TGFB1 genetic polymorphisms and coronary heart disease risk: a meta-analysis
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J. Boer | E. Feskens | A. Goel | O. Favorova | Yingchang Lu | Michael Müller | Roza M Barsova | A. Goel
[1] S. Ghaderian,et al. Association of Transforming Growth Factor-&bgr;1 Gene Polymorphisms With Genetic Susceptibility to Acute Myocardial Infarction , 2011, The American journal of the medical sciences.
[2] Claudio J. Verzilli,et al. Comparative analysis of genome-wide association studies signals for lipids, diabetes, and coronary heart disease: Cardiovascular Biomarker Genetics Collaboration , 2011, European heart journal.
[3] N. Mehta. Large-scale association analysis identifies 13 new susceptibility loci for coronary artery disease. , 2011, Circulation. Cardiovascular genetics.
[4] G. Vriend,et al. Mutations in SMAD3 cause a syndromic form of aortic aneurysms and dissections with early-onset osteoarthritis , 2011, Nature Genetics.
[5] Mark I. McCarthy,et al. A genome-wide association study in Europeans and South Asians identifies five new loci for coronary artery disease , 2011, Nature Genetics.
[6] Scott Mellis,et al. Transforming Growth Factor-&bgr; Signaling Pathway in Patients With Kawasaki Disease , 2011, Circulation. Cardiovascular genetics.
[7] S. Leal,et al. Rare copy number variants disrupt genes regulating vascular smooth muscle cell adhesion and contractility in sporadic thoracic aortic aneurysms and dissections. , 2010, American journal of human genetics.
[8] L. Palmer,et al. Association of an allele on chromosome 9 and abdominal aortic aneurysm. , 2010, Atherosclerosis.
[9] Marie-José Goumans,et al. Signaling by members of the TGF-beta family in vascular morphogenesis and disease. , 2010, Trends in cell biology.
[10] Wolfgang Viechtbauer,et al. Conducting Meta-Analyses in R with the metafor Package , 2010 .
[11] X. Chu,et al. Common polymorphisms in ITGA2, PON1 and THBS2 are associated with coronary atherosclerosis in a candidate gene association study of the Chinese Han population , 2010, Journal of Human Genetics.
[12] A. Hata,et al. Differential Effects of Chromosome 9p21 Variation on Subphenotypes of Intracranial Aneurysm: Site Distribution , 2010, Stroke.
[13] J. Marchini,et al. Genotype imputation for genome-wide association studies , 2010, Nature Reviews Genetics.
[14] Alexander V. Favorov,et al. Complex analysis of association of inflammation gene polymorphisms with myocardial infarction , 2010, Molecular Biology.
[15] Alexander V. Favorov,et al. [Complex analsis of association of inflammation genes with myocardial infarction]. , 2010, Molekuliarnaia biologiia.
[16] L. Baum,et al. Independent predictive roles of eotaxin Ala23Thr, paraoxonase 2 Ser311Cys and β3‐adrenergic receptor Trp64Arg polymorphisms on cardiac disease in Type 2 Diabetes—an 8‐year prospective cohort analysis of 1297 patients , 2010, Diabetic medicine : a journal of the British Diabetic Association.
[17] Alejandro F. Frangi,et al. Genome-wide association study of intracranial aneurysm identifies three new risk loci , 2010, Nature genetics.
[18] D. Dichek,et al. TGF-&bgr;1 Limits Plaque Growth, Stabilizes Plaque Structure, and Prevents Aortic Dilation in Apolipoprotein E–Null Mice , 2009, Arteriosclerosis, thrombosis, and vascular biology.
[19] H. Dietz,et al. Circulating Transforming Growth Factor-&bgr; in Marfan Syndrome , 2009, Circulation.
[20] L. Smeeth,et al. Integrated associations of genotypes with multiple blood biomarkers linked to coronary heart disease risk , 2009, Human molecular genetics.
[21] G. Mintz,et al. Effect of culprit-lesion remodeling versus plaque rupture on three-year outcome in patients with acute coronary syndrome. , 2009, The American journal of cardiology.
[22] J. Golledge,et al. Sequence variant on 9p21 is associated with the presence of abdominal aortic aneurysm disease but does not have an impact on aneurysmal expansion , 2009, European Journal of Human Genetics.
[23] D. Cokkinos,et al. Inflammatory cytokine gene variants in coronary artery disease patients in Greece , 2008, Coronary artery disease.
