Design and Synthesis of 1,3,5-Triazines or Pyrimidines Containing Dithiocarbamate Moiety as PI3Kα Selective Inhibitors.
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Yanfang Zhao | Yajing Liu | Siyu Fu | Jiuyu Liu | Xinzi He | Yanli Zhu | Kang Wang | Chunting Li | Ping Gong | Yunlei Hou | Jiechun Tang | Yuan Li
[1] Siew K. Yeap,et al. Discovery of GDC-0077 (Inavolisib), a Highly Selective Inhibitor and Degrader of Mutant PI3Kα. , 2022, Journal of medicinal chemistry.
[2] Yu Wang,et al. Based on 2-(difluoromethyl)-1-[4,6-di(4-morpholinyl)-1,3,5-triazin-2-yl]-1H-benzimidazole (ZSTK474), design, synthesis and biological evaluation of novel PI3Kα selective inhibitors. , 2022, Bioorganic chemistry.
[3] A. Jemal,et al. Cancer statistics, 2022 , 2022, CA: a cancer journal for clinicians.
[4] R. Liu,et al. Biomimetic Small-Molecule Self-Assembly of PI3K Inhibitor Integrated with Immunomodulator to Amplify Anticancer Efficacy , 2021, Chemical Engineering Journal.
[5] K. Okkenhaug,et al. PI3K inhibitors are finally coming of age , 2021, Nature Reviews Drug Discovery.
[6] Xiaoqing Lv,et al. Structural optimization towards promising β-methyl-4-acrylamido quinoline derivatives as PI3K/mTOR dual inhibitors for anti-cancer therapy: The in vitro and in vivo biological evaluation. , 2021, European journal of medicinal chemistry.
[7] Ping Chen,et al. Structure-Based Drug Design and Synthesis of PI3Kα-Selective Inhibitor (PF-06843195). , 2020, Journal of medicinal chemistry.
[8] Juntuo Zhou,et al. Discovery of 2,4-diarylaminopyrimidine derivatives bearing dithiocarbamate moiety as novel FAK inhibitors with antitumor and anti-angiogenesis activities. , 2019, European journal of medicinal chemistry.
[9] Michael J. Stocks,et al. Class 1 PI3K Clinical Candidates and Recent Inhibitor Design Strategies: A Medicinal Chemistry Perspective. , 2019, Journal of medicinal chemistry.
[10] Lewis C. Cantley,et al. The PI3K Pathway in Human Disease , 2017, Cell.
[11] R. Copeland,et al. Design, synthesis and cytotoxicity studies of dithiocarbamate ester derivatives of emetine in prostate cancer cell lines. , 2015, Bioorganic & medicinal chemistry.
[12] B. Ross,et al. Dual inhibition of allosteric mitogen-activated protein kinase (MEK) and phosphatidylinositol 3-kinase (PI3K) oncogenic targets with a bifunctional inhibitor. , 2015, Bioorganic & medicinal chemistry.
[13] V. Bala,et al. Chemical and medicinal versatility of dithiocarbamates: an overview. , 2014, Mini reviews in medicinal chemistry.
[14] J. Bendell,et al. Phase I dose-escalation and -expansion study of buparlisib (BKM120), an oral pan-Class I PI3K inhibitor, in patients with advanced solid tumors , 2014, Investigational New Drugs.
[15] C. Rommel,et al. PI3K and cancer: lessons, challenges and opportunities , 2014, Nature Reviews Drug Discovery.
[16] Lu Lu,et al. Small molecule fusion inhibitors: design, synthesis and biological evaluation of (Z)-3-(5-(3-benzyl-4-oxo-2-thioxothiazolidinylidene)methyl)-N-(3-carboxy-4-hydroxy)phenyl-2,5-dimethylpyrroles and related derivatives targeting HIV-1 gp41. , 2013, Bioorganic & medicinal chemistry.
[17] D. Mobley,et al. Triazole-dithiocarbamate based selective lysine specific demethylase 1 (LSD1) inactivators inhibit gastric cancer cell growth, invasion, and migration. , 2013, Journal of medicinal chemistry.
[18] G. Çiftçi,et al. Synthesis and Biological Evaluation of Some Pyrazoline Derivatives Bearing a Dithiocarbamate Moiety as New Cholinesterase Inhibitors , 2013, Archiv der Pharmazie.
[19] Zaheer Ul-Haq,et al. Synthesis, biological evaluation, and molecular docking studies of benzyl, alkyl and glycosyl [2-(arylamino)-4,4-dimethyl-6-oxo-cyclohex-1-ene]carbodithioates, as potential immunomodulatory and immunosuppressive agents. , 2012, Bioorganic & medicinal chemistry.
[20] C. Supuran,et al. Dithiocarbamates are strong inhibitors of the beta-class fungal carbonic anhydrases from Cryptococcus neoformans, Candida albicans and Candida glabrata. , 2012, Bioorganic & medicinal chemistry letters.
[21] Claire L. Lill,et al. Synthesis and biological evaluation of novel analogues of the pan class I phosphatidylinositol 3-kinase (PI3K) inhibitor 2-(difluoromethyl)-1-[4,6-di(4-morpholinyl)-1,3,5-triazin-2-yl]-1H-benzimidazole (ZSTK474). , 2011, Journal of medicinal chemistry.
[22] B. Vanhaesebroeck,et al. The emerging mechanisms of isoform-specific PI3K signalling , 2010, Nature Reviews Molecular Cell Biology.
[23] Pixu Liu,et al. Targeting the phosphoinositide 3-kinase pathway in cancer , 2009, Nature Reviews Drug Discovery.
[24] Holger Gerhardt,et al. Angiogenesis selectively requires the p110α isoform of PI3K to control endothelial cell migration , 2008, Nature.
[25] D. Giustarini,et al. Protein S-glutathionylation and platelet anti-aggregating activity of disulfiram. , 2006, Biochemical pharmacology.
[26] Ji Luo,et al. The evolution of phosphatidylinositol 3-kinases as regulators of growth and metabolism , 2006, Nature Reviews Genetics.
[27] S. Hirono,et al. Antitumor activity of ZSTK474, a new phosphatidylinositol 3-kinase inhibitor. , 2006, Journal of the National Cancer Institute.
[28] H. Simmonds,et al. Pyrimidine pathways in health and disease. , 2005, Trends in molecular medicine.
[29] A. Bilancio,et al. Signalling by PI3K isoforms: insights from gene-targeted mice. , 2005, Trends in biochemical sciences.
[30] H. Sasahara,et al. Synthesis and antitumor activity of benzimidazolyl-1,3,5-triazine and benzimidazolylpyrimidine derivatives. , 2000, Chemical & pharmaceutical bulletin.
[31] S. Yaguchi,et al. In vitro cytotoxicity of imidazolyl-1,3,5-triazine derivatives. , 1997, Biological & pharmaceutical bulletin.