Macrophage‐Colony Stimulating Factor in Obese Adipose Tissue: Studies With Heterozygous op/+ Mice

[1]  S. Yamaoka,et al.  Role of the Toll-like Receptor 4/NF-&kgr;B Pathway in Saturated Fatty Acid–Induced Inflammatory Changes in the Interaction Between Adipocytes and Macrophages , 2007, Arteriosclerosis, thrombosis, and vascular biology.

[2]  T. Kadowaki,et al.  Overexpression of Monocyte Chemoattractant Protein-1 in Adipose Tissues Causes Macrophage Recruitment and Insulin Resistance* , 2006, Journal of Biological Chemistry.

[3]  R. Yu,et al.  Mesenteric Adipose Tissue‐Derived Monocyte Chemoattractant Protein‐1 Plays a Crucial Role in Adipose Tissue Macrophage Migration and Activation in Obese Mice , 2006, Obesity.

[4]  R. Kitazawa,et al.  MCP-1 contributes to macrophage infiltration into adipose tissue, insulin resistance, and hepatic steatosis in obesity. , 2006, The Journal of clinical investigation.

[5]  R. Leibel,et al.  CCR2 modulates inflammatory and metabolic effects of high-fat feeding. , 2006, The Journal of clinical investigation.

[6]  T. Suganami,et al.  A Paracrine Loop Between Adipocytes and Macrophages Aggravates Inflammatory Changes: Role of Free Fatty Acids and Tumor Necrosis Factor α , 2005, Arteriosclerosis, thrombosis, and vascular biology.

[7]  Chunsheng Zhang,et al.  Diet induction of monocyte chemoattractant protein-1 and its impact on obesity. , 2005, Obesity research.

[8]  F. Pixley,et al.  CSF-1 regulation of the wandering macrophage: complexity in action. , 2004, Trends in cell biology.

[9]  M. Desai,et al.  Obesity is associated with macrophage accumulation in adipose tissue. , 2003, The Journal of clinical investigation.

[10]  L. Tartaglia,et al.  Chronic inflammation in fat plays a crucial role in the development of obesity-related insulin resistance. , 2003, The Journal of clinical investigation.

[11]  H. Kotani,et al.  Adiposity Elevates Plasma MCP-1 Levels Leading to the Increased CD11b-positive Monocytes in Mice* , 2003, Journal of Biological Chemistry.

[12]  B. Yandell,et al.  The expression of adipogenic genes is decreased in obesity and diabetes mellitus. , 2000, Proceedings of the National Academy of Sciences of the United States of America.

[13]  E. C. Weir,et al.  Nuclear Factor-κB p50 Is Required for Tumor Necrosis Factor-α-Induced Colony-Stimulating Factor-1 Gene Expression in Osteoblasts. , 2000, Endocrinology.

[14]  N. Socci,et al.  Leptin-specific patterns of gene expression in white adipose tissue. , 2000, Genes & development.

[15]  Gregory S. Barsh,et al.  Genetics of body-weight regulation , 2000, Nature.

[16]  J. Rubin,et al.  Role of NFκB in the regulation of macrophage colony stimulating factor by tumor necrosis factor‐α in ST2 bone stromal cells , 1999 .

[17]  P. Libby,et al.  Heterozygous osteopetrotic (op) mutation reduces atherosclerosis in LDL receptor- deficient mice. , 1998, The Journal of clinical investigation.

[18]  J. Levine,et al.  Adipocyte macrophage colony-stimulating factor is a mediator of adipose tissue growth. , 1998, The Journal of clinical investigation.

[19]  A. Greenberg,et al.  Journal of Clinical Endocrinology and Metabolism Printed in U.S.A. Copyright © 1998 by The Endocrine Society Omental and Subcutaneous Adipose Tissues of Obese Subjects Release Interleukin-6: Depot Difference and Regulation by Glucocorticoid* , 1997 .

[20]  菊池 太朗,et al.  ラット椎間板皮下吸収モデルにおけるMCP-1(monocyte chemoattractant protein-1)の動向について , 1996 .

[21]  D. Kufe,et al.  Involvement of a nuclear factor-kappa B-like protein in induction of the macrophage colony-stimulating factor gene by tumor necrosis factor. , 1991, Blood.

[22]  S. Niida,et al.  BriefDefinitive Report Essential Role of Macrophage Colony-Stimulating Factor in the Osteoclast Differentiation Supported by , 2022 .

[23]  W. Wiktor-Jedrzejczak,et al.  Total absence of colony-stimulating factor 1 in the macrophage-deficient osteopetrotic (op/op) mouse. , 1990, Proceedings of the National Academy of Sciences of the United States of America.

[24]  S. Nishikawa,et al.  The murine mutation osteopetrosis is in the coding region of the macrophage colony stimulating factor gene , 1990, Nature.

[25]  Ichiro,et al.  Overexpression of MCP-1 in adipose tissues causes macrophage recruitment and insulin resistance , 2006 .

[26]  L. Pénicaud,et al.  Preadipocyte conversion to macrophage: evidence of plasticity , 2002 .

[27]  E. C. Weir,et al.  Nuclear factor-kappaB p50 is required for tumor necrosis factor-alpha-induced colony-stimulating factor-1 gene expression in osteoblasts. , 2000, Endocrinology.

[28]  K. Motoyoshi,et al.  [Macrophage colony-stimulating factor]. , 1999, Nihon rinsho. Japanese journal of clinical medicine.

[29]  J. Rubin,et al.  Role of NFkappaB in the regulation of macrophage colony stimulating factor by tumor necrosis factor-alpha in ST2 bone stromal cells. , 1999, Journal of cellular physiology.

[30]  B. Spiegelman,et al.  Adipose expression of tumor necrosis factor-alpha: direct role in obesity-linked insulin resistance. , 1993, Science.