Development of Spontaneous Autoimmune Peripheral Polyneuropathy in B7-2–Deficient Nod Mice
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A. Montag | J. Bluestone | H. Hsin | S. Miller | B. Soliven | B. Salomon | L. Rhee | J. Arcella | A. M. Girvin | Hélène Bour-Jordan
[1] Colin McKerlie,et al. Type I Diabetes and Multiple Sclerosis Patients Target Islet Plus Central Nervous System Autoantigens; Nonimmunized Nonobese Diabetic Mice Can Develop Autoimmune Encephalitis1 , 2001, The Journal of Immunology.
[2] J. Bluestone,et al. B7/CD28 costimulation is essential for the homeostasis of the CD4+CD25+ immunoregulatory T cells that control autoimmune diabetes. , 2000, Immunity.
[3] J. Bluestone,et al. A Critical Role for B7/CD28 Costimulation in Experimental Autoimmune Encephalomyelitis: A Comparative Study Using Costimulatory Molecule-Deficient Mice and Monoclonal Antibody Blockade1 , 2000, The Journal of Immunology.
[4] M. M. Griffiths,et al. Mapping autoimmunity genes. , 1999, Current opinion in immunology.
[5] V. Kuchroo,et al. Studies in B7-Deficient Mice Reveal a Critical Role for B7 Costimulation in Both Induction and Effector Phases of Experimental Autoimmune Encephalomyelitis , 1999, The Journal of experimental medicine.
[6] K. Becker,et al. Comparative genetics of type 1 diabetes and autoimmune disease: common loci, common pathways? , 1999, Diabetes.
[7] J. Seidman,et al. QTL influencing autoimmune diabetes and encephalomyelitis map to a 0.15-cM region containing Il2 , 1999, Nature Genetics.
[8] J. Todd,et al. From genome to aetiology in a multifactorial disease, type 1 diabetes , 1999, BioEssays : news and reviews in molecular, cellular and developmental biology.
[9] T. Mak,et al. CD28-deficient mice are highly resistant to collagen-induced arthritis. , 1999, Journal of immunology.
[10] H. Mcdevitt,et al. The role of MHC class II molecules in susceptibility and resistance to autoimmunity. , 1998, Current opinion in immunology.
[11] H. Hartung,et al. Guillain-Barré syndrome, CIDP and other chronic immune-mediated neuropathies. , 1998, Current opinion in neurology.
[12] J. Trent,et al. Clustering of non-major histocompatibility complex susceptibility candidate loci in human autoimmune diseases. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[13] Nitin J. Karandikar,et al. Tissue-specific up-regulation of B7-1 expression and function during the course of murine relapsing experimental autoimmune encephalomyelitis. , 1998, Journal of immunology.
[14] Nitin J. Karandikar,et al. Treatment with intact anti-B7-1 mAb during disease remission enhances epitope spreading and exacerbates relapses in R-EAE , 1997, Journal of Neuroimmunology.
[15] E. Simpson,et al. B7-1 and B7-2 have overlapping, critical roles in immunoglobulin class switching and germinal center formation. , 1997, Immunity.
[16] E. Fuchs,et al. CD28/B7 regulation of Th1 and Th2 subsets in the development of autoimmune diabetes. , 1996, Immunity.
[17] N. Maclaren,et al. Although DR3-DQB1*0201 may be associated with multiple component diseases of the autoimmune polyglandular syndromes, the human leukocyte antigen DR4-DQB1*0302 haplotype is implicated only in beta-cell autoimmunity. , 1996, The Journal of clinical endocrinology and metabolism.
[18] Nitin J. Karandikar,et al. Blockade of CD28/B7-1 interaction prevents epitope spreading and clinical relapses of murine EAE. , 1995, Immunity.
[19] Laurie H Glimcher,et al. B7-1 and B7-2 costimulatory molecules activate differentially the Th1/Th2 developmental pathways: Application to autoimmune disease therapy , 1995, Cell.
[20] J. Bluestone,et al. Differential effects of anti-B7-1 and anti-B7-2 monoclonal antibody treatment on the development of diabetes in the nonobese diabetic mouse , 1995, The Journal of experimental medicine.
[21] A. Cooke,et al. Mycobacteria precipitate an SLE-like syndrome in diabetes-prone NOD mice. , 1994, Immunology.
[22] P. Linsley,et al. Treatment of murine lupus with CTLA4Ig. , 1994, Science.
[23] J. Allison,et al. CD28-mediated signalling co-stimulates murine T cells and prevents induction of anergy in T-cell clones , 1992, Nature.
[24] P. Pozzilli,et al. Parathyroiditis in the non-obese diabetic mouse--a new finding. , 1991, The Journal of endocrinology.
[25] P. Linsley,et al. Binding of the B cell activation antigen B7 to CD28 costimulates T cell proliferation and interleukin 2 mRNA accumulation , 1991, The Journal of experimental medicine.
[26] R. Schwartz,et al. Antigen presentation by chemically modified splenocytes induces antigen- specific T cell unresponsiveness in vitro and in vivo , 1987, The Journal of experimental medicine.
[27] S. Hsu,et al. Use of avidin-biotin-peroxidase complex (ABC) in immunoperoxidase techniques: a comparison between ABC and unlabeled antibody (PAP) procedures. , 1981, The journal of histochemistry and cytochemistry : official journal of the Histochemistry Society.
[28] J. Bluestone,et al. Complexities of CD28/B7: CTLA-4 costimulatory pathways in autoimmunity and transplantation. , 2001, Annual review of immunology.