Linker Histones Are Mobilized during Infection with Herpes Simplex Virus Type 1
暂无分享,去创建一个
[1] T. Haneji,et al. Histone H1.2 is translocated to mitochondria and associates with bak in bleomycin‐induced apoptotic cells , 2008, Journal of cellular biochemistry.
[2] V. Connor,et al. Histone modifications associated with herpes simplex virus type 1 genomes during quiescence and following ICP0-mediated de-repression , 2008, The Journal of general virology.
[3] N. Fraser,et al. Temporal Association of the Herpes Simplex Virus Genome with Histone Proteins during a Lytic Infection , 2007, Journal of Virology.
[4] B. Roizman,et al. Herpes simplex virus-infected cell protein 0 blocks the silencing of viral DNA by dissociating histone deacetylases from the CoREST–REST complex , 2007, Proceedings of the National Academy of Sciences.
[5] R. Everett,et al. Herpes Simplex Virus Type 1 Genomes Are Associated with ND10 Nuclear Substructures in Quiescently Infected Human Fibroblasts , 2007, Journal of Virology.
[6] P. Lomonte,et al. Centromeric protein CENP‐B proteasomal degradation induced by the viral protein ICP0 , 2007, FEBS letters.
[7] Minoru Yoshida,et al. [Potent and specific inhibition of mammalian histone deacetylase both in vivo and in vitro by trichostatin A]. , 1990, Tanpakushitsu kakusan koso. Protein, nucleic acid, enzyme.
[8] S. Weller,et al. Herpes simplex virus type I disrupts the ATR-dependent DNA-damage response during lytic infection , 2006, Journal of Cell Science.
[9] S. Berger,et al. Trimethylation of Histone H3 Lysine 4 by Set1 in the Lytic Infection of Human Herpes Simplex Virus 1 , 2006, Journal of Virology.
[10] J. Harper,et al. Identification and Functional Evaluation of Cellular and Viral Factors Involved in the Alteration of Nuclear Architecture during Herpes Simplex Virus 1 Infection , 2005, Journal of Virology.
[11] G. Eriani,et al. Expression of metazoan replication-dependent histone genes. , 2005, Biochimie.
[12] Noriko Shirata,et al. Activation of Ataxia Telangiectasia-mutated DNA Damage Checkpoint Signal Transduction Elicited by Herpes Simplex Virus Infection* , 2005, Journal of Biological Chemistry.
[13] M. Hendzel,et al. H1 Family Histones in the Nucleus , 2005, Journal of Biological Chemistry.
[14] Alain Verreault,et al. Regulation of histone synthesis and nucleosome assembly. , 2005, Biochimie.
[15] S. Weller,et al. Inhibition of the Herpes Simplex Virus Type 1 DNA Polymerase Induces Hyperphosphorylation of Replication Protein A and Its Accumulation at S-Phase-Specific Sites of DNA Damage during Infection , 2005, Journal of Virology.
[16] Bernard Roizman,et al. Components of the REST/CoREST/histone deacetylase repressor complex are disrupted, modified, and translocated in HSV-1-infected cells. , 2005, Proceedings of the National Academy of Sciences of the United States of America.
[17] Sue Biggins,et al. Histone variants: deviants? , 2005, Genes & development.
[18] S. Berger,et al. During Lytic Infection Herpes Simplex Virus Type 1 Is Associated with Histones Bearing Modifications That Correlate with Active Transcription , 2004, Journal of Virology.
[19] F. J. Herrera,et al. VP16-Dependent Association of Chromatin-Modifying Coactivators and Underrepresentation of Histones at Immediate-Early Gene Promoters during Herpes Simplex Virus Infection , 2004, Journal of Virology.
[20] A. Epstein,et al. Functional Interaction between Class II Histone Deacetylases and ICP0 of Herpes Simplex Virus Type 1 , 2004, Journal of Virology.
[21] D. Knipe,et al. Herpes Simplex Virus 1 UL31 and UL34 Gene Products Promote the Late Maturation of Viral Replication Compartments to the Nuclear Periphery , 2004, Journal of Virology.
[22] T. Misteli,et al. Network of Dynamic Interactions between Histone H1 and High-Mobility-Group Proteins in Chromatin , 2004, Molecular and Cellular Biology.
