolytic adenovirus , 2011, Cancer Gene Therapy.
[6] J. Xie,et al. Rh-I-UEA-1 Polymerized Liposomes Target and Image Adenomatous Polyps in the APCMin/+ Mouse Using Optical Colonography , 2011, Molecular imaging.
[7] Thomas D. Wang,et al. Targeted imaging of colorectal dysplasia in living mice with fluorescence microendoscopy , 2011, Biomedical optics express.
[8] Ying Feng,et al. In Vivo Fluorescence-Based Endoscopic Detection of Colon Dysplasia in the Mouse Using a Novel Peptide Probe , 2011, PloS one.
[9] J. Heyer,et al. KRAS Mouse Models: Modeling Cancer Harboring KRAS Mutations. , 2011, Genes & cancer.
[10] B. Spencer‐Dene,et al. FBXW7 influences murine intestinal homeostasis and cancer, targeting Notch, Jun, and DEK for degradation , 2011, The Journal of experimental medicine.
[11] C. Xiong,et al. In Vivo Small-Animal PET/CT of EphB4 Receptors Using 64Cu-Labeled Peptide , 2011, The Journal of Nuclear Medicine.
[12] A. Jemal,et al. Global Cancer Statistics , 2011 .
[13] S. Cook,et al. V600EBraf induces gastrointestinal crypt senescence and promotes tumour progression through enhanced CpG methylation of p16INK4a , 2010, EMBO molecular medicine.
[14] Yu Sun,et al. A superficial colon tumor model involving subcutaneous colon translocation and orthotopic transplantation of green fluorescent protein-expressing human colon tumor , 2010, Tumor Biology.
[15] P. Snyder,et al. Generation of a Transgenic Mouse for Colorectal Cancer Research with Intestinal Cre Expression Limited to the Large Intestine , 2010, Molecular Cancer Research.
[16] P. Devilee,et al. A new conditional Apc-mutant mouse model for colorectal cancer. , 2010, Carcinogenesis.
[17] Euiheon Chung,et al. In vivo wide-area cellular imaging by side-view endomicroscopy , 2010, Nature Methods.
[18] R. Kucherlapati,et al. An Msh2 conditional knockout mouse for studying intestinal cancer and testing anticancer agents. , 2010, Gastroenterology.
[19] Umar Mahmood,et al. Development of a mouse model for sporadic and metastatic colon tumors and its use in assessing drug treatment , 2010, Proceedings of the National Academy of Sciences.
[20] T. Jacks,et al. Complete deletion of Apc results in severe polyposis in mice , 2009, Oncogene.
[21] David M Sabatini,et al. mTOR and cancer: many loops in one pathway. , 2009, Current opinion in cell biology.
[22] C. Baker,et al. A metastatic colon cancer model using nonoperative transanal rectal injection , 2010, Surgical Endoscopy.
[23] David Allard,et al. Inhibition of Hedgehog Signaling Enhances Delivery of Chemotherapy in a Mouse Model of Pancreatic Cancer , 2009, Science.
[24] Nina M. Muñoz,et al. TGF-beta receptor inactivation and mutant Kras induce intestinal neoplasms in mice via a beta-catenin-independent pathway. , 2009, Gastroenterology.
[25] Dongsheng Tu,et al. K-ras mutations and benefit from cetuximab in advanced colorectal cancer. , 2008, The New England journal of medicine.
[26] R. Kucherlapati,et al. Loss of Rb1 in the gastrointestinal tract of Apc1638N mice promotes tumors of the cecum and proximal colon , 2008, Proceedings of the National Academy of Sciences.
[27] H. Lane,et al. Inhibition of the mTORC1 pathway suppresses intestinal polyp formation and reduces mortality in ApcΔ716 mice , 2008, Proceedings of the National Academy of Sciences.
[28] A. Sweet-Cordero,et al. Differential effects of oncogenic K-Ras and N-Ras on proliferation, differentiation and tumor progression in the colon , 2008, Nature Genetics.
[29] Kathleen R. Cho,et al. Mouse model of colonic adenoma-carcinoma progression based on somatic Apc inactivation. , 2007, Cancer research.
[30] H. Mori,et al. Multistep carcinogenesis of the colon in ApcMin/+ mouse , 2007, Cancer science.
[31] M. Neurath,et al. High resolution colonoscopy in live mice , 2006, Nature Protocols.
[32] B. Dutrillaux,et al. Establishment of human colon cancer cell lines from fresh tumors versus xenografts: comparison of success rate and cell line features. , 2007, Cancer research.
[33] Nina M. Muñoz,et al. Transforming Growth Factor β Receptor Type II Inactivation Induces the Malignant Transformation of Intestinal Neoplasms Initiated by Apc Mutation , 2006 .
[34] Ronald A. DePinho,et al. Model organisms: The mighty mouse: genetically engineered mouse models in cancer drug development , 2006, Nature Reviews Drug Discovery.
[35] Ji Luo,et al. The evolution of phosphatidylinositol 3-kinases as regulators of growth and metabolism , 2006, Nature Reviews Genetics.