[24] Praveen Sethupathy,et al. MicroRNA target site polymorphisms and human disease. , 2008, Trends in genetics : TIG.
[25] A. Piazza,et al. Role of TGF-β1 haplotypes in the occurrence of myocardial infarction in young Italian patients , 2008, BMC Medical Genetics.
[26] H. Stefánsson,et al. The same sequence variant on 9p21 associates with myocardial infarction, abdominal aortic aneurysm and intracranial aneurysm , 2008, Nature Genetics.
[27] C. Gieger,et al. Genomewide association analysis of coronary artery disease. , 2007, The New England journal of medicine.
[28] A. Gylfason,et al. A Common Variant on Chromosome 9p21 Affects the Risk of Myocardial Infarction , 2007, Science.
[29] A. Agrotis,et al. Understanding the role of transforming growth factor-beta1 in intimal thickening after vascular injury. , 2007, Cardiovascular research.
[30] Kanae Oda,et al. Polymorphisms in pro- and anti-inflammatory cytokine genes and susceptibility to atherosclerosis: a pathological study of 1503 consecutive autopsy cases. , 2007, Human molecular genetics.
[31] Jonathan C. Cohen,et al. A Common Allele on Chromosome 9 Associated with Coronary Heart Disease , 2007, Science.
[32] A. Hofman,et al. TGF-&bgr;1 Polymorphisms and Risk of Myocardial Infarction and Stroke: The Rotterdam Study , 2006, Stroke.
[33] A. Kastrati,et al. Association of Transforming Growth Factor-&bgr;1 Gene Polymorphisms With Myocardial Infarction in Patients With Angiographically Proven Coronary Heart Disease , 2006, Arteriosclerosis, thrombosis, and vascular biology.
[34] Marc K. Halushka,et al. Losartan, an AT1 Antagonist, Prevents Aortic Aneurysm in a Mouse Model of Marfan Syndrome , 2006, Science.
[35] O. Tokunaga,et al. Significant Expression of Endoglin (CD105), TGFβ-1 and TGFβ R-2 in the Atherosclerotic Aorta: An Immunohistological Study , 2006 .
[36] C. Hurley,et al. Allelic diversity in the TGFB1 regulatory region: characterization of novel functional single nucleotide polymorphisms , 2006, Human Genetics.
[37] Wolfram Kress,et al. A syndrome of altered cardiovascular, craniofacial, neurocognitive and skeletal development caused by mutations in TGFBR1 or TGFBR2 , 2005, Nature Genetics.
[38] Ammarin Thakkinstian,et al. Systematic review and meta-analysis of the association between {beta}2-adrenoceptor polymorphisms and asthma: a HuGE review. , 2005, American journal of epidemiology.
[39] S. Shete,et al. Mutations in Transforming Growth Factor-&bgr; Receptor Type II Cause Familial Thoracic Aortic Aneurysms and Dissections , 2005, Circulation.
[40] D. Spandidos,et al. Protein and mRNA expression levels of VEGF-A and TGF-beta1 in different types of human coronary atherosclerotic lesions. , 2005, International journal of molecular medicine.
[41] J. Erdmann,et al. Distinct Heritable Patterns of Angiographic Coronary Artery Disease in Families With Myocardial Infarction , 2005, Circulation.
[42] B. Jaber,et al. Transforming growth factor-beta 1 gene polymorphisms and cardiovascular disease in hemodialysis patients. , 2004, Kidney international.
[43] J. Chang-Claude,et al. A Transforming Growth Factorβ1 Signal Peptide Variant Increases Secretion in Vitro and Is Associated with Increased Incidence of Invasive Breast Cancer , 2003 .
[44] R. Siegel,et al. Coronary remodeling of proximal and distal stenotic atherosclerotic plaques within the same artery by intravascular ultrasound study. , 2001, The American journal of cardiology.
[45] M. Yokota,et al. Association of a T29-->C polymorphism of the transforming growth factor-beta1 gene with genetic susceptibility to myocardial infarction in Japanese. , 2000, Circulation.
[46] S Duval,et al. Trim and Fill: A Simple Funnel‐Plot–Based Method of Testing and Adjusting for Publication Bias in Meta‐Analysis , 2000, Biometrics.
[47] V. Sharma,et al. Transforming growth factor-β1 hyperexpression in African-American hypertensives: A novel mediator of hypertension and/or target organ damage , 2000 .
