Loss of intestinal crypt progenitor cells owing to inactivation of both Notch1 and Notch2 is accompanied by derepression of CDK inhibitors p27Kip1 and p57Kip2
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Hans Clevers | Freddy Radtke | Tasuku Honjo | H. Clevers | T. Honjo | J. van Es | M. van Gijn | L. Strobl | U. Zimber-Strobl | F. Radtke | Orbicia Riccio | Marielle E van Gijn | April C Bezdek | Luca Pellegrinet | Johan H van Es | Ursula Zimber‐Strobl | Lothar J Strobl | L. Pellegrinet | O. Riccio | A. Bezdek
[1] H. Hirata,et al. Hes1 Directly Controls Cell Proliferation through the Transcriptional Repression of p27Kip1 , 2005, Molecular and Cellular Biology.
[2] H. Macdonald,et al. Deficient T cell fate specification in mice with an induced inactivation of Notch1. , 1999, Immunity.
[3] A. Gossler,et al. Expression of Notch pathway components in fetal and adult mouse small intestine. , 2002, Gene expression patterns : GEP.
[4] Richard A. Flavell,et al. Nod2-Dependent Regulation of Innate and Adaptive Immunity in the Intestinal Tract , 2005, Science.
[5] S. Artavanis-Tsakonas,et al. Notch signaling: cell fate control and signal integration in development. , 1999, Science.
[6] Ryoichiro Kageyama,et al. Control of endodermal endocrine development by Hes-1 , 2000, Nature Genetics.
[7] Hans Clevers,et al. Notch1 functions as a tumor suppressor in mouse skin , 2003, Nature Genetics.
[8] Hans Clevers,et al. Self-Renewal and Cancer of the Gut: Two Sides of a Coin , 2005, Science.
[9] Hans Clevers,et al. Notch/γ-secretase inhibition turns proliferative cells in intestinal crypts and adenomas into goblet cells , 2005, Nature.
[10] M. Arcangeli,et al. Hierarchy of Notch–Delta interactions promoting T cell lineage commitment and maturation , 2007, The Journal of experimental medicine.
[11] Daniel Metzger,et al. Tissue‐specific and inducible Cre‐mediated recombination in the gut epithelium , 2004, Genesis.
[12] J. Aster. Deregulated NOTCH Signaling in Acute T-Cell Lymphoblastic Leukemia/Lymphoma: New Insights, Questions, and Opportunities , 2005, International journal of hematology.
[13] Jay S. Fine,et al. Chronic Treatment with the γ-Secretase Inhibitor LY-411,575 Inhibits β-Amyloid Peptide Production and Alters Lymphopoiesis and Intestinal Cell Differentiation* , 2004, Journal of Biological Chemistry.
[14] Hans Clevers,et al. The β-Catenin/TCF-4 Complex Imposes a Crypt Progenitor Phenotype on Colorectal Cancer Cells , 2002, Cell.
[15] Neil Vargesson,et al. Delta-Notch signalling controls commitment to a secretory fate in the zebrafish intestine , 2005, Development.
[16] H. Zoghbi,et al. Requirement of Math1 for Secretory Cell Lineage Commitment in the Mouse Intestine , 2001, Science.
[17] L. Hengst,et al. Translational Control of p27Kip1 Accumulation During the Cell Cycle , 1996, Science.
[18] Francois Pognan,et al. Modulation of notch processing by gamma-secretase inhibitors causes intestinal goblet cell metaplasia and induction of genes known to specify gut secretory lineage differentiation. , 2004, Toxicological sciences : an official journal of the Society of Toxicology.
[19] B. Powell,et al. Expression of Notch Receptors and Ligands in the Adult Gut , 2004, The journal of histochemistry and cytochemistry : official journal of the Histochemistry Society.
[20] W. Gordon,et al. Notch1 modulates timing of G1-S progression by inducing SKP2 transcription and p27Kip1 degradation , 2005, The Journal of experimental medicine.
[21] Pumin Zhang,et al. p57 and Hes1 coordinate cell cycle exit with self-renewal of pancreatic progenitors. , 2006, Developmental biology.
[22] S. Artavanis-Tsakonas,et al. Notch signals control the fate of immature progenitor cells in the intestine , 2005, Nature.
[23] Ryoichiro Kageyama,et al. Roles of bHLH genes in neural stem cell differentiation. , 2005, Experimental cell research.