[36] Jennifer J. Lund,et al. Adenomatous Polyposis Coli (APC) Is Required for Normal Development of Skin and Thymus , 2006, PLoS genetics.
[37] H. Cooper,et al. Generation of a unique strain of multiple intestinal neoplasia (Apc+/Min‐FCCC) mice with significantly increased numbers of colorectal adenomas , 2005, Molecular carcinogenesis.
[38] H. Clevers,et al. Cyclin D1 Is Not an Immediate Target of β-Catenin following Apc Loss in the Intestine* , 2005, Journal of Biological Chemistry.
[39] Perry J Pickhardt,et al. Microcomputed tomography colonography for polyp detection in an in vivo mouse tumor model. , 2005, Proceedings of the National Academy of Sciences of the United States of America.
[40] M. Giovannini,et al. Colorectal cancers in a new mouse model of familial adenomatous polyposis: influence of genetic and environmental modifiers , 2004, Laboratory Investigation.
[41] Daniel Metzger,et al. Tissue‐specific and inducible Cre‐mediated recombination in the gut epithelium , 2004, Genesis.
[42] O. Sansom,et al. Inducible Cre-mediated control of gene expression in the murine gastrointestinal tract: effect of loss of beta-catenin. , 2004, Gastroenterology.
[43] Makoto M Taketo,et al. Colonic polyposis caused by mTOR-mediated chromosomal instability in Apc+/Δ716 Cdx2+/− compound mutant mice , 2003, Nature Genetics.
[44] L. Seymour,et al. Clinical predictive value of the in vitro cell line, human xenograft, and mouse allograft preclinical cancer models. , 2003, Clinical cancer research : an official journal of the American Association for Cancer Research.
[45] F. Pierre,et al. Point: From animal models to prevention of colon cancer. Systematic review of chemoprevention in min mice and choice of the model system. , 2003, Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology.
[46] K. Washington,et al. Pathology of mouse models of intestinal cancer: consensus report and recommendations. , 2003, Gastroenterology.
[47] L. Ellis,et al. Effect of the vascular endothelial growth factor receptor‐2 antibody DC101 plus gemcitabine on growth, metastasis and angiogenesis of human pancreatic cancer growing orthotopically in nude mice , 2002, International journal of cancer.
[48] Takuji Tanaka,et al. Enhanced colon carcinogenesis induced by azoxymethane in min mice occurs via a mechanism independent of beta-catenin mutation. , 2002, Cancer letters.
[49] B. Spiegelman,et al. A Phase II Study of Troglitazone, an Activator of the PPARγ Receptor, in Patients with Chemotherapy‐Resistant Metastatic Colorectal Cancer , 2002 .
[50] K. Forde,et al. Colonoscopy in Mice , 2001, Surgical Endoscopy And Other Interventional Techniques.
[51] J. Gordon,et al. Inducible Gene Knockouts in the Small Intestinal and Colonic Epithelium* , 1999, The Journal of Biological Chemistry.
[52] Samuel Singer,et al. Differentiation and reversal of malignant changes in colon cancer through PPARγ , 1998, Nature Medicine.
[53] S. Baba,et al. Piroxicam and Acarbose as Chemopreventive Agents for Spontaneous Intestinal Adenomas in APC Gene 1309 Knockout Mice , 1998, Japanese journal of cancer research : Gann.
[54] T. Noda,et al. Rapid colorectal adenoma formation initiated by conditional targeting of the Apc gene. , 1997, Science.
[55] R. Kucherlapati,et al. Loss of Apc and the entire chromosome 18 but absence of mutations at the Ras and Tp53 genes in intestinal tumors from Apc1638N, a mouse model for Apc-driven carcinogenesis. , 1997, Carcinogenesis.
[56] A. Moser,et al. Homozygosity for the Minallele of Apc results in disruption of mouse development prior to gastrulation , 1995, Developmental dynamics : an official publication of the American Association of Anatomists.
[57] M Oshima,et al. Loss of Apc heterozygosity and abnormal tissue building in nascent intestinal polyps in mice carrying a truncated Apc gene. , 1995, Proceedings of the National Academy of Sciences of the United States of America.
[58] R Fodde,et al. A targeted chain-termination mutation in the mouse Apc gene results in multiple intestinal tumors. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[59] K. Rajewsky,et al. Deletion of a DNA polymerase beta gene segment in T cells using cell type-specific gene targeting. , 1994, Science.
[60] Bert Vogelstein,et al. APC mutations occur early during colorectal tumorigenesis , 1992, Nature.
[61] K. Kinzler,et al. Erratum: Multiple Intestinal Neoplasia Caused By a Mutation in the Murine Homolog of the APC Gene , 1992, Science.
[62] K. Kinzler,et al. Identification of a gene located at chromosome 5q21 that is mutated in colorectal cancers. , 1991, Science.
[63] H. Pitot,et al. A dominant mutation that predisposes to multiple intestinal neoplasia in the mouse. , 1990, Science.