Rebound of disease activity after fingolimod withdrawal: Immunological and gene expression profiling.
暂无分享,去创建一个
[1] C. Tornatore,et al. Fingolimod Rebound: A Review of the Clinical Experience and Management Considerations , 2019, Neurology and Therapy.
[2] S. Schmidt,et al. Severe rebound after cessation of fingolimod treated with ocrelizumab with coincidental transient aggravation: report of two cases , 2019, Therapeutic advances in neurological disorders.
[3] V. Meca-Lallana,et al. Tumefactive multiple sclerosis lesions associated with fingolimod treatment: Report of 5 cases. , 2018, Multiple sclerosis and related disorders.
[4] F. Quintana,et al. Regulation of the Immune Response by the Aryl Hydrocarbon Receptor. , 2018, Immunity.
[5] A. Bar-Or,et al. Reconstitution of the peripheral immune repertoire following withdrawal of fingolimod , 2017, Multiple sclerosis.
[6] E. Waubant,et al. Rebound Syndrome in Patients With Multiple Sclerosis After Cessation of Fingolimod Treatment. , 2016, JAMA neurology.
[7] L. Kappos,et al. Long-term effects of fingolimod in multiple sclerosis , 2015, Neurology.
[8] J. Kira,et al. Peripheral Blood T Cell Dynamics Predict Relapse in Multiple Sclerosis Patients on Fingolimod , 2015, PloS one.
[9] Fan Yang,et al. STAT5 programs a distinct subset of GM-CSF-producing T helper cells that is essential for autoimmune neuroinflammation , 2014, Cell Research.
[10] L. Kappos,et al. Th17 central memory T cells are reduced by FTY720 in patients with multiple sclerosis , 2010, Neurology.
[11] V. Brinkmann. FTY720 (fingolimod) in Multiple Sclerosis: therapeutic effects in the immune and the central nervous system , 2009, British journal of pharmacology.
[12] L. Kappos,et al. FTY720 therapy exerts differential effects on T cell subsets in multiple sclerosis , 2008, Neurology.
[13] L. Cosmi,et al. Phenotypic and functional features of human Th17 cells , 2007, The Journal of experimental medicine.
[14] R. Proia,et al. Lymphocyte egress from thymus and peripheral lymphoid organs is dependent on S1P receptor 1 , 2004, Nature.