CLMP Promotes Leukocyte Migration Across Brain Barriers in Multiple Sclerosis
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P. Duquette | A. Bouthillier | C. Larochelle | J. I. Alvarez | A. Prat | M. Girard | R. Cayrol | M. Charabati | Jorge I Alvarez | R. Moumdjian | Stephanie E. J. Zandee | Hania Kebir | N. Arbour | L. Bourbonnière | Sandra Larouche | A. Fournier | E. Peelen | F. Tea | O. Tastet | Marc Charabati | Olivier Tastet | Boaz Lahav | Wendy Klement | J. Alvarez | W. Klement | S. Larouche | B. Lahav
[1] F. Quintana,et al. DICAM promotes TH17 lymphocyte trafficking across the blood-brain barrier during autoimmune neuroinflammation , 2022, Science Translational Medicine.
[2] A. Rae-Grant,et al. Diagnosis and Treatment of Multiple Sclerosis: A Review. , 2021, JAMA.
[3] F. Rathjen. The CAR group of Ig cell adhesion proteins–Regulators of gap junctions? , 2020, BioEssays : news and reviews in molecular, cellular and developmental biology.
[4] A. Bar-Or,et al. Activated leukocyte cell adhesion molecule regulates B lymphocyte migration across central nervous system barriers , 2019, Science Translational Medicine.
[5] P. Duquette,et al. CD70 defines a subset of proinflammatory and CNS-pathogenic TH1/TH17 lymphocytes and is overexpressed in multiple sclerosis , 2019, Cellular & Molecular Immunology.
[6] F. Rathjen,et al. Cell-cell communication mediated by the CAR subgroup of immunoglobulin cell adhesion molecules in health and disease , 2017, Molecular and Cellular Neuroscience.
[7] B. Engelhardt,et al. The movers and shapers in immune privilege of the CNS , 2017, Nature Immunology.
[8] F. Zipp,et al. Dendritic cells as therapeutic targets in neuroinflammation , 2016, Cellular and Molecular Life Sciences.
[9] D. Pleasure,et al. Therapeutic depletion of monocyte-derived cells protects from long-term axonal loss in experimental autoimmune encephalomyelitis , 2016, Journal of Neuroimmunology.
[10] C. Larochelle,et al. JAML mediates monocyte and CD8 T cell migration across the brain endothelium , 2015, Annals of clinical and translational neurology.
[11] T. Yednock,et al. Melanoma cell adhesion molecule–positive CD8 T lymphocytes mediate central nervous system inflammation , 2015, Annals of neurology.
[12] H. Wiendl,et al. VLA-4 blockade promotes differential routes into human CNS involving PSGL-1 rolling of T cells and MCAM-adhesion of TH17 cells , 2014, Journal of Neuroimmunology.
[13] A. Nusrat,et al. JAM-related proteins in mucosal homeostasis and inflammation , 2014, Seminars in Immunopathology.
[14] D. Centonze,et al. Tumor necrosis factor is elevated in progressive multiple sclerosis and causes excitotoxic neurodegeneration , 2014, Multiple sclerosis.
[15] A. Nusrat,et al. JAM-related proteins in mucosal homeostasis and inflammation , 2014, Seminars in Immunopathology.
[16] T. Yednock,et al. Melanoma cell adhesion molecule identifies encephalitogenic T lymphocytes and promotes their recruitment to the central nervous system. , 2012, Brain : a journal of neurology.
[17] F. Charron,et al. The Hedgehog Pathway Promotes Blood-Brain Barrier Integrity and CNS Immune Quiescence , 2011, Science.
[18] C. Larochelle,et al. How do immune cells overcome the blood–brain barrier in multiple sclerosis? , 2011, FEBS letters.
[19] R. Reynolds,et al. Meningeal inflammation is widespread and linked to cortical pathology in multiple sclerosis. , 2011, Brain : a journal of neurology.
[20] Jeffrey A. Cohen,et al. Diagnostic criteria for multiple sclerosis: 2010 Revisions to the McDonald criteria , 2011, Annals of neurology.
[21] R. Reynolds,et al. A Gradient of neuronal loss and meningeal inflammation in multiple sclerosis , 2010, Annals of neurology.
