Prediction of Cervical Cancer Based on the Life Style, Habits, and Diseases Using Regression Analysis Framework

Cervical cancer is the most common disease in the woman nowadays. Even though its panic diseases, we can control and prevent it by finding the symptoms of growing cancer. It is the disease formed in the genital area of the woman and later it spreads to all the parts of the body and makes the system collapse to stop functioning of the organs. Condylomatosis, wrong sexual practices and hormonal contraceptives are one of the major primary factors for getting the cervical cancer very easily via Human Papilloma Virus. The secondary factors for causing the cervical cancer is smoking and alcoholic usage. Along with these factors molluscum contagiosum, HIV and Hepatitis B also make the humans to get affected by the cervical cancer very easily. All these factors are to be considered for analysing the patient whether they got affected by the cervical cancer. Regression Analysis model framework is used for comparing the various factors to determine the diseases vastly.

[1]  C. Mitchell,et al.  Pelvic inflammatory disease: current concepts in pathogenesis, diagnosis and treatment. , 2013, Infectious disease clinics of North America.

[2]  W. Weichert,et al.  Contribution of human papilloma virus to the incidence of squamous cell carcinoma of the head and neck in a European population with high smoking prevalence. , 2015, European journal of cancer.

[3]  J. Dillner,et al.  Efficacy of HPV DNA testing with cytology triage and/or repeat HPV DNA testing in primary cervical cancer screening. , 2009, Journal of the National Cancer Institute.

[4]  M. Schiffman,et al.  Relationship Between Cigarette Smoking and Human Papilloma Virus Types 16 and 18 DNA Load , 2009, Cancer Epidemiology, Biomarkers & Prevention.

[5]  Maj Liv Eide,et al.  HPV detection methods and genotyping techniques in screening for cervical cancer. , 2012, Annales de pathologie.

[6]  P. Cattani,et al.  Oral condyloma lesions in patients with extensive genital human papillomavirus infection. , 1992, American journal of obstetrics and gynecology.

[7]  B. Yang,et al.  The corrosion behaviour of copper in simulated uterine fluid , 1996 .

[8]  Francisco A. R. Garcia,et al.  A Cross-sectional Study of a Prototype Carcinogenic Human Papillomavirus E6/E7 Messenger RNA Assay for Detection of Cervical Precancer and Cancer , 2007, Clinical Cancer Research.

[9]  J. Ross.  Pelvic inflammatory disease. , 2010, Clinical evidence.

[10]  S. de Sanjosé,et al.  Squamous intraepithelial lesions of the anal squamocolumnar junction: Histopathological classification and HPV genotyping , 2016, Papillomavirus research.

[11]  O. Laccourreye,et al.  Sinonasal inverted papilloma: From diagnosis to treatment. , 2016, European annals of otorhinolaryngology, head and neck diseases.

[12]  M. Gómez-Roig,et al.  Relationship between smoking, HPV infection, and risk of Cervical cancer. , 2015, European journal of gynaecological oncology.

[13]  Dongsup Lee,et al.  Diagnostic Performance of HPV E6/E7 mRNA and HPV DNA Assays for the Detection and Screening of Oncogenic Human Papillomavirus Infection among Woman with Cervical Lesions in China. , 2015, Asian Pacific journal of cancer prevention : APJCP.

[14]  E. Argyri,et al.  Investigating the role of human papillomavirus in lung cancer , 2016, Papillomavirus research.

[15]  M. Tommasino,et al.  Time to change perspectives on HPV in oropharyngeal cancer. A systematic review of HPV prevalence per oropharyngeal sub-site the last 3 years , 2017, Papillomavirus research.

[16]  G. Zimet,et al.  The feminization of HPV: How science, politics, economics and gender norms shaped U.S. HPV vaccine implementation , 2017, Papillomavirus research.

[17]  S. Garland,et al.  Vaccine-preventable anal human papillomavirus in Australian gay and bisexual men , 2017, Papillomavirus research.

[18]  H. Hagström.  Alcohol, smoking and the liver disease patient. , 2017, Best practice & research. Clinical gastroenterology.

[19]  C. Hwang,et al.  Human papillomavirus infection in oral papillary and verrucous lesions is a prognostic indicator of malignant transformation. , 2012, Cancer epidemiology.

[20]  Sumitha Nayak,et al.  Human papilloma virus and its relation to cervical cancer prevention strategies , 2015 .

[21]  Jay L. Devore,et al.  Probability and statistics for engineering and the sciences , 1982 .

[22]  M. Tommasino,et al.  Prevalence of cutaneous beta and gamma human papillomaviruses in the anal canal of men who have sex with women , 2017, Papillomavirus research.

[23]  I. Sivin.  Utility and drawbacks of continuous use of a copper T IUD for 20 years. , 2007, Contraception.

[24]  R. Brunham,et al.  Pelvic inflammatory disease. , 2006, The New England journal of medicine.

[25]  Justine P. Wu,et al.  Extended use of the intrauterine device: a literature review and recommendations for clinical practice. , 2014, Contraception.

[26]  D. Grimes,et al.  Intrauterine devices and pelvic inflammatory disease: recent developments. , 1987, Contraception.

[27]  L. Garrison,et al.  Acceptability of cervical cancer screening using visual inspection among women attending a childhood immunization clinic in Uganda , 2017, Papillomavirus research.