Sulfur mustard induces differentiation in human primary keratinocytes: opposite roles of p38 and ERK1/2 MAPK.
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A. Schmidt | K. Kehe | M. Jochum | D. Steinritz | T. Popp | V. Egea | C. Ries | Virginia Egea
[1] Taro Matsumoto,et al. p38 MAP kinase negatively regulates endothelial cell survival, proliferation, and differentiation in FGF-2–stimulated angiogenesis , 2002, The Journal of cell biology.
[2] P. Neth,et al. Scientific category: Stem cells in hematology MMP-2, MT1-MMP, and TIMP-2 are essential for the invasive capacity of human mesenchymal stem cells: differential regulation by inflammatory cytokines , 2006 .
[3] M. Yaffe,et al. Evidence that involucrin is a covalently linked constituent of highly purified cultured keratinocyte cornified envelopes. , 1993, The Journal of investigative dermatology.
[4] R. Pietro,et al. Regulation of CREB Activation by P38 Mitogen Activated Protein Kinase during Human Primary Erythroblast Differentiation , 2009, International journal of immunopathology and pharmacology.
[5] J. Avruch,et al. Mammalian mitogen-activated protein kinase signal transduction pathways activated by stress and inflammation. , 2001, Physiological reviews.
[6] J. Blenis,et al. ERK and p38 MAPK-Activated Protein Kinases: a Family of Protein Kinases with Diverse Biological Functions , 2004, Microbiology and Molecular Biology Reviews.
[7] C. Simbulan-Rosenthal,et al. Sulfur mustard induces markers of terminal differentiation and apoptosis in keratinocytes via a Ca2+-calmodulin and caspase-dependent pathway. , 1998, The Journal of investigative dermatology.
[8] M. Mahmoudi,et al. Long‐term complications of sulphur mustard poisoning in severely intoxicated Iranian veterans , 2005, Fundamental & clinical pharmacology.
[9] M. Cobb,et al. Mitogen-activated protein (MAP) kinase pathways: regulation and physiological functions. , 2001, Endocrine reviews.
[10] R. Eckert,et al. Regulation of involucrin gene expression. , 2004, The Journal of investigative dermatology.
[11] K. Kehe,et al. Matrix metalloproteinase-9 expression and release from skin fibroblasts interacting with keratinocytes: Upregulation in response to sulphur mustard. , 2009, Toxicology.
[12] Kriston L. McGary,et al. An inhibitor of p38 MAP kinase downregulates cytokine release induced by sulfur mustard exposure in human epidermal keratinocytes. , 2004, Toxicology in vitro : an international journal published in association with BIBRA.
[13] D. Laskin,et al. Role of MAP kinases in regulating expression of antioxidants and inflammatory mediators in mouse keratinocytes following exposure to the half mustard, 2-chloroethyl ethyl sulfide. , 2010, Toxicology and applied pharmacology.
[14] M. Tsujimoto,et al. Involvement of p38 Mitogen-activated Protein Kinase Signaling Pathway in Osteoclastogenesis Mediated by Receptor Activator of NF-κB Ligand (RANKL)* , 2000, The Journal of Biological Chemistry.
[15] P. Neth,et al. Wnt Signaling Regulates the Invasion Capacity of Human Mesenchymal Stem Cells , 2006, Stem cells.
[16] G. Mouchiroud,et al. Macrophage differentiation of myeloid progenitor cells in response to M‐CSF is regulated by the dual‐specificity phosphatase DUSP5 , 2010, Journal of leukocyte biology.
[17] Rainer Schmidt,et al. The cornified envelope: a model of cell death in the skin , 2005, Nature Reviews Molecular Cell Biology.
[18] Anne Deucher,et al. Keratinocyte survival, differentiation, and death: many roads lead to mitogen-activated protein kinase. , 2002, The journal of investigative dermatology. Symposium proceedings.
[19] Juan Peng,et al. Inhibition of p38 MAPK facilitates ex vivo expansion of skin epithelial progenitor cells , 2009, In Vitro Cellular & Developmental Biology - Animal.
[20] T. Kuroki,et al. Novel Protein Kinase C Isoforms Regulate Human Keratinocyte Differentiation by Activating a p38δ Mitogen-activated Protein Kinase Cascade That Targets CCAAT/Enhancer-binding Protein α* , 2002, The Journal of Biological Chemistry.