[48] S E Nissen,et al. Extent and direction of arterial remodeling in stable versus unstable coronary syndromes : an intravascular ultrasound study. , 2000, Circulation.
[49] M. Condron,et al. Distinct patterns of transforming growth factor-beta isoform and receptor expression in human atherosclerotic lesions. Colocalization implicates TGF-beta in fibrofatty lesion development. , 1999, Circulation.
[50] J. Gunn,et al. Transforming growth factor-beta1 gene polymorphisms and coronary artery disease. , 1998, Clinical science.
[51] D. Wilcken,et al. A common polymorphism of the transforming growth factor-beta1 gene and coronary artery disease. , 1998, Clinical science.
[52] J. Gunn,et al. Transforming growth factor-beta1 gene polymorphisms and coronary artery disease. , 1998, Clinical science.
[53] K. Ikeda,et al. Association of a Polymorphism of the Transforming Growth Factor‐β1 Gene with Genetic Susceptibility to Osteoporosis in Postmenopausal Japanese Women , 1998, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.
[54] R. Virmani,et al. Overexpression of transforming growth factor beta1 in arterial endothelium causes hyperplasia, apoptosis, and cartilaginous metaplasia. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[55] D. Arveiler,et al. Polymorphisms of the transforming growth factor-beta 1 gene in relation to myocardial infarction and blood pressure. The Etude Cas-Témoin de l'Infarctus du Myocarde (ECTIM) Study. , 1996, Hypertension.
[56] T. Zderic,et al. βig-h3, a Transforming Growth Factor–β–Inducible Gene, Is Overexpressed in Atherosclerotic and Restenotic Human Vascular Lesions , 1996 .
[57] T. McCaffrey,et al. Decreased type II/type I TGF-beta receptor ratio in cells derived from human atherosclerotic lesions. Conversion from an antiproliferative to profibrotic response to TGF-beta1. , 1995, The Journal of clinical investigation.
[58] R. Derynck,et al. Direct transfer of transforming growth factor beta 1 gene into arteries stimulates fibrocellular hyperplasia. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[59] J. Hermiller,et al. In vivo validation of compensatory enlargement of atherosclerotic coronary arteries. , 1993, The American journal of cardiology.
[60] J. Isner,et al. Expression of transforming growth factor-beta 1 is increased in human vascular restenosis lesions. , 1992, The Journal of clinical investigation.
[61] S. Schwartz,et al. Production of transforming growth factor beta 1 during repair of arterial injury. , 1991, The Journal of clinical investigation.
[62] J. Schofer,et al. Impact of compensatory enlargement of atherosclerotic coronary arteries on angiographic assessment of coronary artery disease. , 1989, Circulation.
[63] C. Zarins,et al. Compensatory enlargement of human atherosclerotic coronary arteries. , 1987, The New England journal of medicine.
[64] G. Moneta. Rare Copy Number Variants Disrupt Genes Regulating Vascular Smooth Muscle Cell Adhesion and Contractility in Sporadic Thoracic Aortic Aneurysms and Dissections , 2011 .
[65] Yi Wang,et al. BMC Medical Genetics , 2007 .
[66] O. Tokunaga,et al. Significant expression of endoglin (CD105), TGFbeta-1 and TGFbeta R-2 in the atherosclerotic aorta: an immunohistological study. , 2006, Journal of atherosclerosis and thrombosis.
[67] J. Chang-Claude,et al. A transforming growth factorbeta1 signal peptide variant increases secretion in vitro and is associated with increased incidence of invasive breast cancer. , 2003, Cancer research.
[68] M. Suthanthiran,et al. Transforming growth factor-beta 1 hyperexpression in African-American hypertensives: A novel mediator of hypertension and/or target organ damage. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[69] T. Spector,et al. Genetic control of the circulating concentration of transforming growth factor type beta1. , 1999, Human molecular genetics.
[70] T. Zderic,et al. Beta ig-h3, a transforming growth factor-beta-inducible gene, is overexpressed in atherosclerotic and restenotic human vascular lesions. , 1996, Arteriosclerosis, thrombosis, and vascular biology.
[71] A. Grace,et al. The serum concentration of active transforming growth factor-β is severely depressed in advanced atherosclerosis , 1995, Nature Medicine.
[72] R. Kerber,et al. Coronary arterial remodeling studied by high-frequency epicardial echocardiography: an early compensatory mechanism in patients with obstructive coronary atherosclerosis. , 1991, Journal of the American College of Cardiology.