Copper, iron, and zinc imbalances in severely degenerated brain regions in Alzheimer's disease: possible relation to oxidative stress
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[1] J. Nobrega,et al. Brain Cytochrome Oxidase in Alzheimer's Disease , 1992, Journal of neurochemistry.
[2] M. Borsaru,et al. Neutron activation analysis , 1972 .
[3] R. Katzman.,et al. Editorial: The prevalence and malignancy of Alzheimer disease. A major killer. , 1976, Archives of neurology.
[4] Maitland Ct. Hospitals in the United States. , 1948, Lancet.
[5] R. Bucala,et al. Advanced glycation end products contribute to amyloidosis in Alzheimer disease. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[6] R. Mohs,et al. Consortium to establish a registry for Alzheimer's disease (CERAD) clinical and neuropsychological assessment of Alzheimer's disease. , 2002, Psychopharmacology bulletin.
[7] W. D. Ehmann,et al. 182 BRAIN TRACE ELEMENT LEVELS IN ALZHEIMERʼS DISEASE BY INSTRUMENTAL NEUTRON ACTIVATION ANALYSIS , 1981 .
[8] M. Folstein,et al. Clinical diagnosis of Alzheimer's disease , 1984, Neurology.
[9] W. D. Ehmann,et al. RNAA for arsenic, cadmium, copper, and molybdenum in CNS tissues from subjects with age-related neurodegenerative diseases , 1994 .
[10] W. D. Ehmann,et al. Determination of trace elements in biological standard kale by neutron activation analysis , 1969 .
[11] S. Landsberger,et al. Design of a GeNaI(Tl) Compton suppression spectrometer and its use in neutron activation analysis , 1990 .
[12] C. W. Scott,et al. Glycated tau protein in Alzheimer disease: a mechanism for induction of oxidant stress. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[13] W. D. Ehmann,et al. Elevated thiobarbituric acid-reactive substances and antioxidant enzyme activity in the brain in Alzheimer's disease , 1995, Neurology.
[14] C. Masters,et al. Rapid induction of Alzheimer A beta amyloid formation by zinc. , 1994, Science.
[15] J. Coyle,et al. Oxidative stress, glutamate, and neurodegenerative disorders. , 1993, Science.
[16] B. Weiss,et al. Food additives as a source of behavioral disturbances in children. , 1986, Neurotoxicology.
[17] W. D. Ehmann,et al. Elemental imbalances in the olfactory pathway in Alzheimer's disease , 1995, Journal of the Neurological Sciences.
[18] J. Ávila,et al. Analysis of microtubule-associated protein tau glycation in paired helical filaments. , 1994, The Journal of biological chemistry.
[19] Patrizia Mecocci,et al. Oxidative damage to mitochondrial DNA is increased in Alzheimer's disease , 1994, Annals of neurology.
[20] R. Domínguez,et al. Copper-zinc superoxide dismutase activity in red blood cells and serum in demented patients and in aging , 1993, Journal of the Neurological Sciences.
[21] S. M. Sumi,et al. The Consortium to Establish a Registry for Alzheimer's Disease (CERAD) , 1991, Neurology.
[22] C. Masters,et al. The Amyloid Precursor Protein of Alzheimer's Disease in the Reduction of Copper(II) to Copper(I) , 1996, Science.
[23] S. Aust,et al. Ferritin and ceruloplasmin in oxidative damage: review and recent findings. , 1993, Canadian journal of physiology and pharmacology.
[24] L. Goodman. Alzheimer's disease; a clinico-pathologic analysis of twenty-three cases with a theory on pathogenesis. , 1953, The Journal of nervous and mental disease.
[25] R. Terry,et al. Senile dementia of the Alzheimer type , 1983, Annals of neurology.
[26] Ferritin: isolation of aluminum-ferritin complex from brain. , 1987, Proceedings of the National Academy of Sciences of the United States of America.
[27] B T Hyman,et al. Functional Alterations in Alzheimer's Disease: Diminution of Cytochrome Oxidase in the Hippocampal Formation , 1993, Journal of neuropathology and experimental neurology.
[28] A. Burns. Clinical diagnosis of Alzheimer's disease , 1991 .
[29] J. Connor,et al. Ceruloplasmin levels in the human superior temporal gyrus in aging and Alzheimer's disease , 1993, Neuroscience Letters.
[30] C. Olanow. A radical hypothesis for neurodegeneration , 1993, Trends in Neurosciences.
[31] W. D. Ehmann,et al. Non-destructive analysis of copper in human brain tissue by neutron activation analysis using coincidence and anti-coincidence techniques , 1997 .
[32] W. D. Ehmann,et al. Regional brain trace-element studies in Alzheimer's disease. , 1988, Neurotoxicology.
[33] S. Rapoport,et al. Impairment in mitochondrial cytochrome oxidase gene expression in Alzheimer disease. , 1994, Brain research. Molecular brain research.
[34] W. Markesbery,et al. Excess brain protein oxidation and enzyme dysfunction in normal aging and in Alzheimer disease. , 1991, Proceedings of the National Academy of Sciences of the United States of America.
[35] M. Mattson,et al. β-Amyloid Peptide Free Radical Fragments Initiate Synaptosomal Lipoperoxidation in a Sequence-Specific Fashion: Implications to Alzheimer′s Disease , 1994 .
[36] M. Mattson,et al. A model for beta-amyloid aggregation and neurotoxicity based on free radical generation by the peptide: relevance to Alzheimer disease. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[37] W. D. Ehmann,et al. Brain trace elements in Alzheimer's disease. , 1986, Neurotoxicology.
[38] R. Domínguez,et al. Copper-zinc superoxide dismutase activity in red blood cells in probable Alzheimer's patients and their first-degree relatives , 1994, Journal of the Neurological Sciences.
[39] D. Walker,et al. Lactotransferrin immunocytochemistry in Alzheimer and normal human brain. , 1993, The American journal of pathology.
[40] D. Perl,et al. Selective accumulation of aluminum and iron in the neurofibrillary tangles of Alzheimer's disease: A laser microprobe (LAMMA) study , 1992, Annals of neurology.
[41] D. Miller,et al. Effects of ceruloplasmin on superoxide-dependent iron release from ferritin and lipid peroxidation. , 1991, Free radical research communications.
[42] Z. Khachaturian. Diagnosis of Alzheimer's disease. , 1985, Archives of neurology.
[43] J. Gutteridge,et al. Antioxidant protection against organic and inorganic oxygen radicals by normal human plasma: the important primary role for iron-binding and iron-oxidising proteins. , 1992, Biochimica et biophysica acta.
[44] N. Ward,et al. Neutron activation analysis techniques for identifying elemental status in Alzheimer's disease , 1987 .
[45] W. D. Ehmann,et al. Instrumental neutron activation analysis of brain aluminum in Alzheimer disease and aging , 1981, Annals of neurology.
[46] F. Burnet. A POSSIBLE ROLE OF ZINC IN THE PATHOLOGY OF DEMENTIA , 1981, The Lancet.
[47] R. Webster,et al. Ceruloplasmin inhibits carbonyl formation in endogenous cell proteins. , 1993, Free radical biology & medicine.
[48] M. Beal,et al. Cortical Cytochrome Oxidase Activity Is Reduced in Alzheimer's Disease , 1994, Journal of neurochemistry.