Effectiveness of community-based folate-oriented tertiary interventions on incidence of fetus and birth defects: a protocol for a single-blind cluster randomized controlled trial

[1]  W. Holzgreve,et al.  Folate supplementation for prevention of congenital heart defects and low birth weight: an update. , 2019, Cardiovascular diagnosis and therapy.

[2]  Hongyan Wang,et al.  Lower Circulating Folate Induced by a Fidgetin Intronic Variant Is Associated With Reduced Congenital Heart Disease Susceptibility , 2017, Circulation.

[3]  M. Phipps,et al.  Folic Acid Supplementation for the Prevention of Neural Tube Defects: US Preventive Services Task Force Recommendation Statement , 2017, JAMA.

[4]  Shiliang Liu,et al.  Effect of Folic Acid Food Fortification in Canada on Congenital Heart Disease Subtypes , 2016, Circulation.

[5]  Krista S Crider,et al.  Optimal Serum and Red Blood Cell Folate Concentrations in Women of Reproductive Age for Prevention of Neural Tube Defects: World Health Organization Guidelines , 2015, MMWR. Morbidity and mortality weekly report.

[6]  M. Brosnan,et al.  Nuclear Enrichment of Folate Cofactors and Methylenetetrahydrofolate Dehydrogenase 1 (MTHFD1) Protect de Novo Thymidylate Biosynthesis during Folate Deficiency* , 2014, The Journal of Biological Chemistry.

[7]  Q. Peng,et al.  Genetic variants reducing MTR gene expression increase the risk of congenital heart disease in Han Chinese populations. , 2014, European heart journal.

[8]  A. Czeizel Is folic acid a risk factor for oral clefts? , 2013, European Journal of Epidemiology.

[9]  G. Lathrop,et al.  Association Between C677T Polymorphism of Methylene Tetrahydrofolate Reductase and Congenital Heart Disease: Meta-Analysis of 7697 Cases and 13 125 Controls , 2013, Circulation. Cardiovascular genetics.

[10]  M. Drummond,et al.  Consolidated Health Economic Evaluation Reporting Standards (CHEERS) statement. , 2013, BMJ.

[11]  Per Magnus,et al.  Association between maternal use of folic acid supplements and risk of autism spectrum disorders in children. , 2013, JAMA.

[12]  B. Qiao,et al.  A functional variant in the cystathionine β-synthase gene promoter significantly reduces congenital heart disease susceptibility in a Han Chinese population , 2012, Cell Research.

[13]  M. Drummond,et al.  Consolidated Health Economic Evaluation Reporting Standards (CHEERS) statement. , 2013, Clinical therapeutics.

[14]  C. Moore,et al.  Folic Acid Use and Nonsyndromic Orofacial Clefts in China: A Prospective Cohort Study , 2012, Epidemiology.

[15]  B. Qiao,et al.  Functional Variant in Methionine Synthase Reductase Intron-1 Significantly Increases the Risk of Congenital Heart Disease in the Han Chinese Population , 2012, Circulation.

[16]  P. R. Mondal,et al.  Spectrum of MTHFR gene SNPs C677T and A1298C: a study among 23 population groups of India , 2011, Molecular Biology Reports.

[17]  J. Ordovás,et al.  Methylenetetrahydrofolate reductase variants associated with hypertension and cardiovascular disease interact with dietary polyunsaturated fatty acids to modulate plasma homocysteine in puerto rican adults. , 2011, The Journal of nutrition.

[18]  J. Murray,et al.  Folic acid and orofacial clefts: A review of the evidence , 2010, Oral diseases.

[19]  S. Cousens,et al.  Folic acid to reduce neonatal mortality from neural tube disorders , 2010, International journal of epidemiology.

[20]  Livia Kapusta,et al.  Protective effect of periconceptional folic acid supplements on the risk of congenital heart defects: a registry-based case-control study in the northern Netherlands. , 2010, European heart journal.

[21]  L. Noel Human organ and tissue transplantation. Resolution WHA 63.22 of the Sixty-Third World Health Assembly. , 2010, Cell and tissue banking.

[22]  S. W. H. Assembly Human organ and tissue transplantation , 2010, Cell and Tissue Banking.

[23]  Yao Ming-zhu Analysis on the composition of 481 neonates with congenital malformation and their associated factors , 2008 .

[24]  H. Hata,et al.  Additional food folate derived exclusively from natural sources improves folate status in young women with the MTHFR 677 CC or TT genotype , 2006, The Journal of nutritional biochemistry.

[25]  Hilde van der Togt,et al.  Publisher's Note , 2003, J. Netw. Comput. Appl..

[26]  S. Cnattingius,et al.  Plasma folate levels and risk of spontaneous abortion. , 2002, JAMA.

[27]  H. Blom,et al.  Is mutated serine hydroxymethyltransferase (SHMT) involved in the etiology of neural tube defects? , 2001, Molecular genetics and metabolism.

[28]  A. Chango,et al.  A polymorphism (80G->A) in the reduced folate carrier gene and its associations with folate status and homocysteinemia. , 2000, Molecular genetics and metabolism.

[29]  Z. Li,et al.  Prevention of neural-tube defects with folic acid in China. China-U.S. Collaborative Project for Neural Tube Defect Prevention. , 2000, The New England journal of medicine.

[30]  Hong Wang,et al.  Correction: Prevention of Neural-Tube Defects with Folic Acid in China. , 1999, The New England journal of medicine.

[31]  A. Czeizel,et al.  Prevention of the first occurrence of neural-tube defects by periconceptional vitamin supplementation. , 1992, The New England journal of medicine.