Slowly eroding lesions in multiple sclerosis
暂无分享,去创建一个
D. Reich | M. Absinta | B. Dewey | G. Nair | V. Sethi | Tianxia Wu | C. Shea | I. Cortese | P. Sati | J. Ohayon | Kelly Yang | A. Venkataraman
[1] Istvan Pirko,et al. Clinical and pathological insights into the dynamic nature of the white matter multiple sclerosis plaque , 2015, Annals of neurology.
[2] C. Crainiceanu,et al. Quantification of multiple-sclerosis-related brain atrophy in two heterogeneous MRI datasets using mixed-effects modeling☆ , 2013, NeuroImage: Clinical.
[3] D. Reich,et al. The effect of daclizumab on brain atrophy in relapsing-remitting multiple sclerosis. , 2013, Multiple sclerosis and related disorders.
[4] A. Gass,et al. Contribution of cortical and white matter lesions to cognitive impairment in multiple sclerosis , 2013, Multiple sclerosis.
[5] D. Reich,et al. Multiple sclerosis shrinks intralesional, and enlarges extralesional, brain parenchymal veins , 2013, Neurology.
[6] M. Calabrese,et al. Cortical lesion load associates with progression of disability in multiple sclerosis. , 2012, Brain : a journal of neurology.
[7] J. Parratt,et al. Oligodendrocytes and the early multiple sclerosis lesion , 2012, Annals of neurology.
[8] Peter A. Calabresi,et al. Revisiting Brain Atrophy and Its Relationship to Disability in Multiple Sclerosis , 2012, PloS one.
[9] Eini Niskanen,et al. Evolution of global and local grey matter atrophy on serial MRI scans during the progression from MCI to AD. , 2012, Current Alzheimer research.
[10] M. Calabrese,et al. Cortical lesions and cognitive impairment in multiple sclerosis , 2010, Neurological Sciences.
[11] F. Barkhof,et al. Diffusely Abnormal White Matter in Progressive Multiple Sclerosis: In Vivo Quantitative MR Imaging Characterization and Comparison between Disease Types , 2010, American Journal of Neuroradiology.
[12] Peter A. Calabresi,et al. A topology-preserving approach to the segmentation of brain images with multiple sclerosis lesions , 2010, NeuroImage.
[13] Frederik Barkhof,et al. Diffusely abnormal white matter in chronic multiple sclerosis: imaging and histopathologic analysis. , 2009, Archives of neurology.
[14] M. Schlamann,et al. First Clinical Study on Ultra-High-Field MR Imaging in Patients with Multiple Sclerosis: Comparison of 1.5T and 7T , 2009, American Journal of Neuroradiology.
[15] H. Lassmann. The pathologic substrate of magnetic resonance alterations in multiple sclerosis. , 2008, Neuroimaging clinics of North America.
[16] DL Collins,et al. Atrophy in white matter fiber tracts in multiple sclerosis is not dependent on tract length or local white matter lesions , 2008, Multiple sclerosis.
[17] A. Thompson,et al. Disability and T2 MRI lesions: a 20-year follow-up of patients with relapse onset of multiple sclerosis. , 2008, Brain : a journal of neurology.
[18] I. Pirko,et al. Neuroimaging of demyelination and remyelination models. , 2008, Current topics in microbiology and immunology.
[19] Hans Lassmann,et al. The Immunopathology of Multiple Sclerosis: An Overview , 2007, Brain pathology.
[20] Axel Wismüller,et al. BMC Neurology BioMed Central , 2006 .
[21] David H. Miller,et al. A controlled trial of natalizumab for relapsing multiple sclerosis. , 2003, The New England journal of medicine.
[22] David H. Miller,et al. Measurement of atrophy in multiple sclerosis: pathological basis, methodological aspects and clinical relevance. , 2002, Brain : a journal of neurology.
[23] Massimo Filippi,et al. Image registration and subtraction to detect active T2 lesions in MS: an interobserver study , 2002, Journal of Neurology.
[24] Alan J. Thompson,et al. Disability and lesion load in MS: a reassessment with MS functional composite score and 3D fast FLAIR , 2002, Journal of Neurology.
[25] E. Cho,et al. Immunopathology of secondary‐progressive multiple sclerosis , 2001, Annals of neurology.
[26] J L Ostuni,et al. Interferon beta-1b and intravenous methylprednisolone promote lesion recovery in multiple sclerosis , 2001, Multiple sclerosis.
[27] T. Riise,et al. Disability and prognosis in multiple sclerosis: demographic and clinical variables important for the ability to walk and awarding of disability pension , 2001, Multiple sclerosis.
[28] N. Mai,et al. Characteristic MR lesion pattern and correlation of T1 and T2 lesion volume with neurologic and neuropsychological findings in cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy (CADASIL). , 1999, AJNR. American journal of neuroradiology.
[29] G. Wolswijk. Chronic Stage Multiple Sclerosis Lesions Contain a Relatively Quiescent Population of Oligodendrocyte Precursor Cells , 1998, The Journal of Neuroscience.
[30] A J Thompson,et al. Multiple sclerosis lesion detection in the brain: A comparison of fast fluid-attenuated inversion recovery and conventional T2-weighted dual spin echo , 1997, Neurology.
[31] M Filippi,et al. Comparison of MR pulse sequences in the detection of multiple sclerosis lesions. , 1997, AJNR. American journal of neuroradiology.
[32] M. Horsfield,et al. Quantitative assessment of MRI lesion load in monitoring the evolution of multiple sclerosis. , 1995, Brain : a journal of neurology.
[33] Interferon beta-1b in the treatment of multiple sclerosis: final outcome of the randomized controlled trial. The IFNB Multiple Sclerosis Study Group and The University of British Columbia MS/MRI Analysis Group. , 1995, Neurology.
[34] E. Cho,et al. Multiple sclerosis: Remyelination of nascent lesions: Remyelination of nascent lesions , 1993 .
[35] E. Cho,et al. Multiple sclerosis: remyelination of nascent lesions. , 1993, Annals of neurology.
[36] J. Dawson,et al. The Histology of Disseminated Sclerosis , 1916, Edinburgh Medical Journal.
[37] J. Dawson,et al. XVIII.–The Histology of Disseminated Sclerosis , 1916, Transactions of the Royal Society of Edinburgh.