Hemotherapy bedside biovigilance involving vital sign values and characteristics of patients with suspected transfusion reactions associated with fluid challenges: can some cases of transfusion‐associated circulatory overload have proinflammatory aspects?

BACKGROUND: Monitoring of patients' vital sign values (VSVs) during hemotherapy may have an important role in the recognition and mitigation of transfusion‐associated circulatory overload (TACO). Knowledge regarding VSVs and other patient characteristics in bedside‐reported TACO or fluid challenge–suspected transfusion reactions (TACO/FC‐STRs) is limited.

[1]  Ognjen Gajic,et al.  Incidence and transfusion risk factors for transfusion‐associated circulatory overload among medical intensive care unit patients , 2011, Transfusion.

[2]  N. Blumberg,et al.  An association between decreased cardiopulmonary complications (transfusion‐related acute lung injury and transfusion‐associated circulatory overload) and implementation of universal leukoreduction of blood transfusions , 2010, Transfusion.

[3]  N. Blumberg,et al.  An association between decreased cardiopulmonary complications (TRALI and TACO) and implementation of universal leukoreduction of blood transfusions , 2010 .

[4]  Janet S. Lee,et al.  Advanced glycation end products on stored red blood cells increase endothelial reactive oxygen species generation through interaction with receptor for advanced glycation end products , 2010, Transfusion.

[5]  S. Glynn,et al.  The red blood cell storage lesion: a method to the madness , 2010, Transfusion.

[6]  B. Casserly,et al.  The role of natriuretic peptides in inflammation and immunity. , 2010, Recent patents on inflammation & allergy drug discovery.

[7]  O. Hammarsten,et al.  Inflammation increases NT-proBNP and the NT-proBNP/BNP ratio , 2010, Clinical Research in Cardiology.

[8]  Liron Pantanowitz,et al.  Development of electronic medical record charting for hospital-based transfusion and apheresis medicine services: Early adoption perspectives , 2010, Journal of pathology informatics.

[9]  A. Eder,et al.  Noninfectious complications of blood transfusion. , 2009, Archives of pathology & laboratory medicine.

[10]  J. Vincent,et al.  Association between duration of storage of transfused red blood cells and morbidity and mortality in adult patients: myth or reality? , 2009, Transfusion.

[11]  J. Struck,et al.  Plasma NT-proBNP increases in response to LPS administration in healthy men. , 2008, Journal of applied physiology.

[12]  V. Izzi,et al.  Brain Natriuretic Peptide (BNP) regulates the production of inflammatory mediators in human THP-1 macrophages , 2008, Regulatory Peptides.

[13]  T. Hornemann,et al.  In critically ill patients, B-type natriuretic peptide (BNP) and N-terminal pro-BNP levels correlate with C-reactive protein values and leukocyte counts. , 2008, International journal of cardiology.

[14]  J. Fildes,et al.  Does Brain Natriuretic Peptide Interact With the Immune System After Cardiac Transplantation? , 2007, Transplantation.

[15]  R. Skeate,et al.  Distinguishing between transfusion related acute lung injury and transfusion associated circulatory overload , 2007, Current opinion in hematology.

[16]  A. Koshkaryev,et al.  Blood banking–induced alteration of red blood cell flow properties , 2007, Transfusion.

[17]  S. Mohapatra Role of natriuretic peptide signaling in modulating asthma and inflammation. , 2007, Canadian journal of physiology and pharmacology.

[18]  T. Gardner To transfuse or not to transfuse. , 2007, Circulation.

[19]  B. Henderson,et al.  Stressing the obvious? Cell stress and cell stress proteins in cardiovascular disease. , 2007, Cardiovascular research.

[20]  T. Lesnick,et al.  Transfusion‐related acute lung injury and pulmonary edema in critically ill patients: a retrospective study , 2006, Transfusion.

[21]  O. Gajic,et al.  Pulmonary edema after transfusion: How to differentiate transfusion-associated circulatory overload from transfusion-related acute lung injury , 2006, Critical care medicine.

[22]  P. Toy,et al.  Designing and testing a computer-based screening system for transfusion-related acute lung injury. , 2005, American journal of clinical pathology.

[23]  C. Andrzejewski,et al.  Transfusion‐associated adverse pulmonary sequelae: widening our perspective , 2005, Transfusion.

[24]  A. Vollmar The role of atrial natriuretic peptide in the immune system , 2005, Peptides.

[25]  O. Baskurt,et al.  Blood Rheology and Hemodynamics , 2003, Seminars in thrombosis and hemostasis.

[26]  A Graham Pockley,et al.  Heat Shock Proteins, Inflammation, and Cardiovascular Disease , 2002, Circulation.

[27]  Dutcher Transfusion Therapy: Clinical Principles and Practice. , 2000, Annals of internal medicine.

[28]  U. de Faire,et al.  Circulating heat shock protein 60 is associated with early cardiovascular disease. , 2000, Hypertension.

[29]  M. McMahon Administration of blood products. , 2000, International journal of trauma nursing.

[30]  G. Telegdy,et al.  Hyperthermic effect of centrally administered natriuretic peptides in the rat , 1999, Peptides.

[31]  M. Popovsky,et al.  Transfusion-associated circulatory overload in orthopedic surgery patients: a multi-institutional study , 1996, Immunohematology.

[32]  J. Fildes,et al.  BNP directly immunoregulates the innate immune system of cardiac transplant recipients in vitro. , 2009, Transplant immunology.

[33]  P. Larson Other Noninfectious Complications of Transfusion , 2001 .

[34]  Circulatory overload. , 1961, New York state journal of medicine.