Biomarker-based asthma phenotypes of corticosteroid response.
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S. Hazen | S. Erzurum | S. Comhair | J. Cowan | D. Taylor | J. Hammel | R. Palmay | D. Cowan | A. Williamson | Laura Peterson | Stanley L Hazen | Rochelle Palmay | Laura Peterson
[1] N. Wilson,et al. Longitudinal relationship between sputum eosinophils and exhaled nitric oxide in children with asthma. , 2013, American journal of respiratory and critical care medicine.
[2] Sandy Jung-Wu. Urinary Bromotyrosine Measures Asthma Control and Predicts Asthma Exacerbations in Children , 2012, Pediatrics.
[3] A. Bush,et al. Use of sputum eosinophil counts to guide management in children with severe asthma , 2012, Thorax.
[4] S. Wenzel,et al. Asthma outcomes: biomarkers. , 2012, The Journal of allergy and clinical immunology.
[5] M. Wegmann. Targeting eosinophil biology in asthma therapy. , 2011, American journal of respiratory cell and molecular biology.
[6] W. Busse,et al. Use of exhaled nitric oxide measurement to identify a reactive, at-risk phenotype among patients with asthma. , 2010, American journal of respiratory and critical care medicine.
[7] G. Roberts,et al. Influence of atopy and asthma on exhaled nitric oxide in an unselected birth cohort study , 2010, Thorax.
[8] S. Willsie. Mepolizumab and Exacerbations of Refractory Eosinophilic Asthma , 2010 .
[9] J. Cowan,et al. Effects of steroid therapy on inflammatory cell subtypes in asthma , 2009, Thorax.
[10] I. Pavord,et al. An official American Thoracic Society/European Respiratory Society statement: asthma control and exacerbations: standardizing endpoints for clinical asthma trials and clinical practice. , 2009, American journal of respiratory and critical care medicine.
[11] E. Israel,et al. Noninvasive Markers of Airway Inflammation in Asthma , 2009, Clinical and translational science.
[12] Ana Sousa,et al. Mepolizumab and exacerbations of refractory eosinophilic asthma. , 2009, The New England journal of medicine.
[13] J. L. Puckett,et al. Partitioned exhaled nitric oxide to non-invasively assess asthma , 2008, Respiratory Physiology & Neurobiology.
[14] A. Michils,et al. Exhaled nitric oxide and asthma control: a longitudinal study in unselected patients , 2008, European Respiratory Journal.
[15] C. Lemière,et al. Airway inflammation assessed by invasive and noninvasive means in severe asthma: eosinophilic and noneosinophilic phenotypes. , 2006, The Journal of allergy and clinical immunology.
[16] P. Paggiaro,et al. Low sputum eosinophils predict the lack of response to beclomethasone in symptomatic asthmatic patients. , 2006, Chest.
[17] G. Heldt,et al. Response profiles to fluticasone and montelukast in mild-to-moderate persistent childhood asthma. , 2006, The Journal of allergy and clinical immunology.
[18] I. Pavord,et al. The use of exhaled nitric oxide concentration to identify eosinophilic airway inflammation: an observational study in adults with asthma , 2005, Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology.
[19] Klas Svensson,et al. Measurement properties and interpretation of three shortened versions of the asthma control questionnaire. , 2005, Respiratory medicine.
[20] E. Israel,et al. Sputum eosinophil counts predict asthma control after discontinuation of inhaled corticosteroids. , 2005, The Journal of allergy and clinical immunology.
[21] D. Knight,et al. Epithelial inducible nitric oxide synthase activity is the major determinant of nitric oxide concentration in exhaled breath , 2004, Thorax.
[22] I. Pavord,et al. Comparison of airway immunopathology of eosinophilic bronchitis and asthma , 2003, Thorax.
[23] S. Anderson,et al. Methods for “Indirect” challenge tests including exercise, eucapnic voluntary hyperpnea, and hypertonic aerosols , 2003, Clinical reviews in allergy & immunology.
[24] I. Pavord,et al. Asthma exacerbations and sputum eosinophil counts: a randomised controlled trial , 2002, The Lancet.
[25] M. Segal,et al. Relationship between airway inflammation, hyperresponsiveness, and obstruction in asthma. , 2001, The Journal of allergy and clinical immunology.
[26] R. Hancox,et al. The predictive value of exhaled nitric oxide measurements in assessing changes in asthma control. , 2001, American journal of respiratory and critical care medicine.
[27] S. Hazen,et al. Eosinophils Are a Major Source of Nitric Oxide-Derived Oxidants in Severe Asthma: Characterization of Pathways Available to Eosinophils for Generating Reactive Nitrogen Species4 , 2001, The Journal of Immunology.
[28] R. Dweik,et al. NO chemical events in the human airway during the immediate and late antigen-induced asthmatic response , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[29] C. Jenkins,et al. Predictive markers of asthma exacerbation during stepwise dose reduction of inhaled corticosteroids. , 2001, American journal of respiratory and critical care medicine.
[30] E. Bacci,et al. Analysis of induced sputum before and after withdrawal of treatment with inhaled corticosteroids in asthmatic patients , 2000, Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology.
[31] S. Hazen,et al. Eosinophils generate brominating oxidants in allergen-induced asthma. , 2000, The Journal of clinical investigation.
[32] P. Barnes,et al. Changes in sputum eosinophils predict loss of asthma control. , 2000, American journal of respiratory and critical care medicine.
[33] G H Guyatt,et al. Development and validation of a questionnaire to measure asthma control. , 1999, The European respiratory journal.
[34] I. Pavord,et al. Non-eosinophilic cor ticosteroid unresponsive asthma , 1999, The Lancet.
[35] K. Chung,et al. Correlation between exhaled nitric oxide, sputum eosinophils, and methacholine responsiveness in patients with mild asthma. , 1998, Thorax.
[36] Third Expert Panel on theDiagnosis,et al. Expert Panel Report 3: Guidelines for the Diagnosis and Management of Asthma , 1997 .
[37] F. Hargreave,et al. Indices of airway inflammation in induced sputum: reproducibility and validity of cell and fluid-phase measurements. , 1996, American journal of respiratory and critical care medicine.
[38] H. Boushey,et al. Cellular and biochemical analysis of induced sputum from asthmatic and from healthy subjects. , 1993, The American review of respiratory disease.
[39] S. Holgate,et al. The effect of inhaled ipratropium bromide alone and in combination with oral terfenadine on bronchoconstriction provoked by adenosine 5'-monophosphate and histamine in asthma. , 1991, The Journal of allergy and clinical immunology.
[40] J. Bousquet,et al. Eosinophilic inflammation in asthma. , 1990, The New England journal of medicine.
[41] H. Brown. Treatment of chronic asthma with prednisolone; significance of eosinophils in the sputum. , 1958, Lancet.