HIV-1 reservoir evolution in infants infected with clade C from Mozambique

[1]  D. Kuritzkes,et al.  Immune correlates of HIV-1 reservoir cell decline in early-treated infants , 2022, Cell reports.

[2]  E. Rosenberg,et al.  Parallel analysis of transcription, integration, and sequence of single HIV-1 proviruses , 2022, Cell.

[3]  R. Siliciano,et al.  A unique viral reservoir landscape in HIV-1 elite controllers , 2020, Nature.

[4]  J. Siliciano,et al.  Selective Decay of Intact HIV-1 Proviral DNA on Antiretroviral Therapy. , 2020, The Journal of infectious diseases.

[5]  F. Bushman,et al.  Single-cell transcriptional landscapes reveal HIV-1–driven aberrant host gene transcription as a potential therapeutic target , 2020, Science Translational Medicine.

[6]  R. Siliciano,et al.  Differential decay of intact and defective proviral DNA in HIV-1-infected individuals on suppressive antiretroviral therapy. , 2020, JCI insight.

[7]  Sudhir Kumar,et al.  Molecular Evolutionary Genetics Analysis (MEGA) for macOS. , 2020, Molecular biology and evolution.

[8]  Wei-Shau Hu,et al.  Intact HIV Proviruses Persist in Children Seven to Nine Years after Initiation of Antiretroviral Therapy in the First Year of Life , 2019, Journal of Virology.

[9]  D. Kuritzkes,et al.  Early antiretroviral therapy in neonates with HIV-1 infection restricts viral reservoir size and induces a distinct innate immune profile , 2019, Science Translational Medicine.

[10]  P. Lemey,et al.  HIV Rebound Is Predominantly Fueled by Genetically Identical Viral Expansions from Diverse Reservoirs. , 2019, Cell host & microbe.

[11]  E. Rosenberg,et al.  Intact HIV-1 proviruses accumulate at distinct chromosomal positions during prolonged antiretroviral therapy , 2019, The Journal of clinical investigation.

[12]  B. Walker,et al.  HIV-1 DNA sequence diversity and evolution during acute subtype C infection , 2019, Nature Communications.

[13]  Sudhir Kumar,et al.  MEGA X: Molecular Evolutionary Genetics Analysis across Computing Platforms. , 2018, Molecular biology and evolution.

[14]  L. Kuhn,et al.  Age at antiretroviral therapy initiation and cell-associated HIV-1 DNA levels in HIV-1-infected children , 2018, PloS one.

[15]  E. Boritz,et al.  Identification of Genetically Intact HIV-1 Proviruses in Specific CD4+ T Cells from Effectively Treated Participants. , 2017, Cell reports.

[16]  E. Rosenberg,et al.  Clonal expansion of genome-intact HIV-1 in functionally polarized Th1 CD4+ T cells , 2017, The Journal of clinical investigation.

[17]  D. Persaud,et al.  Paucity of Intact Non-Induced Provirus with Early, Long-Term Antiretroviral Therapy of Perinatal HIV Infection , 2017, PloS one.

[18]  Hyunseok Kang,et al.  Whole-Exome Sequencing of Salivary Gland Mucoepidermoid Carcinoma , 2016, Clinical Cancer Research.

[19]  H. Kędzierska,et al.  Expression of Genes Involved in Cellular Adhesion and Extracellular Matrix Remodeling Correlates with Poor Survival of Patients with Renal Cancer. , 2016, The Journal of urology.

[20]  S. Hughes,et al.  Clonally expanded CD4+ T cells can produce infectious HIV-1 in vivo , 2016, Proceedings of the National Academy of Sciences.

[21]  J. Mellors,et al.  Early Antiretroviral Therapy in South African Children Reduces HIV-1-Infected Cells and Cell-Associated HIV-1 RNA in Blood Mononuclear Cells. , 2015, The Journal of infectious diseases.

[22]  Michael Q. Zhang,et al.  Integrative analysis of 111 reference human epigenomes , 2015, Nature.

[23]  D. Richman,et al.  HIV type 1 (HIV-1) proviral reservoirs decay continuously under sustained virologic control in HIV-1-infected children who received early treatment. , 2014, The Journal of infectious diseases.

[24]  Brendan B. Larsen,et al.  Proliferation of cells with HIV integrated into cancer genes contributes to persistent infection , 2014, Science.

[25]  P. Lemey,et al.  The HIV-1 reservoir in eight patients on long-term suppressive antiretroviral therapy is stable with few genetic changes over time , 2013, Proceedings of the National Academy of Sciences.

[26]  A. Levine,et al.  The diversity of zinc-finger genes on human chromosome 19 provides an evolutionary mechanism for defense against inherited endogenous retroviruses , 2013, Cell Death and Differentiation.

[27]  Sarah B. Laskey,et al.  Replication-Competent Noninduced Proviruses in the Latent Reservoir Increase Barrier to HIV-1 Cure , 2013, Cell.

[28]  Thomas Lengauer,et al.  Bioinformatics prediction of HIV coreceptor usage , 2007, Nature Biotechnology.

[29]  Tommy F. Liu,et al.  Web resources for HIV type 1 genotypic-resistance test interpretation. , 2006, Clinical infectious diseases : an official publication of the Infectious Diseases Society of America.

[30]  Tom H. Pringle,et al.  The human genome browser at UCSC. , 2002, Genome research.

[31]  D. Margolis,et al.  Longitudinal Dynamics of Intact HIV Proviral DNA and Outgrowth Virus Frequencies in a Cohort of Individuals Receiving Antiretroviral Therapy , 2021 .