Hypogelsolinemia, a disorder of the extracellular actin scavenger system, in patients with multiple sclerosis

[1]  Jeffrey A. Cohen,et al.  Diagnostic criteria for multiple sclerosis: 2010 Revisions to the McDonald criteria , 2011, Annals of neurology.

[2]  I. Ferrer,et al.  Cytoplasmic gelsolin increases mitochondrial activity and reduces Aβ burden in a mouse model of Alzheimer's disease , 2009, Neurobiology of Disease.

[3]  H. Hartung,et al.  Cerebrospinal fluid biomarkers in multiple sclerosis , 2009, Neurobiology of Disease.

[4]  M. Wolf,et al.  Plasma gelsolin and circulating actin correlate with hemodialysis mortality. , 2009, Journal of the American Society of Nephrology : JASN.

[5]  M. Milazzo,et al.  Protein expression profiles in pediatric multiple sclerosis: potential biomarkers , 2009, Multiple sclerosis.

[6]  P. Janmey,et al.  Plasma gelsolin: function, prognostic value, and potential therapeutic use. , 2008, Current protein & peptide science.

[7]  M. Krumbholz,et al.  Compartmentalization of inflammation in the CNS: A major mechanism driving progressive multiple sclerosis , 2008, Journal of the Neurological Sciences.

[8]  P. Janmey,et al.  Extracellular Gelsolin Binds Lipoteichoic Acid and Modulates Cellular Response to Proinflammatory Bacterial Wall Components1 , 2008, The Journal of Immunology.

[9]  T. Stossel,et al.  Decreased levels of the gelsolin plasma isoform in patients with rheumatoid arthritis , 2008, Arthritis research & therapy.

[10]  P. Janmey,et al.  Gelsolin concentration in cerebrospinal fluid from patients with multiple sclerosis and other neurological disorders , 2008, European journal of neurology.

[11]  I. Tsigelny,et al.  Fibrinogen signal transduction as a mediator and therapeutic target in inflammation: lessons from multiple sclerosis. , 2007, Current medicinal chemistry.

[12]  M. Dinubile.  Plasma gelsolin: in search of its raison d'etre. Focus on "Modifications of cellular responses to lysophosphatidic acid and platelet-activating factor by plasma gelsolin". , 2007, American journal of physiology. Cell physiology.

[13]  J. Hartwig,et al.  Modifications of cellular responses to lysophosphatidic acid and platelet-activating factor by plasma gelsolin. , 2007, American journal of physiology. Cell physiology.

[14]  G. Houen,et al.  Comparison of rocket and crossed immuno‐electrophoresis assays for determination of the level of actin complexing of Gc globulin , 2007, Scandinavian journal of clinical and laboratory investigation.

[15]  F. Lammert,et al.  Gc-globulin: Roles in Response to Injury , 2005 .

[16]  X. Mariette,et al.  Association of rheumatoid arthritis with multiple sclerosis: report of 14 cases and discussion of its significance. , 2006, The Journal of rheumatology.

[17]  T. Stossel,et al.  Relationship of Plasma Gelsolin Levels to Outcomes in Critically Ill Surgical Patients , 2006, Annals of surgery.

[18]  F. Barkhof,et al.  Blood–brain barrier alterations in both focal and diffuse abnormalities on postmortem MRI in multiple sclerosis , 2005, Neurobiology of Disease.

[19]  S. Reingold,et al.  Diagnostic criteria for multiple sclerosis: 2005 revisions to the “McDonald Criteria” , 2005, Annals of neurology.

[20]  P. Janmey,et al.  Inactivation of endotoxin by human plasma gelsolin. , 2005, Biochemistry.

[21]  C. Polman,et al.  Biological markers in CSF and blood for axonal degeneration in multiple sclerosis , 2005, The Lancet Neurology.

[22]  À. Rovira,et al.  New diagnostic criteria for multiple sclerosis: Application in first demyelinating episode , 2003 .

[23]  T. Stossel,et al.  Prognostic implications of declining plasma gelsolin levels after allogeneic stem cell transplantation. , 2002, Blood.

[24]  M. Sharief,et al.  Heightened intrathecal release of axonal cytoskeletal proteins in multiple sclerosis is associated with progressive disease and clinical disability , 2002, Journal of Neuroimmunology.

[25]  C. Poser,et al.  Diagnostic criteria for multiple sclerosis , 2001, Clinical Neurology and Neurosurgery.

[26]  Simon C Watkins,et al.  Actin-containing sera from patients with adult respiratory distress syndrome are toxic to sheep pulmonary endothelial cells. , 2000, American journal of respiratory and critical care medicine.

[27]  K. Mounzer,et al.  Relationship of admission plasma gelsolin levels to clinical outcomes in patients after major trauma. , 1999, American journal of respiratory and critical care medicine.

[28]  W. M. Lee,et al.  Decreased plasma gelsolin concentrations in acute liver failure, myocardial infarction, septic shock, and myonecrosis. , 1997, Critical care medicine.

[29]  J. E. Estes,et al.  Vascular clearance and organ uptake of G- and F-actin in the rat. , 1993, The American journal of physiology.

[30]  R. Bouillon,et al.  Polyunsaturated fatty acids decrease the apparent affinity of vitamin D metabolites for human vitamin D-binding protein , 1992, The Journal of Steroid Biochemistry and Molecular Biology.

[31]  W. Lee,et al.  The extracellular actin-scavenger system and actin toxicity. , 1992, The New England journal of medicine.

[32]  H. Yamamoto,et al.  Depression of plasma gelsolin level during acute liver injury. , 1992, Gastroenterology.

[33]  N. Yamamoto,et al.  Vitamin D3 binding protein (group-specific component) is a precursor for the macrophage-activating signal factor from lysophosphatidylcholine-treated lymphocytes. , 1991, Proceedings of the National Academy of Sciences of the United States of America.

[34]  J. W. Sanger,et al.  Angiopathic consequences of saturating the plasma scavenger system for actin. , 1990, Proceedings of the National Academy of Sciences of the United States of America.

[35]  W. M. Lee,et al.  Alterations in Gc levels and complexing in septic shock. , 1989, Circulatory shock.

[36]  P. Halushka,et al.  Alterations in plasma levels and complexing of Gc (vitamin D-binding protein) in rats with endotoxic shock. , 1989, Circulatory shock.

[37]  D. B. Smith,et al.  Depression of gelsolin levels and detection of gelsolin-actin complexes in plasma of patients with acute lung injury. , 1988, The American review of respiratory disease.

[38]  P. Janmey,et al.  Capacity of human serum to depolymerize actin filaments. , 1987, Blood.

[39]  D. B. Smith,et al.  Role of plasma gelsolin and the vitamin D-binding protein in clearing actin from the circulation. , 1986, The Journal of clinical investigation.

[40]  D. Kwiatkowski,et al.  Structure and biosynthesis of cytoplasmic and secreted variants of gelsolin. , 1984, The Journal of biological chemistry.

[41]  T. Stossel,et al.  Control of cytoplasmic actin gel–sol transformation by gelsolin, a calcium-dependent regulatory protein , 1979, Nature.