[23] M. Hendzel,et al. The C-terminal Domain Is the Primary Determinant of Histone H1 Binding to Chromatin in Vivo* , 2004, Journal of Biological Chemistry.
[24] S. Weller,et al. Recruitment of Cellular Recombination and Repair Proteins to Sites of Herpes Simplex Virus Type 1 DNA Replication Is Dependent on the Composition of Viral Proteins within Prereplicative Sites and Correlates with the Induction of the DNA Damage Response , 2004, Journal of Virology.
[25] S. Bachenheimer,et al. Synthesis and metabolism of cellular transcripts in HSV-1 infected cells , 1988, Virus Genes.
[26] Y. Tsujimoto,et al. Involvement of Histone H1.2 in Apoptosis Induced by DNA Double-Strand Breaks , 2003, Cell.
[27] Travis J Taylor,et al. Herpes simplex virus replication compartments can form by coalescence of smaller compartments. , 2003, Virology.
[28] W. Herr,et al. Human Sin3 deacetylase and trithorax-related Set1/Ash2 histone H3-K4 methyltransferase are tethered together selectively by the cell-proliferation factor HCF-1. , 2003, Genes & development.
[29] A. Belmont,et al. Sequential Recruitment of HAT and SWI/SNF Components to Condensed Chromatin by VP16 , 2003, Current Biology.
[30] R. Duronio,et al. Histone mRNA expression: multiple levels of cell cycle regulation and important developmental consequences. , 2002, Current opinion in cell biology.
[31] G. Sourvinos,et al. Visualization of parental HSV‐1 genomes and replication compartments in association with ND10 in live infected cells , 2002, The EMBO journal.
[32] T. Misteli,et al. Competition between histone H1 and HMGN proteins for chromatin binding sites , 2002, EMBO reports.
[33] V. Connor,et al. Herpes Simplex Virus Type 1 Promoter Activity during Latency Establishment, Maintenance, and Reactivation in Primary Dorsal Root Neurons In Vitro , 2001, Journal of Virology.
[34] K. Sullivan,et al. Degradation of Nucleosome-associated Centromeric Histone H3-like Protein CENP-A Induced by Herpes Simplex Virus Type 1 Protein ICP0* , 2001, The Journal of Biological Chemistry.
[35] M. Hendzel,et al. Rapid exchange of histone H1.1 on chromatin in living human cells , 2000, Nature.
[36] Tom Misteli,et al. Dynamic binding of histone H1 to chromatin in living cells , 2000, Nature.
[37] Peter Ghazal,et al. Annexation of the interchromosomal space during viral infection , 2000, Nature Cell Biology.
[38] D. Bazett-Jones,et al. Mitotic Transcription Repression in Vivo in the Absence of Nucleosomal Chromatin Condensation , 2000, The Journal of cell biology.
[39] P. Schaffer,et al. Roscovitine, a Specific Inhibitor of Cellular Cyclin-Dependent Kinases, Inhibits Herpes Simplex Virus DNA Synthesis in the Presence of Viral Early Proteins , 2000, Journal of Virology.
[40] K. Mossman,et al. Truncation of the C-Terminal Acidic Transcriptional Activation Domain of Herpes Simplex Virus VP16 Renders Expression of the Immediate-Early Genes Almost Entirely Dependent on ICP0 , 1999, Journal of Virology.
[41] G. Sudlow,et al. Large-Scale Chromatin Unfolding and Remodeling Induced by VP16 Acidic Activation Domain , 1999, The Journal of cell biology.
[42] C. Panagiotidis,et al. The host-cell architectural protein HMG I(Y) modulates binding of herpes simplex virus type 1 ICP4 to its cognate promoter. , 1999, Virology.
[43] W. Earnshaw,et al. Specific destruction of kinetochore protein CENP‐C and disruption of cell division by herpes simplex virus immediate‐early protein Vmw110 , 1999, The EMBO journal.
[44] W. Helliger,et al. In Vitro Binding of H1 Histone Subtypes to Nucleosomal Organized Mouse Mammary Tumor Virus Long Terminal Repeat Promotor* , 1998, The Journal of Biological Chemistry.
