Respiratory virus-induced EGFR activation suppresses IRF1-dependent interferon λ and antiviral defense in airway epithelium.
暂无分享,去创建一个
L. Lanier | G. Min-oo | J. Nadel | I. Ueki | A. Kalinowski | J. Koff | Eric Ballon-Landa
[1] L. Touqui,et al. Influenza A induces the major secreted airway mucin MUC5AC in a protease-EGFR-extracellular regulated kinase-Sp1-dependent pathway. , 2012, American journal of respiratory cell and molecular biology.
[2] G. Gallagher,et al. Regulation of IFN-λ1 Promoter Activity (IFN-λ1/IL-29) in Human Airway Epithelial Cells , 2011, The Journal of Immunology.
[3] Naveen Kumar,et al. Receptor Tyrosine Kinase Inhibitors That Block Replication of Influenza A and Other Viruses , 2011, Antimicrobial Agents and Chemotherapy.
[4] Adolfo García-Sastre,et al. Dissection of the Influenza A Virus Endocytic Routes Reveals Macropinocytosis as an Alternative Entry Pathway , 2011, PLoS pathogens.
[5] Yuying Liang,et al. Receptor Tyrosine Kinase Inhibitors Block Multiple Steps of Influenza A Virus Replication , 2011, Journal of Virology.
[6] J. Shively,et al. Regulation of CEACAM1 transcription in human breast epithelial cells , 2010, BMC Molecular Biology.
[7] D. Proud,et al. Human rhinovirus-induced epithelial production of CXCL10 is dependent upon IFN regulatory factor-1. , 2010, American journal of respiratory cell and molecular biology.
[8] R. Fouchier,et al. The Epidermal Growth Factor Receptor (EGFR) Promotes Uptake of Influenza A Viruses (IAV) into Host Cells , 2010, PLoS pathogens.
[9] S. Smirnov,et al. Lambda Interferon Is the Predominant Interferon Induced by Influenza A Virus Infection In Vivo , 2010, Journal of Virology.
[10] S. Johnston,et al. Rhinovirus induces MUC5AC in a human infection model and in vitro via NF-&kgr;B and EGFR pathways , 2010, European Respiratory Journal.
[11] S. Günther,et al. Lambda Interferon Renders Epithelial Cells of the Respiratory and Gastrointestinal Tracts Resistant to Viral Infections , 2010, Journal of Virology.
[12] David J. Miller,et al. Role of Double-Stranded RNA Pattern Recognition Receptors in Rhinovirus-Induced Airway Epithelial Cell Responses1 , 2009, The Journal of Immunology.
[13] J. Alcorn,et al. Critical Role of IL-17RA in Immunopathology of Influenza Infection , 2009, The Journal of Immunology.
[14] Wai-ming Lee,et al. Rhinovirus-induced Major Airway Mucin Production Involves a Novel Tlr3-egfr–dependent Pathway , 2009 .
[15] S. Johnston,et al. Respiratory virus induction of alpha‐, beta‐ and lambda‐interferons in bronchial epithelial cells and peripheral blood mononuclear cells , 2009, Allergy.
[16] K. Hartshorn,et al. Differentiated Human Alveolar Type II Cells Secrete Antiviral IL-29 (IFN-λ1) in Response to Influenza A Infection1 , 2009, The Journal of Immunology.
[17] P. Burgel,et al. Epidermal growth factor receptor-mediated innate immune responses and their roles in airway diseases , 2008, European Respiratory Journal.
[18] J. Nadel,et al. Multiple TLRs activate EGFR via a signaling cascade to produce innate immune responses in airway epithelium. , 2008, American journal of physiology. Lung cellular and molecular physiology.
[19] M. Hibbs,et al. Epidermal Growth Factor Receptor Signaling to Erk1/2 and STATs Control the Intensity of the Epithelial Inflammatory Responses to Rhinovirus Infection* , 2008, Journal of Biological Chemistry.
[20] T. Taniguchi,et al. The IRF family transcription factors in immunity and oncogenesis. , 2008, Annual review of immunology.
[21] Eun Joo Lee,et al. Potential use of an anticancer drug gefinitib, an EGFR inhibitor, on allergic airway inflammation , 2007, Experimental & Molecular Medicine.
[22] Stephen T Holgate,et al. Role of deficient type III interferon-λ production in asthma exacerbations , 2006, Nature Medicine.
[23] G. Hunninghake,et al. Activation of the Epidermal Growth Factor Receptor by Respiratory Syncytial Virus Results in Increased Inflammation and Delayed Apoptosis* , 2005, Journal of Biological Chemistry.
[24] J. Nadel,et al. Dual oxidase 1-dependent MUC5AC mucin expression in cultured human airway epithelial cells. , 2005, Proceedings of the National Academy of Sciences of the United States of America.
[25] J. Nadel,et al. Tumor necrosis factor α-converting enzyme mediates MUC5AC mucin expression in cultured human airway epithelial cells , 2003, Proceedings of the National Academy of Sciences of the United States of America.
[26] D. Proud,et al. Effects of tumor necrosis factor-α, epidermal growth factor and transforming growth factor-α on interleukin-8 production by, and human rhinovirus replication in, bronchial epithelial cells , 2001 .
[27] D. Proud,et al. Infection of a human respiratory epithelial cell line with rhinovirus. Induction of cytokine release and modulation of susceptibility to infection by cytokine exposure. , 1995, The Journal of clinical investigation.
[28] A. Choi,et al. Influenza virus A infection induces interleukin‐8 gene expression in human airway epitheial cells , 1992 .
[29] H. Ellerbrok,et al. Inhibition of poxvirus spreading by the anti-tumor drug Gefitinib (Iressa). , 2011, Antiviral research.
[30] S. Johnston,et al. Role of deficient type III interferon-lambda production in asthma exacerbations. , 2006, Nature medicine.
[31] S. Johnston. Overview of virus-induced airway disease. , 2005, Proceedings of the American Thoracic Society.
[32] D. Proud,et al. Effects of tumor necrosis factor-alpha, epidermal growth factor and transforming growth factor-alpha on interleukin-8 production by, and human rhinovirus replication in, bronchial epithelial cells. , 2001, International immunopharmacology.
[33] A. Choi,et al. Influenza virus A infection induces interleukin-8 gene expression in human airway epithelial cells. , 1992, FEBS letters.