[22] P. Duquette,et al. Preferential recruitment of interferon‐γ–expressing TH17 cells in multiple sclerosis , 2009, Annals of neurology.
[23] D. West,et al. Monoclonal antibody-associated progressive multifocal leucoencephalopathy in patients treated with rituximab, natalizumab, and efalizumab: a Review from the Research on Adverse Drug Events and Reports (RADAR) Project. , 2009, The Lancet. Oncology.
[24] P. Couraud,et al. JAM-L–mediated leukocyte adhesion to endothelial cells is regulated in cis by α4β1 integrin activation , 2008, The Journal of cell biology.
[25] D. Bourdette,et al. B-cell depletion with rituximab in relapsing-remitting multiple sclerosis , 2008, Current neurology and neuroscience reports.
[26] A. Prat,et al. The blood-brain barrier induces differentiation of migrating monocytes into Th17-polarizing dendritic cells. , 2008, Brain : a journal of neurology.
[27] Danica Stanimirovic,et al. Activated leukocyte cell adhesion molecule promotes leukocyte trafficking into the central nervous system , 2008, Nature Immunology.
[28] S. Cepok,et al. Immune surveillance in multiple sclerosis patients treated with natalizumab , 2006, Annals of neurology.
[29] R. Alon,et al. Immune cell migration in inflammation: present and future therapeutic targets , 2005, Nature Immunology.
[30] P. Meda,et al. Dual interaction of JAM-C with JAM-B and alpha(M)beta2 integrin: function in junctional complexes and leukocyte adhesion. , 2005, Molecular biology of the cell.
[31] H. Makino,et al. Identification of adipocyte adhesion molecule (ACAM), a novel CTX gene family, implicated in adipocyte maturation and development of obesity. , 2005, The Biochemical journal.
[32] J. Fuxe,et al. CLMP, a Novel Member of the CTX Family and a New Component of Epithelial Tight Junctions* , 2004, Journal of Biological Chemistry.
[33] K. Preissner,et al. The Junctional Adhesion Molecule 3 (JAM-3) on Human Platelets is a Counterreceptor for the Leukocyte Integrin Mac-1 , 2002, The Journal of experimental medicine.
[34] S. Cunningham,et al. JAM2 interacts with alpha4beta1. Facilitation by JAM3. , 2002, The Journal of biological chemistry.
[35] A. Zernecke,et al. JAM-1 is a ligand of the β2 integrin LFA-1 involved in transendothelial migration of leukocytes , 2002, Nature Immunology.
[36] E. Dejana,et al. Junctional Adhesion Molecule, a Novel Member of the Immunoglobulin Superfamily That Distributes at Intercellular Junctions and Modulates Monocyte Transmigration , 1998, The Journal of cell biology.
[37] Y. Yamori,et al. ASTROCYTE‐CONDITIONED MEDIUM INDUCES BLOOD‐BRAIN BARRIER PROPERTIES IN ENDOTHELIAL CELLS , 1997, Clinical and experimental pharmacology & physiology.
[38] C. Raine,et al. The adhesion molecule and cytokine profile of multiple sclerosis lesions , 1995, Annals of neurology.
[39] B. Engelhardt,et al. Evidence for involvement of ICAM-1 and VCAM-1 in lymphocyte interaction with endothelium in experimental autoimmune encephalomyelitis in the central nervous system in the SJL/J mouse. , 1994, The American journal of pathology.
[40] C. Brosnan,et al. Identification of lymphotoxin and tumor necrosis factor in multiple sclerosis lesions. , 1991, The Journal of clinical investigation.
[41] Hans Lassmann,et al. An updated histological classification system for multiple sclerosis lesions , 2016, Acta Neuropathologica.
[42] R. Reynolds,et al. Meningeal B-cell follicles in secondary progressive multiple sclerosis associate with early onset of disease and severe cortical pathology. , 2007, Brain : a journal of neurology.
[43] K. Zen,et al. Neutrophil migration across tight junctions is mediated by adhesive interactions between epithelial coxsackie and adenovirus receptor and a junctional adhesion molecule-like protein on neutrophils. , 2005, Molecular biology of the cell.