Hyperglycemia is associated with enhanced gluconeogenesis in a rat model of permanent cerebral ischemia

[1]  L. Rui,et al.  Hepatic TRAF2 Regulates Glucose Metabolism Through Enhancing Glucagon Responses , 2012, Diabetes.

[2]  E. Feldman,et al.  Cortical neurons develop insulin resistance and blunted Akt signaling: a potential mechanism contributing to enhanced ischemic injury in diabetes. , 2011, Antioxidants & redox signaling.

[3]  M. Montminy,et al.  CREB and the CRTC co-activators: sensors for hormonal and metabolic signals , 2011, Nature Reviews Molecular Cell Biology.

[4]  R. Douglas,et al.  Insulin/PI3K signaling protects dentate neurons from oxygen–glucose deprivation in organotypic slice cultures , 2010, Journal of neurochemistry.

[5]  S. Tokuyama,et al.  The development of glucose intolerance after focal cerebral ischemia participates in subsequent neuronal damage , 2009, Brain Research.

[6]  V. Bhardwaj,et al.  Tumour necrosis factor‐α attenuates insulin action on phosphoenolpyruvate carboxykinase gene expression and gluconeogenesis by altering the cellular localization of Foxa2 in HepG2 cells , 2009, The FEBS journal.

[7]  F. Wondisford,et al.  Metformin and Insulin Suppress Hepatic Gluconeogenesis through Phosphorylation of CREB Binding Protein , 2009, Cell.

[8]  M. Walters,et al.  Management of Hyperglycemia in Acute Stroke: How, When, and for Whom? , 2008, Stroke.

[9]  M. Lazar,et al.  Adipokines and the peripheral and neural control of energy balance. , 2008, Molecular endocrinology.

[10]  G. Hotamisligil,et al.  Inflammation and metabolic disorders , 2006, Nature.

[11]  R. Sapolsky,et al.  Phosphoinositide-3-kinase/Akt survival signal pathways are implicated in neuronal survival after stroke , 2006, Molecular Neurobiology.

[12]  Miet Schetz,et al.  Intensive Insulin Therapy in Mixed Medical/Surgical Intensive Care Units , 2006, Diabetes.

[13]  O. MacDougald,et al.  CCAAT/Enhancer-binding Protein α Mediates Induction of Hepatic Phosphoenolpyruvate Carboxykinase by p38 Mitogen-activated Protein Kinase* , 2006, Journal of Biological Chemistry.

[14]  L. McCullough,et al.  Pharmacological Inhibition of AMP-activated Protein Kinase Provides Neuroprotection in Stroke* , 2005, Journal of Biological Chemistry.

[15]  M. Laakso,et al.  Intense metabolic control by means of insulin in patients with diabetes mellitus and acute myocardial infarction (DIGAMI 2): effects on mortality and morbidity , 2005 .

[16]  D. Porte,et al.  Diabetes, Obesity, and the Brain , 2005, Science.

[17]  W. Lamers,et al.  Mechanisms of glucocorticoid signalling. , 2004, Biochimica et biophysica acta.

[18]  R. Auer,et al.  Optimal blood glucose levels while using insulin to minimize the size of infarction in focal cerebral ischemia. , 2004, Journal of neurosurgery.

[19]  M. Birnbaum,et al.  AMP-kinase regulates food intake by responding to hormonal and nutrient signals in the hypothalamus , 2004, Nature.

[20]  Bruce M. Spiegelman,et al.  Insulin-regulated hepatic gluconeogenesis through FOXO1–PGC-1α interaction , 2003, Nature.

[21]  G. Grunkemeier,et al.  Continuous insulin infusion reduces mortality in patients with diabetes undergoing coronary artery bypass grafting. , 2003, The Journal of thoracic and cardiovascular surgery.

[22]  R. Seeley,et al.  Insulin activation of phosphatidylinositol 3-kinase in the hypothalamic arcuate nucleus: a key mediator of insulin-induced anorexia. , 2003, Diabetes.

[23]  Michael Karin,et al.  A central role for JNK in obesity and insulin resistance , 2002, Nature.

[24]  S. Uchida,et al.  Adiponectin stimulates glucose utilization and fatty-acid oxidation by activating AMP-activated protein kinase , 2002, Nature Medicine.

[25]  R. Grimble Inflammatory status and insulin resistance , 2002, Current opinion in clinical nutrition and metabolic care.

[26]  Zhaohui Feng,et al.  Decreasing hypothalamic insulin receptors causes hyperphagia and insulin resistance in rats , 2002, Nature Neuroscience.

[27]  M. White,et al.  Phosphorylation of Ser307 in Insulin Receptor Substrate-1 Blocks Interactions with the Insulin Receptor and Inhibits Insulin Action* , 2002, The Journal of Biological Chemistry.

[28]  Jiandie D. Lin,et al.  Cytokine stimulation of energy expenditure through p38 MAP kinase activation of PPARgamma coactivator-1. , 2001, Molecular cell.

[29]  H. Gerstein,et al.  Stress Hyperglycemia and Prognosis of Stroke in Nondiabetic and Diabetic Patients: A Systematic Overview , 2001, Stroke.

[30]  T. Kent,et al.  Heterogeneity Affecting Outcome From Acute Stroke Therapy: Making Reperfusion Worse , 2001, Stroke.

[31]  Peter F. Johnson,et al.  Insulin Suppresses Transactivation by CAAT/Enhancer-binding Proteins β (C/EBPβ) , 2001, The Journal of Biological Chemistry.

[32]  Rüdiger Klein,et al.  Role of Brain Insulin Receptor in Control of Body Weight and Reproduction , 2000 .

[33]  M. Greenberg,et al.  Akt Promotes Cell Survival by Phosphorylating and Inhibiting a Forkhead Transcription Factor , 1999, Cell.

[34]  S. Jitrapakdee Transcription factors and coactivators controlling nutrient and hormonal regulation of hepatic gluconeogenesis. , 2012, The international journal of biochemistry & cell biology.

[35]  Wakako Fujita-Hamabe,et al.  RETRACTED: Effect of Orexin-A on Post-ischemic Glucose Intolerance and Neuronal Damage. , 2011, Journal of pharmacological sciences.

[36]  Shih-Yi Lin,et al.  Adipose proinflammatory cytokine expression through sympathetic system is associated with hyperglycemia and insulin resistance in a rat ischemic stroke model. , 2011, American journal of physiology. Endocrinology and metabolism.