Hemotherapy bedside biovigilance involving vital sign values and characteristics of patients with suspected transfusion reactions associated with fluid challenges: can some cases of transfusion‐associated circulatory overload have proinflammatory aspects?
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J. Steingrub | P. Visintainer | M. Popovsky | C. Andrzejewski | L. O'Hearn | Theresa Stec | Mark A. Popovsky | Theresa C. Stec | Jean Provencher
[1] Ognjen Gajic,et al. Incidence and transfusion risk factors for transfusion‐associated circulatory overload among medical intensive care unit patients , 2011, Transfusion.
[2] N. Blumberg,et al. An association between decreased cardiopulmonary complications (transfusion‐related acute lung injury and transfusion‐associated circulatory overload) and implementation of universal leukoreduction of blood transfusions , 2010, Transfusion.
[3] N. Blumberg,et al. An association between decreased cardiopulmonary complications (TRALI and TACO) and implementation of universal leukoreduction of blood transfusions , 2010 .
[4] Janet S. Lee,et al. Advanced glycation end products on stored red blood cells increase endothelial reactive oxygen species generation through interaction with receptor for advanced glycation end products , 2010, Transfusion.
[5] S. Glynn,et al. The red blood cell storage lesion: a method to the madness , 2010, Transfusion.
[6] B. Casserly,et al. The role of natriuretic peptides in inflammation and immunity. , 2010, Recent patents on inflammation & allergy drug discovery.
[7] O. Hammarsten,et al. Inflammation increases NT-proBNP and the NT-proBNP/BNP ratio , 2010, Clinical Research in Cardiology.
[8] Liron Pantanowitz,et al. Development of electronic medical record charting for hospital-based transfusion and apheresis medicine services: Early adoption perspectives , 2010, Journal of pathology informatics.
[9] A. Eder,et al. Noninfectious complications of blood transfusion. , 2009, Archives of pathology & laboratory medicine.
[10] J. Vincent,et al. Association between duration of storage of transfused red blood cells and morbidity and mortality in adult patients: myth or reality? , 2009, Transfusion.
[11] J. Struck,et al. Plasma NT-proBNP increases in response to LPS administration in healthy men. , 2008, Journal of applied physiology.
[12] V. Izzi,et al. Brain Natriuretic Peptide (BNP) regulates the production of inflammatory mediators in human THP-1 macrophages , 2008, Regulatory Peptides.
[13] T. Hornemann,et al. In critically ill patients, B-type natriuretic peptide (BNP) and N-terminal pro-BNP levels correlate with C-reactive protein values and leukocyte counts. , 2008, International journal of cardiology.
[14] J. Fildes,et al. Does Brain Natriuretic Peptide Interact With the Immune System After Cardiac Transplantation? , 2007, Transplantation.
[15] R. Skeate,et al. Distinguishing between transfusion related acute lung injury and transfusion associated circulatory overload , 2007, Current opinion in hematology.
[16] A. Koshkaryev,et al. Blood banking–induced alteration of red blood cell flow properties , 2007, Transfusion.
[17] S. Mohapatra. Role of natriuretic peptide signaling in modulating asthma and inflammation. , 2007, Canadian journal of physiology and pharmacology.
[18] T. Gardner. To transfuse or not to transfuse. , 2007, Circulation.
[19] B. Henderson,et al. Stressing the obvious? Cell stress and cell stress proteins in cardiovascular disease. , 2007, Cardiovascular research.
[20] T. Lesnick,et al. Transfusion‐related acute lung injury and pulmonary edema in critically ill patients: a retrospective study , 2006, Transfusion.
[21] O. Gajic,et al. Pulmonary edema after transfusion: How to differentiate transfusion-associated circulatory overload from transfusion-related acute lung injury , 2006, Critical care medicine.
[22] P. Toy,et al. Designing and testing a computer-based screening system for transfusion-related acute lung injury. , 2005, American journal of clinical pathology.
[23] C. Andrzejewski,et al. Transfusion‐associated adverse pulmonary sequelae: widening our perspective , 2005, Transfusion.
[24] A. Vollmar. The role of atrial natriuretic peptide in the immune system , 2005, Peptides.
[25] O. Baskurt,et al. Blood Rheology and Hemodynamics , 2003, Seminars in thrombosis and hemostasis.
[26] A Graham Pockley,et al. Heat Shock Proteins, Inflammation, and Cardiovascular Disease , 2002, Circulation.
[27] Dutcher. Transfusion Therapy: Clinical Principles and Practice. , 2000, Annals of internal medicine.
[28] U. de Faire,et al. Circulating heat shock protein 60 is associated with early cardiovascular disease. , 2000, Hypertension.
[29] M. McMahon. Administration of blood products. , 2000, International journal of trauma nursing.
[30] G. Telegdy,et al. Hyperthermic effect of centrally administered natriuretic peptides in the rat , 1999, Peptides.
[31] M. Popovsky,et al. Transfusion-associated circulatory overload in orthopedic surgery patients: a multi-institutional study , 1996, Immunohematology.
[32] J. Fildes,et al. BNP directly immunoregulates the innate immune system of cardiac transplant recipients in vitro. , 2009, Transplant immunology.
[33] P. Larson. Other Noninfectious Complications of Transfusion , 2001 .
[34] Circulatory overload. , 1961, New York state journal of medicine.