[21] S. Medicherla,et al. p38 MAPK inhibition reduces diabetes-induced impairment of wound healing , 2009, Diabetes, metabolic syndrome and obesity : targets and therapy.
[22] R. Eckert,et al. Involucrin is a covalently crosslinked constituent of highly purified epidermal corneocytes: evidence for a common pattern of involucrin crosslinking in vivo and in vitro. , 1996, The Journal of investigative dermatology.
[23] F. Watt,et al. Epidermal stem cell diversity and quiescence , 2009, EMBO molecular medicine.
[24] H. Pehamberger,et al. The hsp27kD heat shock protein and p38-MAPK signaling are required for regular epidermal differentiation. , 2011, Journal of dermatological science.
[25] A. Finlay,et al. Numerous keratinocyte subtypes involved in wound re-epithelialization. , 2006, The Journal of investigative dermatology.
[26] Kenneth R Feingold,et al. Modulations in epidermal calcium regulate the expression of differentiation-specific markers. , 2002, The Journal of investigative dermatology.
[27] R. Rupec,et al. Role of NF-kappaB/RelA and MAPK pathways in keratinocytes in response to sulfur mustard. , 2008, The Journal of investigative dermatology.
[28] Jung Weon Lee,et al. Cdc42-dependent Mediation of UV-induced p38 Activation by G Protein βγ Subunits* , 2004, Journal of Biological Chemistry.
[29] T. Shibahara,et al. Morphological evidence of basal keratinocyte migration during the re-epithelialization process , 2008, Histochemistry and Cell Biology.
[30] S. Pelech,et al. Stress-induced Inhibition of ERK1 and ERK2 by Direct Interaction with p38 MAP Kinase* , 2001, The Journal of Biological Chemistry.
[31] R. Eckert,et al. A Regulatory Role for p38δ MAPK in Keratinocyte Differentiation , 2003, Journal of Biological Chemistry.
[32] R. Ray,et al. A Convenient Fluorometric Method to Study Sulfur Mustard–Induced Apoptosis in Human Epidermal Keratinocytes Monolayer Microplate Culture , 2005, Drug and chemical toxicology.
[33] Karen Holbrook,et al. Calcium regulation of growth and differentiation of mouse epidermal cells in culture , 1980, Cell.
[34] G. Dotto,et al. p21(WAF1/Cip1) functions as a suppressor of malignant skin tumor formation and a determinant of keratinocyte stem-cell potential. , 1999, Proceedings of the National Academy of Sciences of the United States of America.
[35] H. Mukhtar,et al. Inhibition of UVB-induced oxidative stress-mediated phosphorylation of mitogen-activated protein kinase signaling pathways in cultured human epidermal keratinocytes by green tea polyphenol (-)-epigallocatechin-3-gallate. , 2001, Toxicology and applied pharmacology.
[36] K. Kehe,et al. Medical aspects of sulphur mustard poisoning. , 2005, Toxicology.
[37] D. Bikle,et al. Calcium regulation of growth and differentiation of normal human keratinocytes: Modulation of differentiation competence by stages of growth and extracellular calcium , 1990, Journal of cellular physiology.
[38] C. H. Goh,et al. p38 mitogen-activated protein kinase regulates osteoblast differentiation through osterix. , 2007, Endocrinology.
[39] P. Rice. Sulphur Mustard Injuries of the Skin , 2003, Toxicological reviews.
[40] C. G. Hurst,et al. Wound Healing of Cutaneous Sulfur Mustard Injuries: Strategies for the Development of Improved Therapies , 2005 .
[41] P. Mansouri,et al. A clinicopathological study on acute cutaneous lesions induced by sulfur mustard gas (yperite). , 2005, European journal of dermatology : EJD.
[42] Thomas Mammone,et al. SIRT1 promotes differentiation of normal human keratinocytes. , 2009, The Journal of investigative dermatology.
[43] J. Dillman,et al. Sulfur mustard induced cytokine production and cell death: Investigating the potential roles of the p38, p53, and NF‐κB signaling pathways with RNA interference , 2010, Journal of biochemical and molecular toxicology.
[44] C. Stewart,et al. PD98059 enhances C2 myoblast differentiation through p38 MAPK activation: a novel role for PD98059. , 2008, The Journal of endocrinology.