[45] N. DeLuca,et al. Persistence and Expression of the Herpes Simplex Virus Genome in the Absence of Immediate-Early Proteins , 1998, Journal of Virology.
[46] M. J. Nicholl,et al. Repression of gene expression upon infection of cells with herpes simplex virus type 1 mutants impaired for immediate-early protein synthesis , 1997, Journal of virology.
[47] S. Weller,et al. Herpes simplex virus type 1 prereplicative sites are a heterogeneous population: only a subset are likely to be precursors to replication compartments , 1997, Journal of virology.
[48] C. Spencer,et al. Repression of host RNA polymerase II transcription by herpes simplex virus type 1 , 1997, Journal of virology.
[49] G. Maul,et al. The periphery of nuclear domain 10 (ND10) as site of DNA virus deposition , 1996, The Journal of cell biology.
[50] R. Everett,et al. Nuclear domain 10 as preexisting potential replication start sites of herpes simplex virus type-1. , 1996, Virology.
[51] P. Schaffer,et al. An activity specified by the osteosarcoma line U2OS can substitute functionally for ICP0, a major regulatory protein of herpes simplex virus type 1 , 1995, Journal of virology.
[52] B. Hamkalo,et al. A proposal for a coherent mammalian histone H1 nomenclature correlated with amino acid sequences , 1994, Protein science : a publication of the Protein Society.
[53] W. Albig,et al. Organization and expression of H1 histone and H1 replacement histone genes , 1994, Journal of cellular biochemistry.
[54] P. Schaffer,et al. Herpes simplex virus type 1 ICP0 regulates expression of immediate-early, early, and late genes in productively infected cells , 1992, Journal of virology.
[55] C. Sorenson,et al. Analysis of herpes simplex virus-induced mRNA destabilizing activity using an in vitro mRNA decay system. , 1991, Nucleic acids research.
[56] R. Harris,et al. Establishment of latency in vitro by the herpes simplex virus type 1 mutant in1814. , 1991, The Journal of general virology.
[57] C. Ascoli,et al. Identification of a novel nuclear domain , 1991, The Journal of cell biology.
[58] C. Smibert,et al. Differential regulation of endogenous and transduced beta-globin genes during infection of erythroid cells with a herpes simplex virus type 1 recombinant , 1990, Journal of virology.
[59] P. Schaffer,et al. Herpes simplex virus type 1 ICP0 plays a critical role in the de novo synthesis of infectious virus following transfection of viral DNA , 1989, Journal of virology.
[60] S. Deshmane,et al. During latency, herpes simplex virus type 1 DNA is associated with nucleosomes in a chromatin structure , 1989, Journal of virology.
[61] C. Smibert,et al. Expression of a cellular gene cloned in herpes simplex virus: rabbit beta-globin is regulated as an early viral gene in infected fibroblasts , 1987, Journal of virology.
[62] E. Wagner,et al. RNA complementary to a herpesvirus alpha gene mRNA is prominent in latently infected neurons. , 1987, Science.
[63] M. Muggeridge,et al. Chromosomal organization of the herpes simplex virus genome during acute infection of the mouse central nervous system , 1986, Journal of virology.
[64] R. Everett. Trans activation of transcription by herpes virus products: requirement for two HSV‐1 immediate‐early polypeptides for maximum activity. , 1984, The EMBO journal.
[65] L. H. Cohen,et al. The histone H1 complements of dividing and nondividing cells of the mouse. , 1983, The Journal of biological chemistry.
[66] J. Pehrson,et al. Histone H1 subfractions and H10 turnover at different rates in nondividing cells. , 1982, Biochemistry.
[67] W. Summers,et al. The structure of herpes simplex virus type 1 DNA as probed by micrococcal nuclease digestion. , 1980, The Journal of general virology.
[68] W. Kistler,et al. H1 histone subfractions of mammalian testes. 1. Organ specificity in the rat. , 1979, Biochemistry.
[69] D. Guétard,et al. Random cleavage of intranuclear herpes simplex virus DNA by micrococcal nuclease , 1979, FEBS letters.
[70] K. Smith,et al. Relationship Between the Envelope and the Infectivity of Herpes Simplex Virus.∗ , 1964, Proceedings of the Society for Experimental Biology and Medicine. Society for Experimental Biology and Medicine.