Cytogénétique Hématologique (GFCH) d'Hématologie Cellulaire (GFHC) and the Groupe Français de gene mutations: a report of 59 cases by the Groupe Français M0 AML, clinical and biologic features of the disease, includingAML1
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O. Casasnovas | E. Macintyre | R. Berger | G. Flandrin | C. Preudhomme | P. Fenaux | V. Eclache | F. Davi | C. Arnoulet | N. Dastugue | H. Jouault | C. Roumier | J. Laï | P. Talmant | M. Maynadié | I. Radford | O. Fenneteau | F. Valensi | P. Lepelley | E. Duchayne | M. Imbert | P. Cornillet | F. Garnache | B. Pedron | Najiba Boudjerra | Chrystele Bilhou-Naberra
[1] C. Preudhomme,et al. New mechanisms of AML1 gene alteration in hematological malignancies , 2003, Leukemia.
[2] G. Ehninger,et al. Analysis of Flt3-activating Mutations in 979 Patients with Acute Myelogenous Leukemia: Association with Fab Subtypes and Identification of Subgroups with Poor Prognosis , 2022 .
[3] J. V. van Dongen,et al. Basic helix-loop-helix proteins E2A and HEB induce immature T-cell receptor rearrangements in nonlymphoid cells. , 2001, Blood.
[4] Claudia Schoch,et al. Acute myeloid leukaemia M0: haematological, immunophenotypic and cytogenetic characteristics and their prognostic significance: an analysis in 241 patients , 2001, British journal of haematology.
[5] G. Morgan,et al. Mutations in the AML1 gene in acute myeloid leukaemia patients with FAB types M0 and M7. , 2000 .
[6] E. Macintyre,et al. Rapid, multifluorescent TCRG Vγ and Jγ typing: application to T cell acute lymphoblastic leukemia and to the detection of minor clonal populations , 2000, Leukemia.
[7] E. Macintyre,et al. High incidence of biallelic point mutations in the Runt domain of the AML1/PEBP2 alpha B gene in Mo acute myeloid leukemia and in myeloid malignancies with acquired trisomy 21. , 2000, Blood.
[8] T. Naoe,et al. Prognostic implication of FLT3 and N-RAS gene mutations in acute myeloid leukemia. , 1999, Blood.
[9] H. Yamasaki,et al. Biallelic and heterozygous point mutations in the runt domain of the AML1/PEBP2alphaB gene associated with myeloblastic leukemias. , 1999, Blood.
[10] K. Tanaka,et al. TLE, the human homolog of groucho, interacts with AML1 and acts as a repressor of AML1-induced transactivation. , 1998, Biochemical and biophysical research communications.
[11] Z. Paroush,et al. Transcriptional repression by AML1 and LEF-1 is mediated by the TLE/Groucho corepressors. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[12] H. Tien,et al. CD7 positive hematopoietic progenitors and acute myeloid leukemia and other minimally differentiated leukemia. , 1998, Leukemia & lymphoma.
[13] F. Davi,et al. Early onset of immunoglobulin heavy chain gene rearrangements in normal human bone marrow CD34+ cells. , 1997, Blood.
[14] M. Krangel,et al. Regulation of T Cell Receptor δ Gene Rearrangement by CBF/PEBP2 , 1997, The Journal of experimental medicine.
[15] G. Papa,et al. Minimally differentiated acute myeloid leukemia (AML-M0): comparison of 25 cases with other French-American-British subtypes. , 1997, Blood.
[16] P. Cony-Makhoul,et al. Allogeneic vs autologous stem cell transplantation vs chemotherapy in patients with acute myeloid leukemia in first remission: the BGMT 87 study. , 1996, Leukemia.
[17] G. Leverger,et al. Simplified strategies for minimal residual disease detection in B‐cell precursor acute lymphoblastic leukaemia , 1996, British journal of haematology.
[18] P. Hurteloup,et al. Treatment of acute myeloblastic leukemia in adults. The GOELAM experience. , 1996, Hematology and cell therapy.
[19] R. Zittoun. The EORTC trials for acute myelogenous leukemia , 1996 .
[20] M. Tribalto,et al. Minimally differentiated acute myeloid leukemia (AML-MO): a distinct clinico-biologic entity with poor prognosis , 1996, Annals of Hematology.
[21] G. Papa,et al. Biological profile of 23 cases of minimally differentiated acute myeloid leukemia (AML-M0) and its clinical implications. , 1996, Blood.
[22] A Orfao,et al. Proposals for the immunological classification of acute leukemias. European Group for the Immunological Characterization of Leukemias (EGIL). , 1995, Leukemia.
[23] P. Cin,et al. Cytogenetic profile of minimally differentiated (FAB M0) acute myeloid leukemia: correlation with clinicobiologic findings. , 1995, Blood.
[24] M. Tribalto,et al. Minimally differentiated acute myeloid leukaemia (AML‐MO): cytochemical, immunophenotypic and cytogenetic analysis of 19 cases , 1994, British journal of haematology.
[25] H. Cavé,et al. Prospective monitoring and quantitation of residual blasts in childhood acute lymphoblastic leukemia by polymerase chain reaction study of delta and gamma T-cell receptor genes. , 1994, Blood.
[26] P. Fenaux,et al. Place de la double induction dans le traitement des leucémies aiguës myéloblastiques de l'adulte : résultats intermédiaires du protocole LAM90 , 1994 .
[27] J. Testa,et al. Minimally differentiated acute nonlymphocytic leukemia: a distinct entity. , 1987, Blood.